Martin, T. John

Control of mammary stem cell function by steroid hormone signalling

Asselin-Labat, M.-L., Vaillant, F., Sheridan, J. M., Pal, B., Wu, D., Simpson, E. R., Yasuda, H., Smyth, G. K., Martin, T. J., Lindeman, G. J., et al. (2010) · Nature

Estrogen and related hormone effectsCancer Cells and Metastasis

Cited by 693

Catabolic effects of continuous human PTH (1-38) in vivo is associated with sustained stimulation of RANKL and inhibition of osteoprotegerin and gene-associated bone formation.

Ma, Y. L., Cain, R. L., Halladay, D. L., Yang, X., Zeng, Q., Miles, R. R., Chandrasekhar, S., Martin, T. J., & Onyia, J. E. (2001) · Endocrinology

Bone Metabolism and DiseasesBone health and osteoporosis research

Cited by 428

A combination of osteoclast differentiation factor and macrophage-colony stimulating factor is sufficient for both human and mouse osteoclast formation in vitro.

Quinn, J. M. W., Elliott, J., Gillespie, M. T., & Martin, T. J. (1998) · Endocrinology

Bone Metabolism and DiseasesOsteomyelitis and Bone Disorders Research

Cited by 407

Osteoprotegerin Reduces Osteoclast Numbers and Prevents Bone Erosion in Collagen-Induced Arthritis

Romas, E., Sims, N. A., Hards, D. K., Lindsay, M., Quinn, J. W. M., Ryan, P. F. J., Dunstan, C. R., Martin, T. J., & Gillespie, M. T. (2002) · The American Journal of Pathology

Bone Metabolism and DiseasesRheumatoid Arthritis Research and Therapies

Cited by 368

Bone as an endocrine organ

Fukumoto, S., & Martin, T. J. (2009) · Trends in Endocrinology & Metabolism

Parathyroid Disorders and TreatmentsBone Metabolism and Diseases

Cited by 311

Transforming Growth Factor β Affects Osteoclast Differentiation via Direct and Indirect Actions

Quinn, J. M. W., Itoh, K., Udagawa, N., Häusler, K., Yasuda, H., Shima, N., Mizuno, A., Higashio, K., Takahashi, N., Suda, T., et al. (2001) · Journal of Bone and Mineral Research

Bone Metabolism and DiseasesTGF-β signaling in diseases

Cited by 278

Bone Morphogenetic Protein 2 Stimulates Osteoclast Differentiation and Survival Supported by Receptor Activator of Nuclear Factor-κB Ligand

Itoh, K., Udagawa, N., Katagiri, T., Iemura, S., Ueno, N., Yasuda, H., Higashio, K., Quinn, J. M. W., Gillespie, M. T., Martin, T. J., et al. (2001) · Endocrinology

Bone Metabolism and DiseasesTGF-β signaling in diseases

Cited by 250

THE MODE OF ACTION OF THYROCALCITONIN

Martin, T. J., Robinson, C. J., & Macintyre, I. (1966) · The Lancet

Thyroid Disorders and TreatmentsParathyroid Disorders and Treatments

Cited by 115

Calcitonin gene-related peptide (CGRP) acts independently of calcitonin on cyclic AMP formation in clonal osteogenic sarcoma cells (UMR 106-01)

Michelangeli, V. P., Findlay, D. M., Fletcher, A., & Martin, T. J. (1986) · Calcified Tissue International

Neuropeptides and Animal PhysiologyBone Metabolism and Diseases

Cited by 49

Isolation and gene expression of haematopoietic-cell-free preparations of highly purified murine osteocytes

Chia, L. Y., Walsh, N. C., Martin, T. J., & Sims, N. A. (2015) · Bone

Bone Metabolism and DiseasesBone and Dental Protein Studies

Cited by 42

Effects of continuous calcitonin treatment on osteoclast-like cell development and calcitonin receptor expression in mouse bone marrow cultures

Ikegame, M., Rakopoulos, M., Martin, T. J., Moseley, J. M., & Dr. Findlay, D. M. (1996) · Journal of Bone and Mineral Research

Bone Metabolism and DiseasesBone health and osteoporosis research

Cited by 36

Calcitonin

Findlay, D. M., Sexton, P. M., & Martin, T. J. (2016) · Endocrinology: Adult and Pediatric

Parathyroid Disorders and TreatmentsBone health and osteoporosis research

Cited by 2