What does Stat3 do?
Integrity signals
Registry
Cites
Statistics
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
Phosphorylation of STAT3 Ser727 by serine/threonine kinases such as MAPKs enhances STAT3 transcriptional activity.
“whereas the phosphorylation of Ser727 by serine/threonine kinases such as the MAPKs (mitogen-activated protein kinases) enhances STAT3 transcriptional activity [5,6].”
From Selective STAT3-α or -β expression reveals spliceform-specific phosphorylation kinetics, nuclear retention and distinct gene expression outcomes (2012) · cites this paper as [5]
Phosphorylation of STAT3 Tyr705 by JAKs is critical for STAT3 dimerization and subsequent cytokine-stimulated nuclear translocation.
“the phosphorylation of STAT3 Tyr705 by JAKs (Janus kinases) is critical for STAT3 dimerization and subsequent cytokine-stimulated nuclear translocation”
From Selective STAT3-α or -β expression reveals spliceform-specific phosphorylation kinetics, nuclear retention and distinct gene expression outcomes (2012) · cites this paper as [5]
Activation of STAT3 requires phosphorylation of Tyr705 and Ser727.
“This activation of STAT3 requires the phosphorylation of Tyr705 and Ser727.”
From Selective STAT3-α or -β expression reveals spliceform-specific phosphorylation kinetics, nuclear retention and distinct gene expression outcomes (2012) · cites this paper as [5]
The citation lists haven’t been fetched yet
References 46 · Cited by 734
- Authors
- Levy, D. E., & Lee, C.-K.
- Affiliations
- NYU Langone Health (US)
- Corresponding author
- Levy, David E.
- Venue
- Journal of Clinical Investigation
- Published
- 2002-05-01
- Volume
- 109
- Issue
- 9
- Pages
- 1143-1148
- ISSN
- 0021-9738 (ISSN-L)
- OpenAlex
- W4249713142
- Cited by
- 734
- References
- 46
- Citations by year
- 2026: 8 · 2025: 24 · 2024: 25 · 2023: 21 · 2022: 35 · 2021: 33 · 2020: 37 · 2019: 37 · 2018: 34 · 2017: 30 · 2016: 34 · 2015: 32 · 2014: 34 · 2013: 28 · 2012: 28
- FWCI
- 12.22
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Journal of Clinical Investigation (publishedVersion)
- Funders
- American Heart Association
- National Institutes of Health
- Topics
- Health Sciences › Medicine › Oncology › Cytokine Signaling Pathways and Interactions
- Life Sciences › Immunology and Microbiology › Immunology › Psoriasis: Treatment and Pathogenesis
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Cancer Research › NF-κB Signaling Pathways
- Keywords
- STAT3, transcription factors, signal transduction, cytokine signaling, gene expression regulation
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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