The DOI matches its Crossref record; 30 of the 30 references it cites were found in a registry or an index.
Key stages in mammary gland development - Involution: apoptosis and tissue remodelling that convert the mammary gland from milk factory to a quiescent organ
Open accessAbstract
Involution of the mammary gland is an essential process that removes the milk-producing epithelial cells when they become redundant at weaning. It is a two-step process that involves the death of the secretory epithelium and its replacement by adipocytes. During the first phase, remodelling is inhibited and apoptotic cells can be seen in the lumena of the alveoli. In the second phase, apoptosis is accompanied by remodelling of the surrounding stroma and re-differentiation of the adipocytes. Considerable effort has been directed towards understanding the molecular mechanisms of the involution process and this has resulted in the identification of the principal signalling pathways involved.
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics
Kerr, J. F. R., Wyllie, A. H., & Currie, A. R. (1972) · British Journal of Cancer · doi:10.1038/bjc.1972.33 · Cited by 15,725
Danial, N. N., & Korsmeyer, S. J. (2004) · Cell · doi:10.1016/s0092-8674(04)00046-7 · Cited by 4,651
Teglund, S., McKay, C., Schuetz, E., van Deursen, J. M., Stravopodis, D., Wang, D., Brown, M., Bodner, S., Grosveld, G., & Ihle, J. N. (1998) · Cell · doi:10.1016/s0092-8674(00)81444-0 · Cited by 1,267
Lund, L. R., Rømer, J., Thomasset, N., Solberg, H., Pyke, C., Bissell, M. J., Danø, K., & Werb, Z. (1996) · Development · doi:10.1242/dev.122.1.181 · Cited by 546
Chapman, R. S., Lourenco, P. C., Tonner, E., Flint, D. J., Selbert, S., Takeda, K., Akira, S., Clarke, A. R., & Watson, C. J. (1999) · Genes & Development · doi:10.1101/gad.13.19.2604 · Cited by 479
Self-citationStein, T., Morris, J. S., Davies, C. R., Weber-Hall, S. J., Duffy, M.-A., Heath, V. J., Bell, A. K., Ferrier, R. K., Sandilands, G. P., & Gusterson, B. A. (2003) · Breast Cancer Research · doi:10.1186/bcr753 · Cited by 388
Li, M., Liu, X., Robinson, G., Bar-Peled, U., Wagner, K.-U., Young, W. S., Hennighausen, L., & Furth, P. A. (1997) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.94.7.3425 · Cited by 375
Clarkson, R. W., Wayland, M. T., Lee, J., Freeman, T., & Watson, C. J. (2003) · Breast Cancer Research · doi:10.1186/bcr754 · Cited by 335
Self-citationRequirement of basement membrane for the suppression of programmed cell death in mammary epithelium
Pullan, S., Wilson, J., Metcalfe, A., Edwards, G. M., Goberdhan, N., Tilly, J., Hickman, J. A., Dive, C., & Streuli, C. H. (1996) · Journal of Cell Science · doi:10.1242/jcs.109.3.631 · Cited by 300
Feng, Z., Marti, A., Jehn, B., Altermatt, H. J., Chicaiza, G., & Jaggi, R. (1995) · The Journal of cell biology · doi:10.1083/jcb.131.4.1095 · Cited by 239
Cited by
Anatomical, Physiological, and Functional Diversity of Adipose Tissue
Zwick, R. K., Guerrero-Juarez, C. F., Horsley, V., & Plikus, M. V. (2018) · Cell Metabolism · doi:10.1016/j.cmet.2017.12.002 · Cited by 527
Distinct functions of AKT isoforms in breast cancer: a comprehensive review
Hinz, N., & Jücker, M. (2019) · Cell Communication and Signaling · doi:10.1186/s12964-019-0450-3 · Cited by 355
Sargeant, T. J., Lloyd-Lewis, B., Resemann, H. K., Ramos-Montoya, A., Skepper, J., & Watson, C. J. (2014) · Nature Cell Biology · doi:10.1038/ncb3043 · Cited by 168
Self-citation
Metadata
- Authors
- Watson, C. J.
- Venue
- Breast Cancer Research
- Published
- 2006-04
- Volume
- 8
- Issue
- 2
- Pages
- 203-203
- ISSN
- 1465-5411 (ISSN-L)
- 1465-542X
- OpenAlex
- W1537272407
- Cited by
- 268
- References
- 30
- FWCI
- 3.71
- Type
- journal article
- Language
- en
- Open access
- gold
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
How to cite
Cite CoChat for the checks on this page, and the paper for its own findings.
- This page
- CoChat. (2026). Citation check: Key stages in mammary gland development - Involution: apoptosis and tissue remodelling that convert the mammary gland from milk factory to a quiescent organ. https://app.cochat.ai/papers/10.1186/bcr1401
- The paper
- Watson, C. J. (2006). Key stages in mammary gland development - Involution: apoptosis and tissue remodelling that convert the mammary gland from milk factory to a quiescent organ. Breast Cancer Research. https://doi.org/10.1186/bcr1401
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
