Adrenergic mechanisms in recovery from hypoglycemia in man: adrenergic blockade.
Integrity signals
Registry
Cites
Statistics
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
Glucose recovery is not due solely to dissipation of insulin.
“Glucose recovery is not due solely to dissipation of insulin.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
Epinephrine compensates largely for deficient glucagon secretion.
“Glucagon plays a primary counterregulatory role; epinephrine compensates largely for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
Glucagon plays a primary counterregulatory role.
“Glucagon plays a primary counterregulatory role; epinephrine compensates largely for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
The principles of hypoglycemic glucose counterregulation and those of nonhypoglycemic glucose counterregulation in both the postprandial and postabsorptive states are fundamentally the same.
“Thus, the principles of hypoglycemic glucose counterregulation (1-7) and those of nonhypoglycemic glucose counterregulation in both the postprandial (8, 9) and the postabsorptive states are fundamentally the same.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
In both hypoglycemic glucose counterregulation and another model of nonhypoglycemic glucose counterregulation, adrenomedullary epinephrine, but not sympathetic neural norepinephrine, compensates for deficient glucagon secretion.
“Second, in both hypoglycemic glucose counterregulation (1-7) and in another model of nonhypoglycemic glucose counterregulation (9), adrenomedullary epinephrine, but not sympathetic neural norepinephrine, compensates for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
Adrenergic mechanisms do not support postabsorptive glucose production in normal humans.
“On the other hand, adrenergic mechanisms do not support postabsorptive glucose production (2) in normal humans”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
Glucose recovery fails to occur only in the absence of both glucagon and epinephrine.
“Glucose recovery fails to occur only in the absence of both glucagon and epinephrine.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [2]
The citation lists haven’t been fetched yet
References 17 · Cited by 73
- Authors
- Clarke, W. L., Santiago, J. V., Thomas, L., Ben-Galim, E., Haymond, M. W., & Cryer, P. E.
- ORCID
- Haymond, M W
- Venue
- American Journal of Physiology-Endocrinology and Metabolism
- Publisher
- American Physiological Society
- Published
- 1979-02-01
- Volume
- 236
- Issue
- 2
- Pages
- E147
- ISSN
- 0193-1849 (ISSN-L)
- 1522-1555
- OpenAlex
- W2411815483
- Cited by
- 73
- References
- 17
- Citations by year
- 2018: 1 · 2017: 1 · 2015: 1 · 2013: 1 · 2012: 1
- FWCI
- 4.88
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- American Journal of Physiology-Endocrinology and Metabolism (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes Management and Research
- Health Sciences › Medicine › Surgery › Pancreatic function and diabetes
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Adrenal Hormones and Disorders
- Keywords
- hypoglycemia, adrenergic mechanisms, adrenergic blockade, catecholamines, counterregulatory hormones
- MeSH terms
- Adult, Alanine, Epinephrine, Glucagon, Glucose, Humans, Hydrocortisone, Hypoglycemia, Insulin, Kinetics, Male, Norepinephrine, Phentolamine, Propranolol, Growth Hormone
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review