The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
Low-level laser (light) therapy (LLLT) on muscle tissue: performance, fatigue and repair benefited by the power of light
Open accessAbstract
The use of low level laser (light) therapy (LLLT) has recently expanded to cover areas of medicine that were not previously thought of as the usual applications such as wound healing and inflammatory orthopedic conditions. One of these novel application areas is LLLT for muscle fatigue and muscle injury. Since it is becoming agreed that mitochondria are the principal photoacceptors present inside cells, and it is known that muscle cells are exceptionally rich in mitochondria, this suggests that LLLT should be highly beneficial in muscle injuries. The ability of LLLT to stimulate stem cells and progenitor cells means that muscle satellite cells may respond well to LLLT and help muscle repair. Furthermore the ability of LLLT to reduce inflammation and lessen oxidative stress is also beneficial in cases of muscle fatigue and injury. This review covers the literature relating to LLLT and muscles in both preclinical animal experiments and human clinical studies. Athletes, people with injured muscles, and patients with Duchenne muscular dystrophy may all benefit.
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Cites
Skeletal muscle hypertrophy and atrophy signaling pathways
Glass, D. J. (2005) · The International Journal of Biochemistry & Cell Biology · doi:10.1016/j.biocel.2005.04.018 · Cited by 1,106 · cited 2×
“Gene expression changes related to endurance training or low intensity exercises include mitochondrial biogenesis involving upregulation of CCO, nuclear respiratory factor (NRF) 1 and 2, transcription factor A mitochondrial (TFAM) and peroxisome proliferator-activated receptor gamma coactivator 1 α (PPARGC-1 α ), all…”
BackgroundPetrella, J. K., Kim, J.-S., Mayhew, D. L., Cross, J. M., & Bamman, M. M. (2008) · Journal of Applied Physiology · doi:10.1152/japplphysiol.01215.2007 · Cited by 435 · cited 1×
“Satellite cells that have already differentiated can be fused to the damaged muscle fibers as new myonuclei, providing gene expression that encodes new contractile proteins or improve the myonuclear domain [62–64].”
mTOR Signaling and the Molecular Adaptation to Resistance Exercise
BODINE, S. C. (2006) · Medicine & Science in Sports & Exercise · doi:10.1249/01.mss.0000233797.24035.35 · Cited by 171
Motor unit recruitment for dynamic tasks: current understanding and future directions
Hodson-Tole, E. F., & Wakeling, J. M. (2009) · Journal of Comparative Physiology B · doi:10.1007/s00360-008-0289-1 · Cited by 145
Global gene expression in skeletal muscle from well-trained strength and endurance athletes
STEPTO, N. K., COFFEY, V. G., CAREY, A. L., PONNAMPALAM, A. P., CANNY, B. J., POWELL, D., & HAWLEY, J. A. (2009) · Medicine & Science in Sports & Exercise · doi:10.1249/mss.0b013e31818c6be9 · Cited by 108
Cited by
Mechanisms and Mitochondrial Redox Signaling in Photobiomodulation
Hamblin, M. R. (2018) · Photochemistry and Photobiology · doi:10.1111/php.12864 · Cited by 820
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Metadata
- Authors
- Ferraresi, C., Hamblin, M. R., & Parizotto, N. A.
- Venue
- Photonics & Lasers in Medicine
- Publisher
- Walter de Gruyter GmbH
- Published
- 2012-01-01
- Volume
- 1
- Issue
- 4
- Pages
- 267-286
- ISSN
- 2193-0635 (ISSN-L)
- 2193-0643
- OpenAlex
- W2069088639
- Cited by
- 209
- References
- 5
- FWCI
- 6.45
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Ferraresi, C., Hamblin, M. R., & Parizotto, N. A. (2012). Low-level laser (light) therapy (LLLT) on muscle tissue: performance, fatigue and repair benefited by the power of light. Photonics & Lasers in Medicine. https://doi.org/10.1515/plm-2012-0032
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