The DOI matches its Crossref record; 53 of the 53 references it cites were found in a registry or an index.
Cisplatin-Associated Nephrotoxicity and Pathological Events
Abstract
Cisplatin (cis-diamminedichloroplatinum(II)) is an effective chemotherapeutic agent, and is successfully used in the treatment of a wide range of tumors. Despite its effectiveness as an anti-tumor drug, nephrotoxic side effects have significantly restricted its clinical use. Tubular epithelial cell deletion following cisplatin treatment is a major cause of renal injury. Oxidative stress significantly contributes to cisplatin-associated cytotoxicity, and use of antioxidants could counteract such cytotoxic effects of cisplatin. The renal microenvironmental changes following cisplatin treatment is a complex process and could be broadly categorized into three main pathological events, which at times might overlap: initial cytotoxic events, inflammatory events and fibroproliferative events. Stress responses and heat shock proteins generated following cisplatin treatment are actively involved in the initiation and progression of these events. In this article, we will briefly summarize factors involved in various phases of cisplatin-induced renal injuries.
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Cited by
Yousef, M. I., & Hussien, H. M. (2015) · Food and Chemical Toxicology · doi:10.1016/j.fct.2015.01.014 · Cited by 104
Nutlin-3 protects kidney cells during cisplatin therapy by suppressing Bax/Bak activation
Jiang, M., Pabla, N., Murphy, R. F., Yang, T., Yin, X.-M., Degenhardt, K., White, E., & Dong, Z. (2007) · Journal of Biological Chemistry · doi:10.1074/jbc.m606928200 · Cited by 97
Metadata
- Authors
- Taguchi, T., Nazneen, A., Abid, M. R., & Razzaque, M. S.
- Venue
- Contributions to Nephrology
- Publisher
- KARGER
- Published
- 2005
- Volume
- 148
- Pages
- 107-121
- OpenAlex
- W2099252920
- Cited by
- 256
- References
- 53
- FWCI
- 58.07
- Type
- book chapter
- Language
- en
- Open access
- closed
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