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Cited by 615

The DOI matches its Crossref record; 89 of the 89 references it cites were found in a registry or an index.

Tumor resistance to apoptosis

Open access
Fulda, Simone·2009·International Journal of Cancer

Abstract

One of the hallmarks of human cancers is the intrinsic or acquired resistance to apoptosis. Evasion of apoptosis may contribute to carcinogenesis, tumor progression and also to treatment resistance, since most current anticancer therapies including chemotherapy, radio‐ and immunotherapy primarily act by activating cell death pathways including apoptosis in cancer cells. Hence, a better understanding of the molecular mechanisms underlying tumor resistance to apoptotic cell death is expected to provide the basis for a rational approach to develop molecular targeted therapies. © 2008 Wiley‐Liss, Inc.

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Crossref · 10.1002/ijc.24064

Cites

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Citations

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Metadata

Authors
Fulda, S.
Venue
International Journal of Cancer
Publisher
Wiley
Published
2008-11-11
Volume
124
Issue
3
Pages
511-515
ISSN
  • 0020-7136 (ISSN-L)
  • 1097-0215
OpenAlex
W2065177893
Cited by
615
References
89
FWCI
8.19
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Tumor resistance to apoptosis. https://app.cochat.ai/papers/10.1002/ijc.24064
The paper
Fulda, S. (2009). Tumor resistance to apoptosis. International Journal of Cancer. https://doi.org/10.1002/ijc.24064

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.