Thomson, David M.
Brigham Young University
10 papers on CoChatORCID 0000-0002-3318-7137
Most cited papers
- Verified
The Role of AMPK in the Regulation of Skeletal Muscle Size, Hypertrophy, and Regeneration
Thomson, D. M. (2018) · International Journal of Molecular Sciences
Muscle Physiology and DisordersMetabolism, Diabetes, and Cancer
Cited by 267
- Verified
AMPK activation attenuates S6K1, 4E-BP1, and eEF2 signaling responses to high-frequency electrically stimulated skeletal muscle contractions
Thomson, D. M., Fick, C. A., & Gordon, S. E. (2008) · Journal of Applied Physiology
Metabolism, Diabetes, and CancerMuscle metabolism and nutrition
Cited by 153
- Verified
Sestrin prevents atrophy of disused and aging muscles by integrating anabolic and catabolic signals
Segalés, J., Perdiguero, E., Serrano, A. L., Sousa-Victor, P., Ortet, L., Jardí, M., Budanov, A. V., Garcia-Prat, L., Sandri, M., Thomson, D. M., et al. (2020) · Nature Communications
Muscle Physiology and DisordersNutrition and Health in Aging
Cited by 153
- Verified
Skeletal muscle and heart LKB1 deficiency causes decreased voluntary running and reduced muscle mitochondrial marker enzyme expression in mice.
Thomson, D. M., Porter, B. B., Tall, J. H., Kim, H.-J., Barrow, J. R., & Winder, W. W. (2007) · American Journal of Physiology-Endocrinology and Metabolism
Metabolism, Diabetes, and CancerMuscle Physiology and Disorders
Cited by 146
- Verified
Diminished overload-induced hypertrophy in aged fast-twitch skeletal muscle is associated with AMPK hyperphosphorylation
Thomson, D. M., & Gordon, S. E. (2005) · Journal of Applied Physiology
Muscle Physiology and DisordersMetabolism, Diabetes, and Cancer
Cited by 131
- Verified
β-Hydroxybutyrate Elicits Favorable Mitochondrial Changes in Skeletal Muscle
Parker, B. A., Walton, C. M., Carr, S. T., Andrus, J. L., Cheung, E. C. K., Duplisea, M. J., Wilson, E. K., Draney, C., Lathen, D. R., Kenner, K. B., et al. (2018) · International Journal of Molecular Sciences
Diet and metabolism studiesMitochondrial Function and Pathology
Cited by 52
- Verified
Mitochondrial and performance adaptations to exercise training in mice lacking skeletal muscle LKB1
Tanner, C. B., Madsen, S. R., Hallowell, D. M., Goring, D. M. J., Moore, T. M., Hardman, S. E., Heninger, M. R., Atwood, D. R., & Thomson, D. M. (2013) · American Journal of Physiology-Endocrinology and Metabolism
Cardiovascular and exercise physiologyAdipose Tissue and Metabolism
Cited by 47
- Verified
Skeletal muscle dysfunction in muscle-specific LKB1 knockout mice.
Thomson, D. M., Hancock, C. R., Evanson, B. G., Kenney, S. G., Malan, B. B., Mongillo, A. D., Brown, J. D., Hepworth, S., Fillmore, N., Parcell, A. C., et al. (2010) · Journal of Applied Physiology
Muscle Physiology and DisordersMetabolism, Diabetes, and Cancer
Cited by 45
- Verified
16p11 Duplication disrupts hippocampal-orbitofrontal-amygdala connectivity, revealing a neural circuit endophenotype for schizophrenia
Bristow, G. C., Thomson, D. M., Openshaw, R. L., Mitchell, E. J., Pratt, J. A., Dawson, N., & Morris, B. J. (2020) · Cell Reports
Schizophrenia research and treatmentGenomic variations and chromosomal abnormalities
Cited by 36
- Verified
Liver kinase B1 inhibits the expression of inflammation-related genes postcontraction in skeletal muscle.
Chen, T., Moore, T. M., Ebbert, M. T. W., McVey, N. L., Madsen, S. R., Hallowell, D. M., Harris, A. M., Char, R. E., Mackay, R. P., Hancock, C. R., et al. (2016) · Journal of Applied Physiology
Exercise and Physiological ResponsesMuscle Physiology and Disorders
Cited by 14
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