The DOI matches its Crossref record; 151 of the 151 references it cites were found in a registry or an index.
Peripheral nerve injury and myelination: Potential therapeutic strategies
Open accessAbstract
Traumatic peripheral nerve injury represents a major clinical and public health problem that often leads to significant functional impairment and permanent disability. Despite modern diagnostic procedures and advanced microsurgical techniques, functional recovery after peripheral nerve repair is often unsatisfactory. Therefore, there is an unmet need for new therapeutic or adjunctive strategies to promote the functional recovery in nerve injury patients. In contrast to the central nervous system, Schwann cells in the peripheral nervous system play a pivotal role in several aspects of nerve repair such as degeneration, remyelination, and axonal growth. Several non‐surgical approaches, including pharmacological, electrical, cell‐based, and laser therapies, have been employed to promote myelination and enhance functional recovery after peripheral nerve injury. This review will succinctly discuss the potential therapeutic strategies in the context of myelination following peripheral neurotrauma.
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Cited by
Peripheral Nerve Injury Treatments and Advances: One Health Perspective
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Metadata
- Authors
- Modrak, M., Talukder, M. A. H., Gurgenashvili, K., Noble, M., & Elfar, J. C.
- Venue
- Journal of Neuroscience Research
- Publisher
- Wiley
- Published
- 2019-10-13
- Volume
- 98
- Issue
- 5
- Pages
- 780-795
- ISSN
- 0360-4012 (ISSN-L)
- 1097-4547
- OpenAlex
- W2980690932
- Cited by
- 266
- References
- 151
- FWCI
- 6.27
- Type
- journal article
- Language
- en
- Open access
- green
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- Modrak, M., Talukder, M. A. H., Gurgenashvili, K., Noble, M., & Elfar, J. C. (2020). Peripheral nerve injury and myelination: Potential therapeutic strategies. Journal of Neuroscience Research. https://doi.org/10.1002/jnr.24538
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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