The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
The dual role of IL-6-type cytokines on bone remodeling and bone tumors
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Citations
Cites
Principles of interleukin (IL)-6-type cytokine signalling and its regulation
HEINRICH, P. C., BEHRMANN, I., HAAN, S., HERMANNS, H. M., MÜLLER-NEWEN, G., & SCHAPER, F. (2003) · Biochemical Journal · doi:10.1042/bj20030407 · Cited by 3,272 · cited 4×
“For example with the OSMR, the adaptor molecule Shc is recruited rather than SHP2, and mediates a higher MAPKs activation than with gp130 or LIFR [10].”
BackgroundInfluentialOsteoblast Programmed Cell Death (Apoptosis): Modulation by Growth Factors and Cytokines
Jilka, R. L., Weinstein, R. S., Bellido, T., Parfitt, A. M., & Manolagas, S. C. (1998) · Journal of Bone and Mineral Research · doi:10.1359/jbmr.1998.13.5.793 · Cited by 578 · cited 1×
“Finally, these cytokines can protect certain osteoblastic cells from apoptosis induced by serum depletion or TNFa [19].”
BackgroundBiologic sequelae of interleukin-6 induced PI3-K/Akt signaling in multiple myeloma
Hideshima, T., Nakamura, N., Chauhan, D., & Anderson, K. C. (2001) · Oncogene · doi:10.1038/sj.onc.1204833 · Cited by 478
Lamoureux, F., Richard, P., Wittrant, Y., Battaglia, S., Pilet, P., Trichet, V., Blanchard, F., Gouin, F., Pitard, B., Heymann, D., et al. (2007) · Cancer Research · doi:10.1158/0008-5472.can-06-4130 · Cited by 181
Self-citationKim, H., & Baumann, H. (1999) · Molecular and Cellular Biology · doi:10.1128/mcb.19.8.5326 · Cited by 170
Cited by
Robling, A. G., & Bonewald, L. F. (2020) · Annual Review of Physiology · doi:10.1146/annurev-physiol-021119-034332 · Cited by 539
Regulation of Osteoclast Differentiation by Cytokine Networks
Amarasekara, D. S., Yun, H., Kim, S., Lee, N., Kim, H., & Rho, J. (2018) · Immune Network · doi:10.4110/in.2018.18.e8 · Cited by 518
Guihard, P., Danger, Y., Brounais, B., David, E., Brion, R., Delecrin, J., Richards, C. D., Chevalier, S., Rédini, F., Heymann, D., et al. (2012) · Stem Cells · doi:10.1002/stem.1040 · Cited by 476
Self-citationPreshaw, P. M., & Taylor, J. J. (2011) · Journal of Clinical Periodontology · doi:10.1111/j.1600-051x.2010.01671.x · Cited by 422
Interleukin-6 in bone metastasis and cancer progression
Ara, T., & DeClerck, Y. A. (2010) · European Journal of Cancer · doi:10.1016/j.ejca.2010.02.026 · Cited by 379
Metadata
- Authors
- Blanchard, F., Duplomb, L., Baud’huin, M., & Brounais, B.
- Venue
- Cytokine & Growth Factor Reviews
- Publisher
- Elsevier BV
- Published
- 2009-02
- Volume
- 20
- Issue
- 1
- Pages
- 19-28
- ISSN
- 1359-6101 (ISSN-L)
- OpenAlex
- W2036947695
- Cited by
- 218
- References
- 5
- FWCI
- 0.45
- Type
- journal article
- Language
- en
- Open access
- closed
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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How to cite
Cite CoChat for the checks on this page, and the paper for its own findings.
- This page
- CoChat. (2026). Citation check: The dual role of IL-6-type cytokines on bone remodeling and bone tumors. https://app.cochat.ai/papers/10.1016/j.cytogfr.2008.11.004
- The paper
- Blanchard, F., Duplomb, L., Baud’huin, M., & Brounais, B. (2009). The dual role of IL-6-type cytokines on bone remodeling and bone tumors. Cytokine & Growth Factor Reviews. https://doi.org/10.1016/j.cytogfr.2008.11.004
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
