Student or educator? Sign up with your school email and get 6 months of Pro free.
Cited by 846

The DOI matches its Crossref record; 465 of the 465 references it cites were found in a registry or an index.

2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy

Open access
Towbin, Jeffrey A., McKenna, William J., Abrams, Dominic J., Ackerman, Michael J., Calkins, Hugh, Darrieux, Francisco C.C., Daubert, James P., de Chillou, Christian, DePasquale, Eugene C., Desai, Milind Y., Estes, N.A. Mark, Hua, Wei, Indik, Julia H., Ingles, Jodie, James, Cynthia A., John, Roy M., Judge, Daniel P., Keegan, Roberto, Krahn, Andrew D., Link, Mark S., Marcus, Frank I., McLeod, Christopher J., Mestroni, Luisa, Priori, Silvia G., Saffitz, Jeffrey E., Sanatani, Shubhayan, Shimizu, Wataru, van Tintelen, J. Peter, Wilde, Arthur A.M., Zareba, Wojciech·2019·Heart Rhythm

Abstract

Arrhythmogenic cardiomyopathy (ACM) is an arrhythmogenic disorder of the myocardium not secondary to ischemic, hypertensive or valvular heart disease. ACM incorporates a broad spectrum of genetic, systemic, infectious, and inflammatory disorders. This designation includes, but is not limited to, arrhythmogenic right/left ventricular cardiomyopathy, cardiac amyloid and sarcoidosis, Chagas' disease and left ventricular noncompaction. The ACM phenotype overlaps with other cardiomyopathies, particularly dilated cardiomyopathy with arrhythmia presentation which may be associated with ventricular dilatation and/or impaired systolic function. This expert consensus statement provides the clinician with guidance on evaluation and management of ACM and includes clinically relevant information on genetics and disease mechanisms. PICO (Patient, Intervention, Comparison, Outcome) questions were utilized to evaluate contemporary evidence and provide clinical guidance related to exercise in arrhythmogenic right ventricular cardiomyopathy. Recommendations were developed and approved by an expert writing group, after a systematic literature search with evidence tables, and discussion of their own clinical experience, to present the current knowledge in the field. Each recommendation is presented using the Class of Recommendation and Level of Evidence system formulated by the ACC and AHA and is accompanied by references and explanatory text, to provide essential context. The ongoing recognition of the genetic basis of ACM provides the opportunity to examine the diverse triggers and potential common pathway for the development of disease and arrhythmia.

