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ADAM10 Missense Mutations Potentiate β-Amyloid Accumulation by Impairing Prodomain Chaperone Function

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Aβ reduction in BACE1 heterozygous null 5XFAD mice is associated with transgenic APP level

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NRF2/ARE pathway negatively regulates BACE1 expression and ameliorates cognitive deficits in mouse Alzheimer’s models

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The Potent BACE1 Inhibitor LY2886721 Elicits Robust Central Aβ Pharmacodynamic Responses in Mice, Dogs, and Humans

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Transgenic mouse models of alzheimer disease: Developing a better model as a tool for therapeutic interventions.

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Modulation of Kv7 channels and excitability in the brain

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BACE1 is at the crossroad of a toxic vicious cycle involving cellular stress and β-amyloid production in Alzheimer’s disease

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BACE1 Deficiency Causes Altered Neuronal Activity and Neurodegeneration

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Alzheimer Disease Aβ Production in the Absence of S-Palmitoylation-dependent Targeting of BACE1 to Lipid Rafts

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BACE1 (β-secretase) transgenic and knockout mice: identification of neurochemical deficits and behavioral changes

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Disease modifying therapy for AD?¹

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BACE1 gene deletion: Impact on behavioral function in a model of Alzheimer’s disease

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Proteomic identification of sorting nexin 6 as a negative regulator of BACE1‐mediated APP processing

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RAGE mediates Aβ accumulation in a mouse model of Alzheimer’s disease via modulation of β- and γ-secretase activity

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miR‐186 is decreased in aged brain and suppresses <scp>BACE</scp>1 expression

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BACE1-/- mice exhibit seizure activity that does not correlate with sodium channel level or axonal localization

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Axonal organization defects in the hippocampus of adult conditional BACE1 knockout mice

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Physiological Roles of β-amyloid in Regulating Synaptic Function: Implications for AD Pathophysiology

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Modulation of γ- and β-secretases as early prevention against Alzheimer's disease

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Characterization of plasma β-secretase (BACE1) activity and soluble amyloid precursor proteins as potential biomarkers for Alzheimer’s disease

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Depletion of GGA1 and GGA3 Mediates Postinjury Elevation of BACE1

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The Cause of Alzheimer’s Disease: The Theory of Multipathology Convergence to Chronic Neuronal Stress

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BACE1 Protein Endocytosis and Trafficking Are Differentially Regulated by Ubiquitination at Lysine 501 and the Di-leucine Motif in the Carboxyl Terminus

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BACE1 inhibitory effects of lavandulyl flavanones from Sophora flavescens

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The Molecular Clock and Neurodegenerative Disease: A Stressful Time

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Biological and Computational Evaluation of Resveratrol Inhibitors against Alzheimer's Disease

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CutA Divalent Cation Tolerance Homolog (Escherichia coli) (CUTA) Regulates β-Cleavage of β-Amyloid Precursor Protein (APP) through Interacting with β-Site APP Cleaving Protein 1 (BACE1)

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