The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
B and T Lymphocytes Are the Primary Sources of RANKL in the Bone Resorptive Lesion of Periodontal Disease
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Citations
Cites
Osteoclast differentiation and activation
Boyle, W. J., Simonet, W. S., & Lacey, D. L. (2003) · Nature · doi:10.1038/nature01658 · Cited by 6,670 · cited 1×
“RANKL expressed by T cells, as well as by osteoblasts and bone marrow stromal cells, triggers signaling in osteoclast precursor cells that elicits the differentiation into their mature form.(8) OPG is expressed ubiquitously by many types of cells and tissues, and it counterregulates”
BackgroundOPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph node organogenesis.
Kong, Y.-Y., Yoshida, H., Sarosi, I., Tan, H.-L., Timms, E., Capparelli, C., Morony, S., Oliveira-dos-Santos, A. J., Van, G., Itie, A., et al. (1999) · Nature · doi:10.1038/16852 · Cited by 3,351
Kong, Y.-Y., Feige, U., Sarosi, I., Bolon, B., Tafuri, A., Morony, S., Capparelli, C., Li, J., Elliott, R., McCabe, S., et al. (1999) · Nature · doi:10.1038/46303 · Cited by 1,856
Hofbauer, L. C., Gori, F., Riggs, B. L., Lacey, D. L., Dunstan, C. R., Spelsberg, T. C., & Khosla, S. (1999) · Endocrinology · doi:10.1210/endo.140.10.7034 · Cited by 794
Severe osteoporosis in mice lacking osteoclastogenesis inhibitory factor/osteoprotegerin.
Mizuno, A., Amizuka, N., Irie, K., Murakami, A., Fujise, N., Kanno, T., Sato, Y., Nakagawa, N., Yasuda, H., Mochizuki, S.-I., et al. (1998) · Biochemical and Biophysical Research Communications · doi:10.1006/bbrc.1998.8697 · Cited by 775
Cited by
Inflammatory and immune pathways in the pathogenesis of periodontal disease
Cekici, A., Kantarci, A., Hasturk, H., & Van Dyke, T. E. (2014) · Periodontology 2000 · doi:10.1111/prd.12002 · Cited by 1,415
“It is now clear that RANKL and osteoprotegerin are the key regulators of bone remodeling and are directly in- volved in the differentiation, activation and survival of osteoclasts and osteoclast precursors (2, 129, 261).”
BackgroundInfluentialInflammation and Bone Loss in Periodontal Disease
Cochran, D. L. (2008) · Journal of Periodontology · doi:10.1902/jop.2008.080233 · Cited by 732
Garlet, G. P. (2010) · Journal of Dental Research · doi:10.1177/0022034510376402 · Cited by 720
Li, Y., Toraldo, G., Li, A., Yang, X., Zhang, H., Qian, W.-P., & Weitzmann, M. N. (2007) · Blood · doi:10.1182/blood-2006-07-037994 · Cited by 520
Self-citationBasic biology and role of interleukin‐17 in immunity and inflammation
Zenobia, C., & Hajishengallis, G. (2015) · Periodontology 2000 · doi:10.1111/prd.12083 · Cited by 470
Metadata
- Authors
- Kawai, T., Matsuyama, T., Hosokawa, Y., Makihira, S., Seki, M., Karimbux, N. Y., Goncalves, R. B., Valverde, P., Dibart, S., Li, Y.-P., Miranda, L. A., Ernst, C. W. O., Izumi, Y., & Taubman, M. A.
- Venue
- The American Journal of Pathology
- Publisher
- Elsevier BV
- Published
- 2006-09
- Volume
- 169
- Issue
- 3
- Pages
- 987-998
- ISSN
- 0002-9440 (ISSN-L)
- OpenAlex
- W2090543816
- Cited by
- 565
- References
- 5
- FWCI
- 7.61
- Type
- journal article
- Language
- en
- Open access
- bronze
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.