Overview

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Integrity signals

Registry

Crossref · 10.1016/j.hrthm.2019.05.007

Cites

465 of 465 references verified0 citation errors · 0 not checkable

Statistics

Verification

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Citations

Cites

Show all 465 references

Research works printed but not linked: 77

  1. Towbin, 2011, Desmosomal gene variants in patients with “possible ARVC.”, Heart Rhythm, 8, 719, 10.1016/j.hrthm.2011.03.040 · doi:10.1016/j.hrthm.2011.03.040
  2. Marcus, 2009, The electrocardiogram in right ventricular cardiomyopathy/dysplasia. How can the electrocardiogram assist in understanding the pathologic and functional changes of the heart in this disease?, J Electrocardiol, 42, 10.1016/j.jelectrocard.2008.12.011 · doi:10.1016/j.jelectrocard.2008.12.011
  3. Lohrmann, 2016, Electrocardiographic abnormalities in disease-free black South Africans and correlations with echocardiographic indexes and early repolarization, Am J Cardiol, 118, 765, 10.1016/j.amjcard.2016.06.006 · doi:10.1016/j.amjcard.2016.06.006
  4. Tanawuttiwat, 2016, Electroanatomic correlates of depolarization abnormalities in arrhythmogenic right ventricular dysplasia/cardiomyopathy, J Cardiovasc Electrophysiol, 27, 443, 10.1111/jce.12925 · doi:10.1111/jce.12925
  5. Posch, 2008, Variations in DSG2: V56M, V158G and V920G are not pathogenic for arrhythmogenic right ventricular dysplasia/cardiomyopathy, Nat Clin Pract Cardiovasc Med, 5, E1, 10.1038/ncpcardio1434 · doi:10.1038/ncpcardio1434
  6. Christensen, 2010, Missense variants in plakophilin-2 in arrhythmogenic right ventricular cardiomyopathy patients—disease-causing or innocent bystanders?, Cardiology, 115, 148, 10.1159/000263456 · doi:10.1159/000263456
  7. Gandjbakhch, 2011, Plakophilin 2A is the dominant isoform in human heart tissue: consequences for the genetic screening of arrhythmogenic right ventricular cardiomyopathy, Heart, 97, 844, 10.1136/hrt.2010.205880 · doi:10.1136/hrt.2010.205880
  8. Roberts, 2013, Detection of genomic deletions of PKP2 in arrhythmogenic right ventricular cardiomyopathy, Clin Genet, 83, 452, 10.1111/j.1399-0004.2012.01950.x · doi:10.1111/j.1399-0004.2012.01950.x
  9. Kato, 2016, LMNA cardiomyopathy detected in Japanese arrhythmogenic right ventricular cardiomyopathy cohort, J Cardiol, 68, 346, 10.1016/j.jjcc.2015.10.013 · doi:10.1016/j.jjcc.2015.10.013
  10. Valtuille, 2013, A case of lamin A/C mutation cardiomyopathy with overlap features of ARVC: a critical role of genetic testing, Int J Cardiol, 168, 4325, 10.1016/j.ijcard.2013.04.177 · doi:10.1016/j.ijcard.2013.04.177
  11. Wlodarska, 2006, Thromboembolic complications in patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy, Europace, 8, 596, 10.1093/europace/eul053 · doi:10.1093/europace/eul053
  12. Berruezo, 2017, Europace, 19, 607, 10.1093/europace/eux056 · doi:10.1093/europace/eux056
  13. Honarbakhsh, 2013, Successful right ventricular tachycardia ablation in a patient with left ventricular non-compaction cardiomyopathy, Indian Pacing Electrophysiol J, 13, 181, 10.1016/S0972-6292(16)30671-4 · doi:10.1016/s0972-6292(16)30671-4
  14. Jackson, 2015, Case report: ablation of diffuse inter-trabecular substrate in a patient with isolated ventricular non-compaction, Indian Pacing Electrophysiol J, 15, 162, 10.1016/j.ipej.2015.09.002 · doi:10.1016/j.ipej.2015.09.002
  15. Chung, 2017, Catheter ablation of ventricular tachycardia/fibrillation in a patient with right ventricular amyloidosis with initial manifestations mimicking arrhythmogenic right ventricular dysplasia/cardiomyopathy, Korean Circ J, 47, 282, 10.4070/kcj.2016.0328 · doi:10.4070/kcj.2016.0328
  16. Mlcochova, 2006, Catheter ablation of ventricular fibrillation storm in patients with infiltrative amyloidosis of the heart, J Cardiovasc Electrophysiol, 17, 426, 10.1111/j.1540-8167.2005.00321.x · doi:10.1111/j.1540-8167.2005.00321.x
