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Cited by 259

The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

Placenta Growth Factor

Persico, M. Graziella·2001·Vascular Morphogenesis in the Female Reproductive System

Abstract

The search for regulators of angiogenesis has led to the identification of a number of genes that, when tested in vitro or in vivo (in animal models), help to elucidate the complex phenomenon of vascular formation during development and pathologic conditions. Many new growth factors and receptors and other molecules previously thought to be specific for other systems (e.g., the nervous system) have now been found to play a role in angiogenesis and are currently under analysis. One such example is neuropilin-1 , which is expressed predominantly in developing neurons (for a review see Fujisawa and Kitsukawa, 1998). Neuropilin-1 null mutants die during embryonic development due to disorganization of nerve pathways (Kitsukawa et al, 1997) as well as several vascular defects (Kawasaki et al, 1999). These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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Crossref · 10.1007/978-1-4612-0213-4_16

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Metadata

Authors
Persico, M. G.
Venue
Vascular Morphogenesis in the Female Reproductive System
Published
2001
Pages
301-314
OpenAlex
W207250171
Cited by
259
References
3
FWCI
30.08
Type
book chapter
Language
en
Open access
closed

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CoChat. (2026). Citation check: Placenta Growth Factor. https://app.cochat.ai/papers/10.1007/978-1-4612-0213-4_16
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Persico, M. G. (2001). Placenta Growth Factor. Vascular Morphogenesis in the Female Reproductive System. https://doi.org/10.1007/978-1-4612-0213-4_16

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.