Accumulation of α-synuclein/NACP is a cytopathological feature common to Lewy body disease and multiple system atrophy

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Items 101–200 of 344

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2. Alpha‐synuclein pathology in the olfactory pathways of dementia patients
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3. Increased sensitivity to MPTP in human α-synuclein A30P transgenic mice
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4. Multiple System Atrophy
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5. Detecting Morphologically Distinct Oligomeric Forms of α-Synuclein
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6. α-Synuclein Inclusions in Amygdala in the Brains of Patients with the Parkinsonism-Dementia Complex of Guam
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7. Absence of inclusion body formation in the MPTP mouse model of Parkinson's disease
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8. The N terminus of α-synuclein dictates fibril formation
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9. Developmental stages of cortical Lewy bodies and their relation to axonal transport blockage in brains of patients with dementia with Lewy bodies
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10. alpha-Synuclein forms a complex with transcription factor Elk-1
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11. Vacuole-creating protein in neurodegenerative diseases in humans
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13. Review: Formation and Properties of Amyloid-like Fibrils Derived from α-Synuclein and Related Proteins
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14. Melatonin reduces the neuronal loss, downregulation of dopamine transporter, and upregulation of D2 receptor in rotenone‐induced parkinsonian rats
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15. An autopsy case of early (“minimal change”) olivopontocerebellar atrophy (multiple system atrophy-cerebellar)
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16. α-Synuclein Fibrils Exhibit Gain of Toxic Function, Promoting Tau Aggregation and Inhibiting Microtubule Assembly
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17. Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy
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18. ɑ‐Synuclein strains and the variable pathologies of synucleinopathies
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19. Exacerbated synucleinopathy in mice expressing A53T SNCA on a Snca null background
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20. Argyrophilic glial inclusions in the midbrain of patients with Parkinson's disease and diffuse Lewy body disease are immunopositive for NACP/α-synuclein
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21. Relationship Among α-Synuclein Accumulation, Dopamine Synthesis, and Neurodegeneration in Parkinson Disease Substantia Nigra
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22. Immunohistochemical comparison of α- and β-synuclein in adult rat central nervous system
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23. Role of Synucleins in Alzheimer’s Disease
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24. Immunocytochemical localization of synphilin-1, an α-synuclein-associated protein, in neurodegenerative disorders
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25. C-terminal α-synuclein immunoreactivity in structures other than Lewy bodies in neurodegenerative disorders
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26. α‐Synuclein–synaptosomal membrane interactions
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27. Fibrillogenic and Non-fibrillogenic Ensembles of SDS-bound Human α-Synuclein
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28. Parkinson's disease, dementia with Lewy bodies and multiple system atrophy are α-synucleinopathies
    M. G. Spillantini · 1999 · Parkinsonism & Related Disorders
29. Widespread occurrence of argyrophilic glial inclusions in Parkinson's disease
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30. Endogenous alpha-synuclein monomers, oligomers and resulting pathology: let’s talk about the lipids in the room
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31. α-Synuclein immunoreactivity in neuronal nuclear inclusions and neurites in multiple system atrophy
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32. Genetics of Synucleinopathies
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33. Keap1 is localized in neuronal and glial cytoplasmic inclusions in various neurodegenerative diseases
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34. Generation of Reactive Oxygen Species by Mitochondrial Complex I: Implications in Neurodegeneration
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35. α-Synuclein Accumulates in Purkinje Cells in Lewy Body Disease but not in Multiple System Atrophy
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36. Co-localization of α-synuclein and phosphorylated tau in neuronal and glial cytoplasmic inclusions in a patient with multiple system atrophy of long duration
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37. Antidepressants reduce neuroinflammatory responses and astroglial alpha‐synuclein accumulation in a transgenic mouse model of multiple system atrophy
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38. Haploinsufficiency for trkB and trkC receptors induces cell loss and accumulation of α‐synuclein in the substantia nigra
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39. Distribution of α-synuclein in the spinal cord and dorsal root ganglia in an autopsy cohort of elderly persons
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40. A novel grading scale for striatonigral degeneration (multiple system atrophy)
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41. Intercellular transfer of pathogenic α-synuclein by extracellular vesicles is induced by the lipid peroxidation product 4-hydroxynonenal
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42. Ultrastructural and biochemical classification of pathogenic tau, α-synuclein and TDP-43
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43. Expression of α-synuclein, the precursor of non-amyloid β component of Alzheimer's disease amyloid, in human cerebral blood vessels
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44. Agrin binds α-synuclein and modulates α-synuclein fibrillation
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45. Accumulation of histone deacetylase 6, an aggresome-related protein, is specific to Lewy bodies and glial cytoplasmic inclusions
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46. α-Synuclein-immunoreactive structure formation is enhanced in sympathetic ganglia of patients with multiple system atrophy
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47. Progressive striatonigral degeneration in a transgenic mouse model of multiple system atrophy: translational implications for interventional therapies
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48. α-Synuclein pathology affecting Bergmann glia of the cerebellum in patients with α-synucleinopathies
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49. Three‐Layered Structure Shared Between Lewy Bodies and Lewy Neurites—Three‐Dimensional Reconstruction of Triple‐Labeled Sections
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50. Primary progressive aphasia as the initial manifestation of corticobasal degeneration and unusual tauopathies
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51. Primary involvement of the motor area in association with the nigrostriatal pathway in multiple system atrophy: neuropathological and morphometric evaluations
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52. α-Synuclein Aggregates Interfere with Parkin Solubility and Distribution
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53. Role of TPPP/p25 on α-synuclein-mediated oligodendroglial degeneration and the protective effect of SIRT2 inhibition in a cellular model of multiple system atrophy
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54. NUB1 Suppresses the Formation of Lewy Body-Like Inclusions by Proteasomal Degradation of Synphilin-1
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55. Juvenile-onset generalized neuroaxonal dystrophy (Hallervorden-Spatz disease) with diffuse neurofibrillary and Lewy body pathology
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56. Regional Distribution of Proteinase K-Resistant α-Synuclein Correlates with Lewy Body Disease Stage
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57. Late‐onset neurodegenerative diseases—the role of protein insolubility
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58. α-Synuclein pathology in the neostriatum in Parkinson’s disease
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59. No mutation in the entire coding region of the α-synuclein gene in pathologically confirmed cases of multiple system atrophy
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61. Widespread occurrence of α‐synuclein/NACP‐immunoreactive neuronal inclusions in juvenileand adult‐onset Hallervorden–Spatz diseasewith Lewy bodies
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75. Paroxetine treatment improves motor symptoms in patients with multiple system atrophy
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76. Olfactory bulb in multiple system atrophy
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77. Alteration of Upstream Autophagy‐Related Proteins ( ULK1 , ULK2 , B eclin1, VPS34 and AMBRA1 ) in L ewy Body Disease
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78. Accumulation of HtrA2/Omi in Neuronal and Glial Inclusions in Brains With α-Synucleinopathies
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80. The -synuclein gene in multiple system atrophy
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81. Heterogeneity of Nigral and Cortical Lewy Bodies Differentiated by Amplified Triple-Labeling for Alpha-Synuclein, Ubiquitin, and Thiazin Red
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96. α-Synuclein Inclusions in Alzheimer and Lewy Body Diseases
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