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Peripheral reduction of β-amyloid is sufficient to reduce brain β-amyloid: Implications for Alzheimer's disease

Sutcliffe, J. Gregor, Hedlund, Peter B., Thomas, Elizabeth A., Bloom, Floyd E., Hilbush, Brian S.·2011·Journal of Neuroscience Research

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Research works printed but not linked: 26

  1. Barten, 2008, Therapeutic strategies for Alzheimer's disease, Mol Neurobiol, 37, 171, 10.1007/s12035-008-8031-2 · DOI
  2. Bassez, 2004, Pleiotropic and diverse expression of ZFHX1B gene transcripts during mouse and human development supports the various clinical manifestations of the “Mowat-Wilson” syndrome, Neurobiol Dis, 15, 240, 10.1016/j.nbd.2003.10.004 · DOI
  3. Blazejczyk, 2006, Ca2+-independent binding and cellular expression profiles question a significant role of calmyrin in transduction of Ca2+ signals to Alzheimer's disease-related presenilin 2 in forebrain, Biochim Biophys Acta, 1762, 66, 10.1016/j.bbadis.2005.09.006 · DOI
  4. Cheung, 2008, Mechanism of Ca2+ disruption in Alzheimer's disease by presenilin regulation of InsP3 receptor channel gating, Neuron, 58, 871, 10.1016/j.neuron.2008.04.015 · DOI
  5. Gatti, 2007, Genome-level analysis of genetic regulation of liver gene expression networks, Hepatology, 46, 548, 10.1002/hep.21682 · DOI
  6. He, 2010, Gamma-secretase activating protein is a therapeutic target for Alzheimer's disease, Nature, 467, 95, 10.1038/nature09325 · DOI
  7. Hustinx, 2004, Differentially expressed genes in pancreatic ductal adenocarcinomas identified through serial analysis of gene expression, Cancer Biol Ther, 3, 1254, 10.4161/cbt.3.12.1238 · DOI
  8. Ishigaki, 2002, Two novel genes, human neugrin and mouse m-neugrin, are upregulated with neuronal differentiation in neuroblastoma cells, Biochem Biophys Res Commun, 279, 526, 10.1006/bbrc.2000.3971 · DOI
  9. Isohata, 2009, Hedgehog and epithelial-mesenchymal transition signaling in normal and malignant epithelial cells of the esophagus, Int J Cancer, 125, 1212, 10.1002/ijc.24400 · DOI
  10. Kulnane, 2001, Neuropathological characterization of mutant amyloid precursor protein yeast artificial chromosome transgenic mice, Neurobiol Dis, 8, 982, 10.1006/nbdi.2001.0446 · DOI · Paper page
  11. Lehman, 2003, Genetic background regulates beta-amyloid precursor protein processing and beta-amyloid deposition in the mouse, Hum Mol Genet, 12, 2949, 10.1093/hmg/ddg322 · DOI · Paper page
  12. Netzer, 2003, Gleevec inhibits beta-amyloid production but not Notch cleavage, Proc Natl Acad Sci U S A, 100, 12444, 10.1073/pnas.1534745100 · DOI
  13. Ryman, 2008, Genetic loci modulating amyloid-beta levels in a mouse model of Alzheimer's disease, Neurobiol Aging, 29, 1190, 10.1016/j.neurobiolaging.2007.02.017 · DOI
  14. Selkoe, 2001, Alzheimer's disease: genes, proteins, and therapy, Physiol Rev, 81, 741, 10.1152/physrev.2001.81.2.741 · DOI · Paper page
  15. Stabler, 1999, A myristoylated calcium-binding protein that preferentially interacts with the Alzheimer's disease presenilin 2 protein, J Cell Biol, 145, 1277, 10.1083/jcb.145.6.1277 · DOI
  16. Takayama, 2002, Imatinib mesylate has limited activity against the central nervous system involvement of Philadelphia chromosome-positive acute lymphoblastic leukaemia due to poor penetration into cerebrospinal fluid, Br J Haematol, 119, 106, 10.1046/j.1365-2141.2002.03881.x · DOI
  17. Tanzi, 1989, Molecular genetics of Alzheimer's disease and the amyloid beta peptide precursor gene, Ann Med, 21, 91, 10.3109/07853898909149191 · DOI
  18. Terry, 2006, Alzheimer's disease and the aging brain, J Geriatr Psychiatry Neurol, 19, 125, 10.1177/0891988706291079 · DOI
  19. Verstappen, 2008, Atypical Mowat-Wilson patient confirms the importance of the novel association between ZFHX1B/SIP1 and NuRD corepressor complex, Hum Mol Genet, 17, 1175, 10.1093/hmg/ddn007 · DOI
  20. Wang, 2003, WebQTL: web-based complex trait analysis, Neuroinformatics, 1, 299, 10.1385/NI:1:4:299 · DOI
  21. Waring, 2008, Genome-wide association studies in Alzheimer disease, Arch Neurol, 65, 329, 10.1001/archneur.65.3.329 · DOI
  22. Williams, 2007, Cytokine-dependent imatinib resistance in mouse BCR-ABL +, Arf-null lymphoblastic leukemia, Genes Dev, 21, 2283, 10.1101/gad.1588607 · DOI
  23. White, 2006, CIB1, a ubiquitously expressed Ca2+-binding protein ligand of the InsP3 receptor Ca2+ release channel, J Biol Chem, 281, 20825, 10.1074/jbc.M602175200 · DOI
  24. Wolfe, 2006, The gamma-secretase complex: membrane-embedded proteolytic ensemble, Biochemistry, 45, 7931, 10.1021/bi060799c · DOI · Paper page
  25. Wolff, 2003, The CNS is a sanctuary for leukemic cells in mice receiving imatinib mesylate for Bcr/Abl-induced leukemia, Blood, 101, 5010, 10.1182/blood-2002-10-3059 · DOI
  26. Zweier, 2002, “Mowat-Wilson” syndrome with and without Hirschsprung disease is a distinct, recognizable multiple congenital anomalies-mental retardation syndrome caused by mutations in the zinc finger homeo box 1B gene, Am J Med Genet, 108, 177, 10.1002/ajmg.10226 · DOI

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Metadata

Authors
Sutcliffe, J. G., Hedlund, P. B., Thomas, E. A., Bloom, F. E., & Hilbush, B. S.
Venue
Journal of Neuroscience Research
Publisher
Wiley
Published
2011-03-03
Volume
89
Issue
6
Pages
808-814
ISSN
0360-4012
Cited by
105
References
2
Language
en

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