Anderson, E A

Hyperinsulinemia produces both sympathetic neural activation and vasodilation in normal humans.

Anderson, E. A., Hoffman, R. P., Balon, T. W., Sinkey, C. A., & Mark, A. L. (1991) · Journal of Clinical Investigation

Blood Pressure and Hypertension StudiesDiabetes, Cardiovascular Risks, and Lipoproteins

Cited by 1,075

Elevated sympathetic nerve activity in borderline hypertensive humans. Evidence from direct intraneural recordings.

Anderson, E. A., Sinkey, C. A., Lawton, W. J., & Mark, A. L. (1989) · Hypertension

Blood Pressure and Hypertension StudiesHeart Rate Variability and Autonomic Control

Cited by 632

Reduced sympathetic nervous activity. A potential mechanism predisposing to body weight gain.

Spraul, M., Ravussin, E., Fontvieille, A. M., Rising, R., Larson, D. E., & Anderson, E. A. (1993) · Journal of Clinical Investigation

Regulation of Appetite and ObesityObesity, Physical Activity, Diet

Cited by 293

The vasodilator action of insulin. Implications for the insulin hypothesis of hypertension.

Anderson, E. A., & Mark, A. L. (1993) · Hypertension

Blood Pressure and Hypertension StudiesNitric Oxide and Endothelin Effects

Cited by 242

Dissociation of sympathoexcitatory and vasodilator actions of modestly elevated plasma insulin levels

Hausberg, M., Mark, A. L., Hoffman, R. P., Sinkey, C. A., & Anderson, E. A. (1995) · Journal of Hypertension

Diabetes, Cardiovascular Risks, and LipoproteinsBlood Pressure and Hypertension Studies

Cited by 90

Evaluative judgments of aspects of life as a function of vicarious exposure to hedonic extremes

Dermer, M., Cohen, S. J., Jacobsen, E., & Anderson, E. A. (1979) · Journal of Personality and Social Psychology

Psychological Well-being and Life SatisfactionMental Health via Writing

Cited by 67