  17. Magage, 2005, Fabry disease: percutaneous transluminal septal myocardial ablation markedly improved symptomatic left ventricular hypertrophy and outflow tract obstruction in a classically affected male, Echocardiography, 22, 333, 10.1111/j.1540-8175.2005.03191.x · doi:10.1111/j.1540-8175.2005.03191.x
  18. Berruezo, 2017, Epicardial ablation may not be necessary in all patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy and frequent ventricular tachycardia: author's reply, Europace, 19, 2047, 10.1093/europace/eux056 · doi:10.1093/europace/eux056
  19. Shi, 2008, The cardiac sodium channel mutation delQKP 1507-1509 is associated with the expanding phenotypic spectrum of LQT3, conduction disorder, dilated cardiomyopathy, and high incidence of youth sudden death, Europace, 10, 1329, 10.1093/europace/eun202 · doi:10.1093/europace/eun202
  20. Shan, 2008, SCN5A variants in Japanese patients with left ventricular noncompaction and arrhythmia, Mol Genet Metab, 93, 468, 10.1016/j.ymgme.2007.10.009 · doi:10.1016/j.ymgme.2007.10.009
  21. Beckermann, 2014, Novel SCN5A mutation in amiodarone-responsive multifocal ventricular ectopy-associated cardiomyopathy, Heart Rhythm, 11, 1446, 10.1016/j.hrthm.2014.04.042 · doi:10.1016/j.hrthm.2014.04.042
  22. Xi, 2012, Loss of function of hNav1.5 by a ZASP1 mutation associated with intraventricular conduction disturbances in left ventricular noncompaction, Circ Arrhythm Electrophysiol, 5, 1017, 10.1161/CIRCEP.111.969220 · DOI
  23. Brette, 2006, Density and sub-cellular distribution of cardiac and neuronal sodium channel isoforms in rat ventricular myocytes, Biochem Biophys Res Commun, 348, 1163, 10.1016/j.bbrc.2006.07.189 · doi:10.1016/j.bbrc.2006.07.189
  24. van Spaendonck-Zwarts, 2012, Recurrent and founder mutations in the Netherlands: the cardiac phenotype of DES founder mutations p.S13F and p.N342D, Neth Heart J, 20, 219, 10.1007/s12471-011-0233-y · doi:10.1007/s12471-011-0233-y
  25. Arita, 1993, [Cellular electrophysiological basis of proarrhythmic and antiarrhythmic effects of ischemia-related lipid metabolites], Rinsho Byori, 41, 401
  26. Schmilinsky-Fluri, 1997, Modulation of cardiac gap junctions: the mode of action of arachidonic acid, J Mol Cell Cardiol, 29, 1703, 10.1006/jmcc.1997.0409 · doi:10.1006/jmcc.1997.0409
  27. Bove, 1973, Focal lipid cardiomyopathy in an infant with paroxysmal atrial tachycardia, Arch Pathol, 95, 26
  28. Ferrans, 1976, Infantile cardiomyopathy with histiocytoid change in cardiac muscle cells. Report of six patients, Circulation, 53, 708, 10.1161/01.CIR.53.4.708 · doi:10.1161/01.cir.53.4.708
  29. Shehata, 1998, Histiocytoid cardiomyopathy: three new cases and a review of the literature, Pediatr Dev Pathol, 1, 56, 10.1007/s100249900007 · doi:10.1007/s100249900007
  30. Malhotra, 1994, Infantile histiocytoid cardiomyopathy: three cases and literature review, Am Heart J, 128, 1009, 10.1016/0002-8703(94)90601-7 · doi:10.1016/0002-8703(94)90601-7
  31. MacMahon, 1971, Infantile xanthomatous cardiomyopathy, Pediatrics, 48, 312, 10.1542/peds.48.2.312 · doi:10.1542/peds.48.2.312
  32. Ruszkiewicz, 1995, Sudden death in an infant due to histiocytoid cardiomyopathy. A light-microscopic, ultrastructural, and immunohistochemical study, Am J Forensic Med Pathol, 16, 74, 10.1097/00000433-199503000-00017 · doi:10.1097/00000433-199503000-00017
  33. Prahlow, 1993, Histiocytoid cardiomyopathy: case report and literature review, J Forensic Sci, 38, 1427, 10.1520/JFS13547J · doi:10.1520/jfs13547j
  34. Heifetz, 1995, Pathological case of the month. Histiocytoid (oncocytic) cardiomyopathy, Arch Pediatr Adolesc Med, 149, 464, 10.1001/archpedi.1995.02170160118020 · doi:10.1001/archpedi.1995.02170160118020
  35. Suarez, 1987, Foamy myocardial transformation of infancy: an inherited disease, J Clin Pathol, 40, 329, 10.1136/jcp.40.3.329 · doi:10.1136/jcp.40.3.329
  36. Franciosi, 1988, Oncocytic cardiomyopathy syndrome, Hum Pathol, 19, 1361, 10.1016/S0046-8177(88)80296-X · doi:10.1016/s0046-8177(88)80296-x
  37. Grech, 2000, Cardiac malformations associated with the congenital nephrotic syndrome, Pediatr Nephrol, 14, 1115, 10.1007/s004670000401 · doi:10.1007/s004670000401
