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Cited by 163

The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

Eukaryotic Translation Initiation Factor 4E–Induced Progression of Primary Human Mammary Epithelial Cells along the Cancer Pathway Is Associated with Targeted Translational Deregulation of Oncogenic Drivers and Inhibitors

Larsson, Ola, Li, Shunan, Issaenko, Olga A., Avdulov, Svetlana, Peterson, Mark, Smith, Karen, Bitterman, Peter B., Polunovsky, Vitaly A.·2007·Cancer Research

Abstract

Pathologic redirection of translational control by constitutive activation of eukaryotic translation initiation factor 4F (eIF4F), the cap-dependent translation initiation apparatus, is an obligatory step in oncogenesis; however, its mechanism remains undefined. Here, we simulate this pro-oncogenic state by overexpressing eIF4E, the rate-limiting component of eIF4F, in primary human mammary epithelial cells (HMECs) and examine the resultant changes in cell biology and gene expression profiles of total and polyribosome-bound mRNA genome wide. Overexpressed eIF4E rescues primary HMECs from telomere-independent growth arrest and disables checkpoints governing S-phase entry as well as apoptosis in HMECs immortalized by telomerase, imparting cells with proliferative and survival autonomy. Although the transcriptional response to increased eIF4E was modest, the translational response was large, selective, and bidirectional. In addition to translational activation of known and novel eIF4E-responsive oncogenic drivers regulating cell growth and survival, our data unveil previously unrecognized cellular defenses including translational activation of tumor suppressors, translational repression of transcripts enriched with miRNA target sites, and translational modulation of genes governing translation itself. These findings provide insight into the proneoplastic and compensatory mechanisms embedded in the oncogenic translational program. They support a model whereby deregulated eIF4E moves human epithelial cells along the cancer pathway by profoundly altering ribosomal recruitment to cancer-related transcripts, and eIF4E-modified cells counter these potentially oncogenic alterations with a compensatory translational mechanism that mitigates acquisition of malignancy. [Cancer Res 2007;67(14):6814–24]

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Metadata

Authors
Larsson, O., Li, S., Issaenko, O. A., Avdulov, S., Peterson, M., Smith, K., Bitterman, P. B., & Polunovsky, V. A.
Venue
Cancer Research
Published
2007-07-15
Volume
67
Issue
14
Pages
6814-6824
ISSN
  • 0008-5472 (ISSN-L)
  • 1538-7445
OpenAlex
W2168805216
Cited by
163
References
3
FWCI
3.71
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Eukaryotic Translation Initiation Factor 4E–Induced Progression of Primary Human Mammary Epithelial Cells along the Cancer Pathway Is Associated with Targeted Translational Deregulation of Oncogenic Drivers and Inhibitors. https://app.cochat.ai/papers/10.1158/0008-5472.can-07-0752
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Larsson, O., Li, S., Issaenko, O. A., Avdulov, S., Peterson, M., Smith, K., Bitterman, P. B., & Polunovsky, V. A. (2007). Eukaryotic Translation Initiation Factor 4E–Induced Progression of Primary Human Mammary Epithelial Cells along the Cancer Pathway Is Associated with Targeted Translational Deregulation of Oncogenic Drivers and Inhibitors. Cancer Research. https://doi.org/10.1158/0008-5472.can-07-0752

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