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The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.

BACE1 Regulates Hippocampal Astrogenesis via the Jagged1-Notch Pathway

Hu, Xiangyou, He, Wanxia, Luo, Xiaoyang, Tsubota, Katherine Elyse, Yan, Riqiang·2013·Cell Reports
DOI 10.1016/j.celrep.2013.06.005PMC PMC3740554
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  • BACE1 cleavage of Jag1 regulates the Jag1-Notch signaling pathway important in the control of astrogenesis and neurogenesis.

    “BACE1 cleavage of Jagged 1 (Jag1) regulates the Jag1‐Notch signaling pathway important in the control of astrogenesis and neurogenesis.”

    From BACE1: More than just a β‐secretase (2022) · cites this paper as [29]

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Research works printed but not linked: 35

  1. Boyer-Di Ponio, 2007, Biological function of mutant forms of JAGGED1 proteins in Alagille syndrome: inhibitory effect on Notch signaling, Hum. Mol. Genet., 16, 2683, 10.1093/hmg/ddm222 · DOI
  2. Brunne, 2010, Origin, maturation, and astroglial transformation of secondary radial glial cells in the developing dentate gyrus, Glia, 58, 1553, 10.1002/glia.21029 · DOI
  3. Bushong, 2004, Maturation of astrocyte morphology and the establishment of astrocyte domains during postnatal hippocampal development, Int. J. Dev. Neurosci., 22, 73, 10.1016/j.ijdevneu.2003.12.008 · DOI
  4. Cai, 2001, BACE1 is the major beta-secretase for generation of Abeta peptides by neurons, Nat. Neurosci., 4, 233, 10.1038/85064 · DOI
  5. Cai, 2012, β-Secretase (BACE1) inhibition causes retinal pathology by vascular dysregulation and accumulation of age pigment, EMBO Mol Med, 4, 980, 10.1002/emmm.201101084 · DOI · Paper page
  6. Doetsch, 2003, The glial identity of neural stem cells, Nat. Neurosci., 6, 1127, 10.1038/nn1144 · DOI · Paper page
  7. Ever, 2005, Radial ‘glial’ progenitors: neurogenesis and signaling, Curr. Opin. Neurobiol., 15, 29, 10.1016/j.conb.2005.01.005 · DOI
  8. Feng, 1994, Brain lipid-binding protein (BLBP): a novel signaling system in the developing mammalian CNS, Neuron, 12, 895, 10.1016/0896-6273(94)90341-7 · DOI · Paper page
  9. Gundersen, 1988, The new stereological tools: disector, fractionator, nucleator and point sampled intercepts and their use in pathological research and diagnosis, APMIS, 96, 857, 10.1111/j.1699-0463.1988.tb00954.x · DOI
  10. Gundersen, 1988, Some new, simple and efficient stereological methods and their use in pathological research and diagnosis, APMIS, 96, 379, 10.1111/j.1699-0463.1988.tb05320.x · DOI · Paper page
  11. Hardy, 2002, The amyloid hypothesis of Alzheimer’s disease: progress and problems on the road to therapeutics, Science, 297, 353, 10.1126/science.1072994 · DOI · Paper page
  12. He, 2004, Reticulon family members modulate BACE1 activity and amyloid-beta peptide generation, Nat. Med., 10, 959, 10.1038/nm1088 · DOI
  13. Hitt, 2010, BACE1-/- mice exhibit seizure activity that does not correlate with sodium channel level or axonal localization, Mol. Neurodegener., 5, 31, 10.1186/1750-1326-5-31 · DOI · Paper page
  14. Hu, 2006, Bace1 modulates myelination in the central and peripheral nervous system, Nat. Neurosci., 9, 1520, 10.1038/nn1797 · DOI · Paper page
  15. Hu, 2008, Genetic deletion of BACE1 in mice affects remyelination of sciatic nerves, FASEB J., 22, 2970, 10.1096/fj.08-106666 · DOI · Paper page
  16. Hussain, 1999, Identification of a novel aspartic protease (Asp 2) as beta-secretase, Mol. Cell. Neurosci., 14, 419, 10.1006/mcne.1999.0811 · DOI · Paper page
  17. Ikeuchi, 2003, The Notch ligands, Delta1 and Jagged2, are substrates for presenilin-dependent “gamma-secretase” cleavage, J. Biol. Chem., 278, 7751, 10.1074/jbc.C200711200 · DOI
