The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.
Cell cycle regulatory proteins in renal disease: role in hypertrophy, proliferation, and apoptosis
Abstract
The response to glomerular and tubulointerstitial cell injury in most forms of renal disease includes changes in cell number (proliferation and apoptosis) and cell size (hyerptrophy). These events typically precede and may be reponsible for the accumulation of extracellular matrix proteins that leads to a decrease in renal function. There is increasing evidence showing that positive (cyclins and cyclin-dependent kinases) and negative (cyclin-dependent kinase inhibitors) cell cycle regulatory proteins have a critical role in regulating these fundamental cellular responses to immune and nonimmune forms of injury. Data now show that altering specific cell cycle proteins affects renal cell proliferation and improves renal function. Equally exciting is the expanding body of literature showing novel biological roles for cell cycle proteins in the regulation of cell hypertrophy and apoptosis. With increasing understanding of the role for cell cyle regulatory proteins in renal disease comes the hope for potential therapeutic inverventions.
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Citations
Cites
The retinoblastoma protein and cell cycle control
Weinberg, R. A. (1995) · Cell · doi:10.1016/0092-8674(95)90385-2 · Cited by 4,862
The p21 inhibitor of cyclin-dependent kinases controls DNA replication by interaction with PCNA
Waga, S., Hannon, G. J., Beach, D., & Stillman, B. (1994) · Nature · doi:10.1038/369574a0 · Cited by 1,655
Radiation-induced cell cycle arrest compromised by p21 deficiency
Brugarolas, J., Chandrasekaran, C., Gordon, J. I., Beach, D., Jacks, T., & Hannon, G. J. (1995) · Nature · doi:10.1038/377552a0 · Cited by 1,242 · cited 1×
“Because p21-knockout mice do not have developmental deficiencies or tumors, it has been suggested that the role of p21 is check-point control of G1/S-phase transition rather than withdrawal from the cell cycle and differentiation (7).”
Background
Cited by
Cell biology of the glomerular podocyte
Pavenstädt, H., Kriz, W., & Kretzler, M. (2003) · Physiological Reviews · doi:10.1152/physrev.00020.2002 · Cited by 1,499
The podocyte’s response to injury: role in proteinuria and glomerulosclerosis
Shankland, S. J. (2006) · Kidney International · doi:10.1038/sj.ki.5000410 · Cited by 846
Self-citationRole of the Microvascular Endothelium in Progressive Renal Disease
Kang, D.-H., Kanellis, J., Hugo, C., Truong, L., Anderson, S., Kerjaschki, D., Schreiner, G. F., & Johnson, R. J. (2002) · Journal of the American Society of Nephrology · doi:10.1681/asn.v133806 · Cited by 466
Accelerated senescence in the kidneys of patients with type 2 diabetic nephropathy
Verzola, D., Gandolfo, M. T., Gaetani, G., Ferraris, A., Mangerini, R., Ferrario, F., Villaggio, B., Gianiorio, F., Tosetti, F., Weiss, U., et al. (2008) · American Journal of Physiology-Renal Physiology · doi:10.1152/ajprenal.90302.2008 · Cited by 302
Pathophysiology of the Diabetic Kidney
Vallon, V., & Komers, R. (2011) · Comprehensive Physiology · doi:10.1002/cphy.c100049 · Cited by 259
Metadata
- Authors
- Shankland, S. J., & Wolf, G.
- Venue
- American Journal of Physiology-Renal Physiology
- Publisher
- American Physiological Society
- Published
- 2000-04-01
- Volume
- 278
- Issue
- 4
- Pages
- F515-F529
- ISSN
- 1522-1466 (ISSN-L)
- 1931-857X
- OpenAlex
- W2186115249
- Cited by
- 194
- References
- 3
- FWCI
- 10.20
- Type
- journal article
- Language
- en
- Open access
- closed
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- Shankland, S. J., & Wolf, G. (2000). Cell cycle regulatory proteins in renal disease: role in hypertrophy, proliferation, and apoptosis. American Journal of Physiology-Renal Physiology. https://doi.org/10.1152/ajprenal.2000.278.4.f515
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