The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.
The RpoS-Mediated General Stress Response in Escherichia coli
Open accessAbstract
Under conditions of nutrient deprivation or stress, or as cells enter stationary phase, Escherichia coli and related bacteria increase the accumulation of RpoS, a specialized sigma factor. RpoS-dependent gene expression leads to general stress resistance of cells. During rapid growth, RpoS translation is inhibited and any RpoS protein that is synthesized is rapidly degraded. The complex transition from exponential growth to stationary phase has been partially dissected by analyzing the induction of RpoS after specific stress treatments. Different stress conditions lead to induction of specific sRNAs that stimulate RpoS translation or to induction of small-protein antiadaptors that stabilize the protein. Recent progress has led to a better, but still far from complete, understanding of how stresses lead to RpoS induction and what RpoS-dependent genes help the cell deal with the stress.
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Cites
EcoCyc: A comprehensive view of Escherichia coli biology
Keseler, I. M., Bonavides-Martinez, C., Collado-Vides, J., Gama-Castro, S., Gunsalus, R. P., Johnson, D. A., Krummenacker, M., Nolan, L. M., Paley, S., Paulsen, I. T., et al. (2009) · Nucleic Acids Research · doi:10.1093/nar/gkn751 · Cited by 373 · cited 1×
“Mutations characterized by their effects on a particular phenotype or fusion were shown to be in a common gene, now called rpoS, encoding an alternative sigma factor referred to as RpoS (to be used here) or σ38 [see multiple alternative names listed in EcoCyc: katF, appR, otsX, csi2, nur (68)].”
rRNA Transcription in Escherichia coli
Paul, B. J., Ross, W., Gaal, T., & Gourse, R. L. (2004) · Annual Review of Genetics · doi:10.1146/annurev.genet.38.072902.091347 · Cited by 336
Compensatory Evolution of Gene Regulation in Response to Stress by Escherichia coli Lacking RpoS
Stoebel, D. M., Hokamp, K., Last, M. S., & Dorman, C. J. (2009) · PLoS Genetics · doi:10.1371/journal.pgen.1000671 · Cited by 99
Cited by
ATP-Dependent Persister Formation in Escherichia coli
Shan, Y., Brown Gandt, A., Rowe, S. E., Deisinger, J. P., Conlon, B. P., & Lewis, K. (2017) · mBio · doi:10.1128/mbio.02267-16 · Cited by 508
Maisonneuve, E., Castro-Camargo, M., & Gerdes, K. (2013) · Cell · doi:10.1016/j.cell.2013.07.048 · Cited by 438
RetractedKurtz, C. B., Millet, Y. A., Puurunen, M. K., Perreault, M., Charbonneau, M. R., Isabella, V. M., Kotula, J. W., Antipov, E., Dagon, Y., Denney, W. S., et al. (2019) · Science Translational Medicine · doi:10.1126/scitranslmed.aau7975 · Cited by 399
Thomason, M. K., Bischler, T., Eisenbart, S. K., Förstner, K. U., Zhang, A., Herbig, A., Nieselt, K., Sharma, C. M., & Storz, G. (2015) · Journal of Bacteriology · doi:10.1128/jb.02096-14 · Cited by 335
Identity and function of a large gene network underlying mutagenic repair of DNA breaks
Al Mamun, A. A. M., Lombardo, M.-J., Shee, C., Lisewski, A. M., Gonzalez, C., Lin, D., Nehring, R. B., Saint-Ruf, C., Gibson, J. L., Frisch, R. L., et al. (2012) · Science · doi:10.1126/science.1226683 · Cited by 224
Metadata
- Authors
- Battesti, A., Majdalani, N., & Gottesman, S.
- Venue
- Annual Review of Microbiology
- Publisher
- Annual Reviews
- Published
- 2011-10-13
- Volume
- 65
- Issue
- 1
- Pages
- 189-213
- ISSN
- 0066-4227 (ISSN-L)
- 1545-3251
- OpenAlex
- W2165202736
- Cited by
- 918
- References
- 3
- FWCI
- 2.56
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