  38. Ferrans, 1989, Pathologic anatomy of the dilated cardiomyopathies, Am J Cardiol, 64, 9C, 10.1016/0002-9149(89)90677-2 · doi:10.1016/0002-9149(89)90677-2
  39. Saffitz, 1983, Histiocytoid cardiomyopathy: a cause of sudden death in apparently healthy infants, Am J Cardiol, 52, 215, 10.1016/0002-9149(83)90103-0 · doi:10.1016/0002-9149(83)90103-0
  40. Kauffman, 1972, Idiopathic infantile cardiomyopathy with involvement of the conduction system, Am J Cardiol, 30, 648, 10.1016/0002-9149(72)90603-0 · doi:10.1016/0002-9149(72)90603-0
  41. Maurer, 2019, Tafamidis for transthyretin amyloid cardiomyopathy, N Engl J Med, 380, 196, 10.1056/NEJMc1814074 · doi:10.1056/nejmc1814074
  42. Mueller, 2000, Symptomatic ischemic heart disease resulting from obstructive intramural coronary amyloidosis, Am J Med, 109, 181, 10.1016/S0002-9343(00)00471-X · doi:10.1016/s0002-9343(00)00471-x
  43. Mathew, 1997, Symptomatic conduction system disease in cardiac amyloidosis, Am J Cardiol, 80, 1491, 10.1016/S0002-9149(97)82785-3 · doi:10.1016/s0002-9149(97)82785-3
  44. Rezk, 2018, Role of implantable intracardiac defibrillators in patients with cardiac immunoglobulin light chain amyloidosis, Br J Haematol, 182, 145
  45. Eriksson, 1984, The sinoatrial node in familial amyloidosis with polyneuropathy. A clinico-pathological study of nine cases from northern Sweden, Virchows Arch A Pathol Anat Histopathol, 402, 239, 10.1007/BF00695078 · doi:10.1007/bf00695078
  46. Eriksson, 1984, Pacemaker treatment in familial amyloidosis with polyneuropathy, Pacing Clin Electrophysiol, 7, 702, 10.1111/j.1540-8159.1984.tb05599.x · doi:10.1111/j.1540-8159.1984.tb05599.x
  47. Olofsson, 1983, The sick sinus syndrome in familial amyloidosis with polyneuropathy, Int J Cardiol, 4, 71, 10.1016/0167-5273(83)90217-6 · doi:10.1016/0167-5273(83)90217-6
  48. Barbhaiya, 2016, Electrophysiologic assessment of conduction abnormalities and atrial arrhythmias associated with amyloid cardiomyopathy, Heart Rhythm, 13, 383, 10.1016/j.hrthm.2015.09.016 · doi:10.1016/j.hrthm.2015.09.016
  49. Capone, 1972, Systemic amyloidosis, functional coronary insufficiency, and autonomic impairment, Ann Intern Med, 76, 599, 10.7326/0003-4819-76-4-599 · doi:10.7326/0003-4819-76-4-599
  50. Gertz, 1985, Worsening of congestive heart failure in amyloid heart disease treated by calcium channel-blocking agents, Am J Cardiol, 55, 1645, 10.1016/0002-9149(85)90995-6 · doi:10.1016/0002-9149(85)90995-6
  51. Griffiths, 1982, Cardiac amyloidosis with asymmetrical septal hypertrophy and deterioration after nifedipine, Thorax, 37, 711, 10.1136/thx.37.9.711 · doi:10.1136/thx.37.9.711
  52. Tan, 2016, Catheter ablation for atrial arrhythmias in patients with cardiac amyloidosis, J Cardiovasc Electrophysiol, 27, 1167, 10.1111/jce.13046 · doi:10.1111/jce.13046
  53. Dubrey, 1998, The clinical features of immunoglobulin light-chain (AL) amyloidosis with heart involvement, QJM, 91, 141, 10.1093/qjmed/91.2.141 · doi:10.1093/qjmed/91.2.141
  54. Lin, 2010, Successful termination of a ventricular arrhythmia by implantable cardioverter defibrillator therapy in a patient with cardiac amyloidosis: insight into mechanisms of sudden death, Eur Heart J, 31, 1538, 10.1093/eurheartj/ehp592 · doi:10.1093/eurheartj/ehp592
  55. Patel, 2014, Life-saving implantable cardioverter defibrillator therapy in cardiac AL amyloidosis, BMJ Case Rep, 2014, 10.1136/bcr-2014-206600 · doi:10.1136/bcr-2014-206600
  56. Tukkie, 2004, Delay in right ventricular activation contributes to Brugada syndrome, Circulation, 109, 1272, 10.1161/01.CIR.0000118467.53182.D1 · doi:10.1161/01.cir.0000118467.53182.d1
  57. Papavassiliu, 2010, Spontaneous type 1 electrocardiographic pattern is associated with cardiovascular magnetic resonance imaging changes in Brugada syndrome, Heart Rhythm, 7, 1790, 10.1016/j.hrthm.2010.09.004 · DOI
  58. Ohkubo, 2010, Right ventricular histological substrate and conduction delay in patients with Brugada syndrome, Int Heart J, 51, 17, 10.1536/ihj.51.17 · doi:10.1536/ihj.51.17