  18. Kempermann, 2004, Milestones of neuronal development in the adult hippocampus, Trends Neurosci., 27, 447, 10.1016/j.tins.2004.05.013 · DOI
  19. Kim, 2011, Reduced sodium channel Na(v)1.1 levels in BACE1-null mice, J. Biol. Chem., 286, 8106, 10.1074/jbc.M110.134692 · DOI
  20. Kopan, 2009, The canonical Notch signaling pathway: unfolding the activation mechanism, Cell, 137, 216, 10.1016/j.cell.2009.03.045 · DOI
  21. LaVoie, 2003, The Notch ligands, Jagged and Delta, are sequentially processed by alpha-secretase and presenilin/gamma-secretase and release signaling fragments, J. Biol. Chem., 278, 34427, 10.1074/jbc.M302659200 · DOI
  22. Lin, 2000, Human aspartic protease memapsin 2 cleaves the beta-secretase site of beta-amyloid precursor protein, Proc. Natl. Acad. Sci. USA, 97, 1456, 10.1073/pnas.97.4.1456 · DOI
  23. Liu, 2009, TACE-mediated ectodomain shedding of the type I TGF-beta receptor downregulates TGF-beta signaling, Mol. Cell, 35, 26, 10.1016/j.molcel.2009.06.018 · DOI
  24. Luo, 2001, Mice deficient in BACE1, the Alzheimer’s beta-secretase, have normal phenotype and abolished beta-amyloid generation, Nat. Neurosci., 4, 231, 10.1038/85059 · DOI · Paper page
  25. Luo, 2011, Cleavage of neuregulin-1 by BACE1 or ADAM10 protein produces differential effects on myelination, J. Biol. Chem., 286, 23967, 10.1074/jbc.M111.251538 · DOI
  26. Malatesta, 2008, Radial glia and neural stem cells, Cell Tissue Res., 331, 165, 10.1007/s00441-007-0481-8 · DOI
  27. Morrison, 2000, Transient Notch activation initiates an irreversible switch from neurogenesis to gliogenesis by neural crest stem cells, Cell, 101, 499, 10.1016/S0092-8674(00)80860-0 · DOI
  28. Parr-Sturgess, 2010, Ectodomain shedding of the Notch ligand Jagged1 is mediated by ADAM17, but is not a lipid-raft-associated event, Biochem. J., 432, 283, 10.1042/BJ20100321 · DOI
  29. Roberds, 2001, BACE knockout mice are healthy despite lacking the primary beta-secretase activity in brain: implications for Alzheimer’s disease therapeutics, Hum. Mol. Genet., 10, 1317, 10.1093/hmg/10.12.1317 · DOI · Paper page
  30. Sinha, 1999, Purification and cloning of amyloid precursor protein beta-secretase from human brain, Nature, 402, 537, 10.1038/990114 · DOI · Paper page
  31. Stump, 2002, Notch1 and its ligands Delta-like and Jagged are expressed and active in distinct cell populations in the postnatal mouse brain, Mech. Dev., 114, 153, 10.1016/S0925-4773(02)00043-6 · DOI
  32. von Bohlen und Halbach, 2011, Immunohistological markers for proliferative events, gliogenesis, and neurogenesis within the adult hippocampus, Cell Tissue Res., 345, 1, 10.1007/s00441-011-1196-4 · DOI
  33. Wang, 2008, BACE1 knock-outs display deficits in activity-dependent potentiation of synaptic transmission at mossy fiber to CA3 synapses in the hippocampus, J. Neurosci., 28, 8677, 10.1523/JNEUROSCI.2440-08.2008 · DOI
  34. Yan, 1999, Membrane-anchored aspartyl protease with Alzheimer’s disease beta-secretase activity, Nature, 402, 533, 10.1038/990107 · DOI · Paper page
  35. Yan, 2001, The transmembrane domain of the Alzheimer’s beta-secretase (BACE1) determines its late Golgi localization and access to beta -amyloid precursor protein (APP) substrate, J. Biol. Chem., 276, 36788, 10.1074/jbc.M104350200 · DOI

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Metadata

Authors
Hu, X., He, W., Luo, X., Tsubota, K. E., & Yan, R.
Venue
Cell Reports
Publisher
Elsevier BV
Published
2013-07
Volume
4
Issue
1
Pages
40-49
ISSN
2211-1247
DOI
10.1016/j.celrep.2013.06.005
PMC
PMC3740554
Cited by
81
References
5
Language
en
Licence

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