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Cited by 424

The DOI matches its Crossref record; 98 of the 98 references it cites were found in a registry or an index.

Occupational asthma

Open access
Chan-Yeung, Moira·1986·Clinical Reviews in Allergy

Abstract

Many toxic compounds found in air emissions may induce bronchoconstriction. In the workplace, workers are exposed to these compounds, often in much higher concentrations. Some of these compounds act as sensitizers. Of these, some compounds induce asthma by producing specific IgE antibodies to the compound or its protein conjugate, while others induce asthma through yet unidentified immunologic mechanisms. Some compounds, when inhaled in high concentrations, act as irritants and produce bronchoconstriction probably by inducing acute airway inflammation. The latter condition is called Reactive Airways Dysfunction Syndrome (RADS) or irritant-induced asthma. Occupational asthma is an excellent model to study the pathogenesis and the natural history of adult onset asthma because the responsible agent can be identified, complete avoidance is possible, and exposure can be measured or estimated.

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Crossref · 10.1007/bf02993160

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Metadata

Authors
Chan-Yeung, M.
Venue
Clinical Reviews in Allergy
Published
1986-07
Volume
4
Issue
3
Pages
251-266
ISSN
0731-8235 (ISSN-L)
OpenAlex
W1800063825
Cited by
424
References
98
FWCI
3.28
Type
journal article
Language
en
Open access
gold

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CoChat. (2026). Citation check: Occupational asthma. https://app.cochat.ai/papers/10.1007/bf02993160
The paper
Chan-Yeung, M. (1986). Occupational asthma. Clinical Reviews in Allergy. https://doi.org/10.1007/bf02993160

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.