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Cited by 231

The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.

Cutting Edge: CD7 Delivers a Pro-Apoptotic Signal During Galectin-1-Induced T Cell Death

Open access
Pace, Karen E, Hahn, Hejin P, Pang, Mabel, Nguyen, Julie T, Baum, Linda G·2000·The Journal of Immunology

Abstract

Galectin-1, an endogenous lectin expressed in lymphoid organs and immune-privileged sites, induces death of human and murine thymocytes and T cells. Galectin-1 binds to several glycoproteins on the T cell surface, including CD7. However, the T cell surface glycoprotein receptors responsible for delivering the galectin-1 death signal have not been identified. We show that CD7 is required for galectin-1-mediated death. This demonstrates a novel function for CD7 as a death trigger and identifies galectin-1/CD7 as a new biologic death signaling pair.

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Crossref · 10.4049/jimmunol.165.5.2331

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Metadata

Authors
Pace, K. E., Hahn, H. P., Pang, M., Nguyen, J. T., & Baum, L. G.
Venue
The Journal of Immunology
Published
2000-09-01
Volume
165
Issue
5
Pages
2331-2334
ISSN
  • 0022-1767 (ISSN-L)
  • 1550-6606
OpenAlex
W1874098526
Cited by
231
References
1
FWCI
4.95
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Cutting Edge: CD7 Delivers a Pro-Apoptotic Signal During Galectin-1-Induced T Cell Death. https://app.cochat.ai/papers/10.4049/jimmunol.165.5.2331
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Pace, K. E., Hahn, H. P., Pang, M., Nguyen, J. T., & Baum, L. G. (2000). Cutting Edge: CD7 Delivers a Pro-Apoptotic Signal During Galectin-1-Induced T Cell Death. The Journal of Immunology. https://doi.org/10.4049/jimmunol.165.5.2331

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.