Cited by 110Diabetes, Cardiovascular Risks, and LipoproteinsPancreatic function and diabetesDiabetes Management and Research

Insulin sensitivity in African-American children with and without family history of type 2 diabetes.

Danadian, K, Balasekaran, G, Lewy, V, Meza, M P, Robertson, R, Arslanian, S A·1999·Diabetes Care

Abstract

OBJECTIVE: African-Americans are at increased risk for type 2 diabetes. We have previously demonstrated that African-American children are hyperinsulinemic and insulin resistant compared with their white American peers. The aim of the present investigation was to assess the impact of family history of type 2 diabetes on insulin sensitivity in African-American children. RESEARCH DESIGN AND METHODS: A total of 13 prepubertal healthy children with negative family history (FH-) and 9 with positive family history (FH+) of type 2 diabetes underwent a 3-h hyperinsulinemic (40 mU x m(-2) x min(-1))-euglycemic clamp study to assess insulin sensitivity. The groups were comparable for age, pubertal status, total body adiposity determined by dual-energy X-ray absorptiometry, abdominal adiposity assessed by computed tomography scan at the level of L4-5 lumbar vertebra, and physical fitness measured by maximal oxygen consumption (VO2max). RESULTS: The FH+, compared with the FH-, group had lower insulin-stimulated glucose disposal (10.9+/-1.2 vs. 14.2+/-0.9 mg x kg(-1) x min(-1), P = 0.035) and lower nonoxidative glucose disposal (5.7+/-0.8 vs. 8.3+/-0.6 mg x kg(-1) x min(-1), P = 0.015), with no differences in rates of glucose oxidation, fat oxidation, or insulin-mediated free fatty acid suppression. Fasting hepatic glucose production assessed with [6,6-2H2]glucose and basal rates of glucose and fat oxidation were not different between the two groups. CONCLUSIONS: These data suggest that in African-American children, family history of type 2 diabetes is a risk factor for insulin resistance. These children manifest important metabolic alterations, including impaired insulin-stimulated total and nonoxidative glucose disposal early in the first decade of life. We propose that this familial tendency, combined with environmental influences, could lead to type 2 diabetes decades later.

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Crossref · 10.2337/diacare.22.8.1325

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  • Lean healthy offspring of diabetic parents have a significant decrease in insulin-stimulated total-body and muscle glucose disposal, similar in magnitude to that in their diabetic parents.

    “Thus, lean healthy offspring of diabetic parents have a significant decrease in insulin-stimulated total body and muscle glucose disposal which is of a similar magnitude to that observed in their diabetic parents [30–32].”

    From Pathogenesis of insulin resistance in skeletal muscle (2010) · cites this paper as [31]

  • Lean normal-glucose-tolerant insulin-resistant offspring of type 2 diabetic parents have a 30-40% decrease in metabolic flux through the tricarboxylic acid cycle and oxidative phosphorylation in skeletal muscle under basal conditions.

    “Lean normal-glucose-tolerant (NGT), insulin-resistant offspring of T2DM parents have a 30–40% decrease in metabolic flux through the tricarboxylic acid cycle [104] and oxidative phosphorylation [31] in skeletal muscle under basal conditions.”

    From Pathogenesis of insulin resistance in skeletal muscle (2010) · cites this paper as [31]

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Authors
Danadian, K., Balasekaran, G., Lewy, V, Meza, M. P., Robertson, R., & Arslanian, S. A.
Affiliations
University of Pittsburgh (US)
Venue
Diabetes Care
Published
1999-08-01
Volume
22
Issue
8
Pages
1325-1329
ISSN
  • 0149-5992 (ISSN-L)
  • 1935-5548
OpenAlex
W2164206422
Cited by
110
References
0
Citations by year
2024: 2 · 2023: 2 · 2022: 2 · 2021: 1 · 2018: 1 · 2017: 2 · 2015: 3 · 2014: 7 · 2013: 1 · 2012: 5
FWCI
3.88
Type
journal article
Language
en
Open access
closed
Locations
  • Diabetes Care (publishedVersion)
  • PubMed (publishedVersion)
Funders
  • National Center for Research Resources (MO-1-RR-00084, M01RR000084)
  • Eunice Kennedy Shriver National Institute of Child Health and Human Development (HD-27503)
Topics
  • Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes, Cardiovascular Risks, and Lipoproteins
  • Health Sciences › Medicine › Surgery › Pancreatic function and diabetes
  • Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes Management and Research
Keywords
insulin sensitivity, insulin resistance, type 2 diabetes, family history of diabetes, African American children, hyperinsulinemic-euglycemic clamp, glucose disposal, nonoxidative glucose disposal, hepatic glucose production, fat oxidation, prepubertal children, diabetes risk factors
MeSH terms
Basal Metabolism, Black or African American, Child, Diabetes Mellitus, Type 2, Family Health, Female, Humans, Hyperinsulinism, Insulin Resistance, Male, Medical History Taking, Glucose Clamp Technique, Black People

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