  59. Kharbanda, 2017, Long QT syndrome and left ventricular noncompaction in 4 family members across 2 generations with KCNQ1 mutation, Eur J Med Genet, 60, 233, 10.1016/j.ejmg.2017.02.003 · doi:10.1016/j.ejmg.2017.02.003
  60. Nakashima, 2013, A left ventricular noncompaction in a patient with long QT syndrome caused by a KCNQ1 mutation: a case report, Heart Vessels, 28, 126, 10.1007/s00380-012-0235-8 · doi:10.1007/s00380-012-0235-8
  61. Ogawa, 2009, Isolated non-compaction of the ventricular myocardium associated with long QT syndrome: a report of 2 cases, Circ J, 73, 2169, 10.1253/circj.CJ-08-0339 · doi:10.1253/circj.cj-08-0339
  62. Murakami, 2003, Identification and characterization of the murine TRPM4 channel, Biochem Biophys Res Commun, 307, 522, 10.1016/S0006-291X(03)01186-0 · doi:10.1016/s0006-291x(03)01186-0
  63. Liu, 2013, Molecular genetics and functional anomalies in a series of 248 Brugada cases with 11 mutations in the TRPM4 channel, PLoS One, 8, e54131, 10.1371/journal.pone.0054131 · doi:10.1371/journal.pone.0054131
  64. Saito, 2018, TRPM4 Mutation in Patients With Ventricular Noncompaction and Cardiac Conduction Disease, Circ Genom Precis Med, 11, e002103, 10.1161/CIRCGEN.118.002103 · doi:10.1161/circgen.118.002103
  65. Posch, 2009, Genetic deletion of arginine 14 in phospholamban causes dilated cardiomyopathy with attenuated electrocardiographic R amplitudes, Heart Rhythm, 6, 480, 10.1016/j.hrthm.2009.01.016 · doi:10.1016/j.hrthm.2009.01.016
  66. Groeneweg, 2013, Left-dominant arrhythmogenic cardiomyopathy in a large family: associated desmosomal or nondesmosomal genotype?, Heart Rhythm, 10, 548, 10.1016/j.hrthm.2012.12.020 · doi:10.1016/j.hrthm.2012.12.020
  67. Groeneweg, 2013, Arrhythmogenic right ventricular dysplasia/cardiomyopathy according to revised 2010 task force criteria with inclusion of non-desmosomal phospholamban mutation carriers, Am J Cardiol, 112, 1197, 10.1016/j.amjcard.2013.06.017 · doi:10.1016/j.amjcard.2013.06.017
  68. Bartram, 2007, Primary noncompaction of the ventricular myocardium from the morphogenetic standpoint, Pediatr Cardiol, 28, 325, 10.1007/s00246-006-0054-8 · doi:10.1007/s00246-006-0054-8
  69. Finsterer, 2017, Left ventricular noncompaction cardiomyopathy: cardiac, neuromuscular, and genetic factors, Nat Rev Cardiol, 14, 224, 10.1038/nrcardio.2016.207 · doi:10.1038/nrcardio.2016.207
  70. Kawel, 2012, Trabeculated (noncompacted) and compact myocardium in adults: the multi-ethnic study of atherosclerosis, Circ Cardiovasc Imaging, 5, 357, 10.1161/CIRCIMAGING.111.971713 · doi:10.1161/circimaging.111.971713
  71. Weir-McCall, 2016, Left ventricular noncompaction: anatomical phenotype or distinct cardiomyopathy?, J Am Coll Cardiol, 68, 2157, 10.1016/j.jacc.2016.08.054 · doi:10.1016/j.jacc.2016.08.054
  72. Rhee, 2017, Navigating genetic and phenotypic uncertainty in left ventricular noncompaction, Circ Cardiovasc Genet, 10, e001857, 10.1161/CIRCGENETICS.117.001857 · doi:10.1161/circgenetics.117.001857
  73. Bainbridge, 2015, Loss of function mutations in NNT are associated with left ventricular noncompaction, Circ Cardiovasc Genet, 8, 544, 10.1161/CIRCGENETICS.115.001026 · doi:10.1161/circgenetics.115.001026
  74. Andreini, 2016, Long-term prognostic value of cardiac magnetic resonance in left ventricle noncompaction: a prospective multicenter study, J Am Coll Cardiol, 68, 2166, 10.1016/j.jacc.2016.08.053 · doi:10.1016/j.jacc.2016.08.053
  75. Muser, 2017, Ventricular arrhythmias associated with left ventricular noncompaction: electrophysiologic characteristics, mapping, and ablation, Heart Rhythm, 14, 166, 10.1016/j.hrthm.2016.11.014 · doi:10.1016/j.hrthm.2016.11.014
  76. Sidhu, 2014, Defining left ventricular noncompaction using cardiac computed tomography, J Thorac Imaging, 29, 60, 10.1097/RTI.0b013e31828e9b3d · doi:10.1097/rti.0b013e31828e9b3d
  77. Kawel-Boehm, 2017, Hypertrabeculated left ventricular myocardium in relationship to myocardial function and fibrosis: the Multi-Ethnic Study of Atherosclerosis, Radiology, 284, 667, 10.1148/radiol.2017161995 · doi:10.1148/radiol.2017161995

