The DOI matches its Crossref record; 59 of the 59 references it cites were found in a registry or an index.
Stress-Related Cardiomyopathy Syndromes
Open accessAbstract
T he relationship between the heart and the brain is complex and integral in the maintenance of normal cardiovascular function.Certain pathological conditions can interfere with the normal brain-heart regulatory mechanisms and result in impaired cardiovascular function.The mechanisms through which the central and autonomic nervous systems regulate the heart and the manner in which their impairment adversely affects cardiovascular function have recently been reviewed by Samuels. 1 The purpose of this article is to provide an up-to-date review of the clinical presentation of the stress-related cardiomyopathy syndromes, discuss possible causal mechanisms, and highlight the similarities and differences between them.[2][3][4][5][6][7][8][9][10][11][12][13][14][15][16] The stress-related cardiomyopathies appear similar in that they seemingly occur during times of enhanced sympathetic tone and may be precipitated in part or entirely by excessive endogenous or exogenous catecholamine stimulation of the myocardium.Although significant clinical overlap exists in those presenting with stress-associated cardiomyopathy, it is unclear whether myocardial adrenergic hyperstimulation is the only pathophysiological mechanism responsible for these syndromes (Tables 1 and 2).
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Neurohumoral Features of Myocardial Stunning Due to Sudden Emotional Stress
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Profound but Reversible Myocardial Depression in Patients with Septic Shock
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Apical ballooning syndrome or takotsubo cardiomyopathy: a systematic review
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Self-citationMechanical Factors in Arterial Aging
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[Myocardial stunning due to simultaneous multivessel coronary spasms: a review of 5 cases].
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Acute and Reversible Cardiomyopathy Provoked by Stress in Women From the United States
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Cited by
2014 AHA/ACC Guideline for the Management of Patients With Non–ST-Elevation Acute Coronary Syndromes
Amsterdam, E. A., Wenger, N. K., Brindis, R. G., Casey, D. E., Ganiats, T. G., Holmes, D. R., Jaffe, A. S., Jneid, H., Kelly, R. F., Kontos, M. C., et al. (2014) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2014.09.017 · Cited by 3,595
Clinical Features and Outcomes of Takotsubo (Stress) Cardiomyopathy
Templin, C., Ghadri, J. R., Diekmann, J., Napp, L. C., Bataiosu, D. R., Jaguszewski, M., Cammann, V. L., Sarcon, A., Geyer, V., Neumann, C. A., et al. (2015) · New England Journal of Medicine · doi:10.1056/nejmoa1406761 · Cited by 2,595
Self-citationAmsterdam, E. A., Wenger, N. K., Brindis, R. G., Casey, D. E., Ganiats, T. G., Holmes, D. R., Jaffe, A. S., Jneid, H., Kelly, R. F., Kontos, M. C., et al. (2014) · Circulation · doi:10.1161/cir.0000000000000133 · Cited by 1,254
Amsterdam, E. A., Wenger, N. K., Brindis, R. G., Casey, D. E., Ganiats, T. G., Holmes, D. R., Jaffe, A. S., Jneid, H., Kelly, R. F., Kontos, M. C., et al. (2014) · Circulation · doi:10.1161/cir.0000000000000134 · Cited by 1,059
Pathophysiology of Takotsubo Syndrome
Pelliccia, F., Kaski, J. C., Crea, F., & Camici, P. G. (2017) · Circulation · doi:10.1161/circulationaha.116.027121 · Cited by 732
Metadata
- Authors
- Bybee, K. A., & Prasad, A.
- Venue
- Circulation
- Published
- 2008-07-22
- Volume
- 118
- Issue
- 4
- Pages
- 397-409
- ISSN
- 0009-7322 (ISSN-L)
- 1524-4539
- OpenAlex
- W2115070464
- Cited by
- 577
- References
- 59
- FWCI
- 26.62
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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