Mittal, Vivek

Epithelial-to-mesenchymal transition is not required for lung metastasis but contributes to chemoresistance

Fischer, K. R., Durrans, A., Lee, S., Sheng, J., Li, F., Wong, S. T. C., Choi, H., El Rayes, T., Ryu, S., Troeger, J., et al. (2015) · Nature

Cancer Cells and MetastasisTGF-β signaling in diseases

Cited by 1,791

Epithelial Mesenchymal Transition in Tumor Metastasis

Mittal, V. (2018) · Annual Review of Pathology: Mechanisms of Disease

Cancer Cells and MetastasisTGF-β signaling in diseases

Cited by 1,528

Endothelial Progenitor Cells Control the Angiogenic Switch in Mouse Lung Metastasis

Gao, D., Nolan, D. J., Mellick, A. S., Bambino, K., McDonnell, K., & Mittal, V. (2008) · Science

Angiogenesis and VEGF in CancerCancer Cells and Metastasis

Cited by 632

Rb inactivation promotes genomic instability by uncoupling cell cycle progression from mitotic control

Hernando, E., Nahlé, Z., Juan, G., Diaz-Rodriguez, E., Alaminos, M., Hemann, M., Michel, L., Mittal, V., Gerald, W., Benezra, R., et al. (2004) · Nature

Microtubule and mitosis dynamicsCancer-related Molecular Pathways

Cited by 562

TGF-β IL-6 axis mediates selective and adaptive mechanisms of resistance to molecular targeted therapy in lung cancer

Yao, Z., Fenoglio, S., Gao, D. C., Camiolo, M., Stiles, B., Lindsted, T., Schlederer, M., Johns, C., Altorki, N., Mittal, V., et al. (2010) · Proceedings of the National Academy of Sciences

Lung Cancer Treatments and MutationsTGF-β signaling in diseases

Cited by 389

Adipogenesis is inhibited by brief, daily exposure to high-frequency, extremely low-magnitude mechanical signals

Rubin, C. T., Capilla, E., Luu, Y. K., Busa, B., Crawford, H., Nolan, D. J., Mittal, V., Rosen, C. J., Pessin, J. E., & Judex, S. (2007) · Proceedings of the National Academy of Sciences

Adipokines, Inflammation, and Metabolic DiseasesAdipose Tissue and Metabolism

Cited by 274