The DOI matches its Crossref record; 108 of the 108 references it cites were found in a registry or an index.
Vertebrate Segmentation: From Cyclic Gene Networks to Scoliosis
Open accessAbstract
The body axis of vertebrates is composed of a serial repetition of similar anatomical modules, termed segments or metameres. This particular mode of organization is especially conspicuous at the level of the periodic arrangement of vertebrae in the spine. The segmental pattern is established during embryogenesis when the somites, the embryonic segments of vertebrates, are rhythmically produced from the paraxial mesoderm. This process involves the segmentation clock, a traveling oscillator that interacts with a maturation wave called the wavefront to produce the periodic series of somites. Recent studies have shed light on several aspects of the segmentation mechanism and provide a conceptual framework to explain human spine malformations, such as congenital scoliosis.
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Cites
Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1
Li, L., Krantz, I. D., Deng, Y., Genin, A., Banta, A. B., Collins, C. C., Qi, M., Trask, B. J., Kuo, W. L., Cochran, J., et al. (1997) · Nature Genetics · doi:10.1038/ng0797-243 · Cited by 1,284
Mutations in the human Jagged1 gene are responsible for Alagille syndrome
Oda, T., Elkahloun, A. G., Pike, B. L., Okajima, K., Krantz, I. D., Genin, A., Piccoli, D. A., Meltzer, P. S., Spinner, N. B., Collins, F. S., et al. (1997) · Nature Genetics · doi:10.1038/ng0797-235 · Cited by 1,158
Embryonic retinoic acid synthesis is essential for early mouse post-implantation development
Niederreither, K., Subbarayan, V., Dollé, P., & Chambon, P. (1999) · Nature Genetics · doi:10.1038/7788 · Cited by 1,077
Palmeirim, I., Henrique, D., Ish-Horowicz, D., & Pourquié, O. (1997) · Cell · doi:10.1016/s0092-8674(00)80451-1 · Cited by 948
Self-citationNotch1 is required for the coordinate segmentation of somites
Conlon, R. A., Reaume, A. G., & Rossant, J. (1995) · Development · doi:10.1242/dev.121.5.1533 · Cited by 852
The Avian Embryo: Structural and Functional Development
Evans, H. E., & Romanoff, A. L. (1960) · Avian Diseases · doi:10.2307/1587707 · Cited by 811
Cooke, J., & Zeeman, E. C. (1976) · Journal of Theoretical Biology · doi:10.1016/s0022-5193(76)80131-2 · Cited by 784
Autoinhibition with Transcriptional Delay
Lewis, J. (2003) · Current Biology · doi:10.1016/s0960-9822(03)00534-7 · Cited by 746
Dubrulle, J., McGrew, M. J., & Pourquié, O. (2001) · Cell · doi:10.1016/s0092-8674(01)00437-8 · Cited by 667
Self-citationGlycosylation regulates Notch signalling
Haines, N., & Irvine, K. D. (2003) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm1228 · Cited by 645
Cited by
Notch signaling pathway: architecture, disease, and therapeutics
Zhou, B., Lin, W., Long, Y., Yang, Y., Zhang, H., Wu, K., & Chu, Q. (2022) · Signal Transduction and Targeted Therapy · doi:10.1038/s41392-022-00934-y · Cited by 1,114
Retinoic acid signalling during development
Rhinn, M., & Dollé, P. (2012) · Development · doi:10.1242/dev.065938 · Cited by 849
Loh, K. M., Chen, A., Koh, P. W., Deng, T. Z., Sinha, R., Tsai, J. M., Barkal, A. A., Shen, K. Y., Jain, R., Morganti, R. M., et al. (2016) · Cell · doi:10.1016/j.cell.2016.06.011 · Cited by 489
Notch signaling in human development and disease
Penton, A. L., Leonard, L. D., & Spinner, N. B. (2012) · Seminars in Cell & Developmental Biology · doi:10.1016/j.semcdb.2012.01.010 · Cited by 332
Dynamic Ligand Discrimination in the Notch Signaling Pathway
Nandagopal, N., Santat, L. A., LeBon, L., Sprinzak, D., Bronner, M. E., & Elowitz, M. B. (2018) · Cell · doi:10.1016/j.cell.2018.01.002 · Cited by 322
Metadata
- Authors
- Pourquié, O.
- Venue
- Cell
- Publisher
- Elsevier BV
- Published
- 2011-05
- Volume
- 145
- Issue
- 5
- Pages
- 650-663
- ISSN
- 0092-8674 (ISSN-L)
- OpenAlex
- W2033797505
- Cited by
- 338
- References
- 108
- FWCI
- 13.40
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.