Cited by

See all 846

Metadata

Authors
Towbin, J. A., McKenna, W. J., Abrams, D. J., Ackerman, M. J., Calkins, H., Darrieux, F. C. C., Daubert, J. P., de Chillou, C., DePasquale, E. C., Desai, M. Y., Estes, N. A. M., Hua, W., Indik, J. H., Ingles, J., James, C. A., John, R. M., Judge, D. P., Keegan, R., Krahn, A. D., Link, M. S., Marcus, F. I., McLeod, C. J., Mestroni, L., Priori, S. G., Saffitz, J. E., Sanatani, S., Shimizu, W., van Tintelen, J. P., Wilde, A. A. M., & Zareba, W.
Venue
Heart Rhythm
Publisher
Elsevier BV
Published
2019-11
Volume
16
Issue
11
Pages
e301-e372
ISSN
1547-5271 (ISSN-L)
OpenAlex
W2944140727
Cited by
846
References
465
FWCI
63.74
Type
journal article
Language
en
Open access
bronze
Licence
www.elsevier.com/tdm/userlicense/1.0/ (tdm)

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review

How to cite

Cite CoChat for the checks on this page, and the paper for its own findings.

This page
CoChat. (2026). Citation check: 2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy. https://app.cochat.ai/papers/10.1016/j.hrthm.2019.05.007
The paper
Towbin, J. A., McKenna, W. J., Abrams, D. J., Ackerman, M. J., Calkins, H., Darrieux, F. C. C., Daubert, J. P., de Chillou, C., DePasquale, E. C., Desai, M. Y., Estes, N. A. M., Hua, W., Indik, J. H., Ingles, J., James, C. A., John, R. M., Judge, D. P., Keegan, R., Krahn, A. D., . . . Zareba, W. (2019). 2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy. Heart Rhythm. https://doi.org/10.1016/j.hrthm.2019.05.007

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.