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Cited by 345

The DOI matches its Crossref record; 42 of the 42 references it cites were found in a registry or an index.

Musashi: a translational regulator of cell fate

Okano, Hideyuki, Imai, Takao, Okabe, Masataka·2002·Journal of Cell Science

Abstract

Transcription is thought to have a major role in the regulation of cell fate; the importance of translational regulation in this process has been less certain. Recent findings demonstrate that translational regulation contributes to cell-fate specification. The evolutionarily conserved, neural RNA-binding protein Musashi, for example, controls neural cell fate. The protein functions in maintenance of the stem-cell state, differentiation, and tumorigenesis by repressing translation of particular mRNAs. In mammals it might play an important role in activating Notch signalling by repressing translation of the Notch inhibitor m-Numb.

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Metadata

Authors
Okano, H., Imai, T., & Okabe, M.
Venue
Journal of Cell Science
Published
2002-04-01
Volume
115
Issue
7
Pages
1355-1359
ISSN
  • 0021-9533 (ISSN-L)
  • 1477-9137
OpenAlex
W1826852120
Cited by
345
References
42
FWCI
4.97
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Musashi: a translational regulator of cell fate. https://app.cochat.ai/papers/10.1242/jcs.115.7.1355
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Okano, H., Imai, T., & Okabe, M. (2002). Musashi: a translational regulator of cell fate. Journal of Cell Science. https://doi.org/10.1242/jcs.115.7.1355

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.