Gerencser, Akos A.

Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype

Wiley, C. D., Velarde, M. C., Lecot, P., Liu, S., Sarnoski, E. A., Freund, A., Shirakawa, K., Lim, H. W., Davis, S. S., Ramanathan, A., et al. (2016) · Cell Metabolism

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 1,275

Suppressors of Superoxide-H 2 O 2 Production at Site I Q of Mitochondrial Complex I Protect against Stem Cell Hyperplasia and Ischemia-Reperfusion Injury

Brand, M. D., Goncalves, R. L. S., Orr, A. L., Vargas, L., Gerencser, A. A., Borch Jensen, M., Wang, Y. T., Melov, S., Turk, C. N., Matzen, J. T., et al. (2016) · Cell Metabolism

Mitochondrial Function and PathologyRedox biology and oxidative stress

Cited by 228

The mechanism of superoxide production by the antimycin-inhibited mitochondrial Q-cycle

Quinlan, C. L., Gerencser, A. A., Treberg, J. R., & Brand, M. D. (2011) · Journal of Biological Chemistry

Mitochondrial Function and PathologyCoenzyme Q10 studies and effects

Cited by 200

Spatial mapping of cellular senescence: emerging challenges and opportunities

Gurkar, A. U., Gerencser, A. A., Mora, A. L., Nelson, A. C., Zhang, A. R., Lagnado, A. B., Enninful, A., Benz, C., Furman, D., Beaulieu, D., et al. (2023) · Nature Aging

Telomeres, Telomerase, and SenescenceSingle-cell and spatial transcriptomics

Cited by 141

NIH SenNet Consortium to map senescent cells throughout the human lifespan to understand physiological health.

SenNet Consortium, Writing Group, Lee, P. J., Benz, C. C., Blood, P., Börner, K., Campisi, J., Chen, F., Daldrup-Link, H., De Jager, P., et al. (2022) · Nature Aging

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 122

Forward operation of adenine nucleotide translocase during F 0 F 1 ‐ATPase reversal: critical role of matrix substrate‐level phosphorylation

Chinopoulos, C., Gerencser, A. A., Mandi, M., Mathe, K., Töröcsik, B., Doczi, J., Turiak, L., Kiss, G., Konràd, C., Vajda, S., et al. (2010) · The FASEB Journal

Mitochondrial Function and PathologyATP Synthase and ATPases Research

Cited by 103

Plasma Membrane Potential Oscillations in Insulin Secreting Ins-1 832/13 Cells Do Not Require Glycolysis and Are Not Initiated by Fluctuations in Mitochondrial Bioenergetics

Goehring, I., Gerencser, A. A., Schmidt, S., Brand, M. D., Mulder, H., & Nicholls, D. G. (2012) · Journal of Biological Chemistry

Pancreatic function and diabetesMitochondrial Function and Pathology

Cited by 42

A Fully-Automated Senescence Test (FAST) for the high-throughput quantification of senescence-associated markers

Neri, F., Takajjart, S. N., Lerner, C. A., Desprez, P.-Y., Schilling, B., Campisi, J., & Gerencser, A. A. (2024) · GeroScience

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 17

Senescent cell heterogeneity and responses to senolytic treatment are related to cell cycle status during senescence induction

Neri, F., Zheng, S., Watson, M. A., Desprez, P.-Y., Gerencser, A. A., Campisi, J., Wirtz, D., Wu, P.-H., & Schilling, B. (2025) · Aging

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 6