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Cited by 988

The DOI matches its Crossref record; 4 of the 4 references it cites were found in a registry or an index.

Oxidative stress

Open access
Burton, Graham J., Jauniaux, Eric·2011·Best Practice & Research Clinical Obstetrics & Gynaecology
DOI 10.1016/j.bpobgyn.2010.10.016
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Abstract

Considerable evidence implicates oxidative stress in the pathophysiology of many complications of human pregnancy, and this topic has now become a major focus of both clinical and basic science research. Oxidative stress arises when the production of reactive oxygen species overwhelms the intrinsic anti-oxidant defences. Reactive oxygen species play important roles as second messengers in many intracellular signalling cascades aimed at maintaining the cell in homeostasis with its immediate environment. At higher levels, they can cause indiscriminate damage to biological molecules, leading to loss of function and even cell death. In this chapter, we will review how reactive oxygen species are generated and detoxified in the human placenta, and what roles they may play at homeostatic concentrations. We will then consider their involvement in normal placental development, and in complications ranging from miscarriage to pre-eclampsia and premature rupture of the membranes.

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Metadata

Authors
Burton, G. J., & Jauniaux, E.
Venue
Best Practice & Research Clinical Obstetrics & Gynaecology
Publisher
Elsevier BV
Published
2011-06
Volume
25
Issue
3
Pages
287-299
ISSN
1521-6934 (ISSN-L)
DOI
10.1016/j.bpobgyn.2010.10.016
OpenAlex
W4245575456
Cited by
988
References
4
FWCI
24.44
Type
journal article
Language
en
Open access
hybrid

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Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.