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Cited by 274

The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

Osteoporosis, genetics and hormones

Open access
Rizzoli, R, Bonjour, JP, Ferrari, SL·2001·Journal of Molecular Endocrinology

Abstract

Osteoporosis is a systemic skeletal disease characterized by low bone mass and microarchitectural deterioration of bone tissue. At a given age, bone mass results from the amount of bone acquired during growth, i.e. the peak bone mass (Bonjour et al., 1991, Theintz et al. 1992) minus the age-related bone loss which particularly accelerates after menopause. The rate and magnitude of bone mass gain during the pubertal years and of bone loss in later life may markedly differ from one skeletal site to another, as well as from one individual to another. Bone mass gain is mainly related to increases in bone size, that is in bone external dimensions, with minimal changes in bone microarchitecture. In contrast, postmenopausal and age-related decreases in bone mass result from thinning of both cortices and trabeculae, from perforation and eventually disappearance of the latter, leading to significant alterations of the bone microarchitecture (Fig. 1).

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Crossref · 10.1677/jme.0.0260079

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Metadata

Authors
Rizzoli, R., Bonjour, J., & Ferrari, S.
Venue
Journal of Molecular Endocrinology
Published
2001-04-01
Volume
26
Issue
2
Pages
79-94
ISSN
  • 0952-5041 (ISSN-L)
  • 1479-6813
OpenAlex
W2114575674
Cited by
274
References
6
FWCI
12.70
Type
journal article
Language
en
Open access
bronze

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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