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Cited by 24Lipoproteins and Cardiovascular HealthAlzheimer's disease research and treatmentsLipid metabolism and disorders

Apolipoprotein E Mutants, Hyperlipidemia and Arteriosclerosis

Utermann, Gerd·1985·Drugs Affecting Lipid Metabolism VIII
DOI 10.1007/978-1-4613-2459-1_14
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Crossref · 10.1007/978-1-4613-2459-1_14

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2 of 2 references verified0 citation errors · 0 not checkable

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  • Utermann et al. found that genotypes with the ε2 allele had the highest and those with the ε4 allele had the lowest average serum apoE levels.

    “Utermann et al. studied the association between levels of apoE in serum and the common APOE genotypes, and found that genotypes with the ε2 allele had highest and those with the ε4 allele had lowest average levels [20,43].”

    From Plasma levels of apolipoprotein E, APOE genotype and risk of dementia and ischemic heart disease: A review (2016) · cites this paper as [43]

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Authors
Utermann, G.
Affiliations
Philipps University of Marburg (DE)
Corresponding author
Utermann, Gerd
Venue
Drugs Affecting Lipid Metabolism VIII
Publisher
Springer US
Published
1985
Volume
183
Pages
173-188
DOI
10.1007/978-1-4613-2459-1_14
OpenAlex
W22344295
Cited by
24
References
2
Citations by year
2023: 1 · 2016: 1 · 2015: 1 · 2014: 1
FWCI
1.98
Type
book chapter
Language
en
Open access
closed
Locations
  • Springer eBooks (publishedVersion)
  • PubMed (publishedVersion)
Topics
  • Health Sciences › Medicine › Surgery › Lipoproteins and Cardiovascular Health
  • Health Sciences › Medicine › Physiology › Alzheimer's disease research and treatments
  • Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Lipid metabolism and disorders
Keywords
apolipoprotein E, APOE polymorphism, apolipoprotein E isoforms, apolipoprotein E2, APOE ε4, apolipoprotein E3, hyperlipidemia, arteriosclerosis, dysbetalipoproteinemia, single base substitution, German population
MeSH terms
Amino Acid Sequence, Apolipoproteins E, Arteriosclerosis, Gene Frequency, Humans, Hyperlipoproteinemia Type III, Isoelectric Point, Mutation, Phenotype, Polymorphism, Genetic, Risk, Structure-Activity Relationship

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