The DOI matches its Crossref record; 465 of the 465 references it cites were found in a registry or an index.
2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy
Open accessAbstract
Arrhythmogenic cardiomyopathy (ACM) is an arrhythmogenic disorder of the myocardium not secondary to ischemic, hypertensive or valvular heart disease. ACM incorporates a broad spectrum of genetic, systemic, infectious, and inflammatory disorders. This designation includes, but is not limited to, arrhythmogenic right/left ventricular cardiomyopathy, cardiac amyloid and sarcoidosis, Chagas' disease and left ventricular noncompaction. The ACM phenotype overlaps with other cardiomyopathies, particularly dilated cardiomyopathy with arrhythmia presentation which may be associated with ventricular dilatation and/or impaired systolic function. This expert consensus statement provides the clinician with guidance on evaluation and management of ACM and includes clinically relevant information on genetics and disease mechanisms. PICO (Patient, Intervention, Comparison, Outcome) questions were utilized to evaluate contemporary evidence and provide clinical guidance related to exercise in arrhythmogenic right ventricular cardiomyopathy. Recommendations were developed and approved by an expert writing group, after a systematic literature search with evidence tables, and discussion of their own clinical experience, to present the current knowledge in the field. Each recommendation is presented using the Class of Recommendation and Level of Evidence system formulated by the ACC and AHA and is accompanied by references and explanatory text, to provide essential context. The ongoing recognition of the genetic basis of ACM provides the opportunity to examine the diverse triggers and potential common pathway for the development of disease and arrhythmia.
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Richards, S., Aziz, N., Bale, S., Bick, D., Das, S., Gastier-Foster, J., Grody, W. W., Hegde, M., Lyon, E., Spector, E., et al. (2015) · Genetics in Medicine · doi:10.1038/gim.2015.30 · Cited by 32,594
McDonagh, T. A., Metra, M., Adamo, M., Gardner, R. S., Baumbach, A., Böhm, M., Burri, H., Butler, J., Čelutkienė, J., Chioncel, O., et al. (2021) · European Heart Journal · doi:10.1093/eurheartj/ehab670 · Cited by 19,442
2013 ACCF/AHA Guideline for the Management of Heart Failure
Yancy, C. W., Jessup, M., Bozkurt, B., Butler, J., Casey, D. E., Drazner, M. H., Fonarow, G. C., Geraci, S. A., Horwich, T., Januzzi, J. L., et al. (2013) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2013.05.019 · Cited by 12,536
2016 ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure
Ponikowski, P., Voors, A. A., Anker, S. D., Bueno, H., Cleland, J. G. F., Coats, A. J. S., Falk, V., González-Juanatey, J. R., Harjola, V.-P., Jankowska, E. A., et al. (2016) · European Heart Journal · doi:10.1093/eurheartj/ehw128 · Cited by 11,429
Self-citationAnalysis of protein-coding genetic variation in 60,706 humans
Exome Aggregation Consortium, Lek, M., Karczewski, K. J., Minikel, E. V., Samocha, K. E., Banks, E., Fennell, T., O’Donnell-Luria, A. H., Ware, J. S., Hill, A. J., et al. (2016) · Nature · doi:10.1038/nature19057 · Cited by 9,587
Garber, C. E., Blissmer, B., Deschenes, M. R., Franklin, B. A., Lamonte, M. J., Lee, I.-M., Nieman, D. C., & Swain, D. P. (2011) · Medicine & Science in Sports & Exercise · doi:10.1249/mss.0b013e318213fefb · Cited by 9,319 · cited 1×
“Vigorousintensity activities are generally considered those requiring 6 METs.(251) The 2011 Adult Compendium of Physical Activities provides a comprehensive listingof theMETsassociatedwith a variety of physical activities (https://sites.”
BackgroundJanuary, C. T., Wann, L. S., Alpert, J. S., Calkins, H., Cigarroa, J. E., Cleveland, J. C., Conti, J. B., Ellinor, P. T., Ezekowitz, M. D., Field, M. E., et al. (2014) · Circulation · doi:10.1161/cir.0000000000000041 · Cited by 7,115
Self-citationAngiotensin-neprilysin inhibition versus enalapril in heart failure
McMurray, J. J. V., Packer, M., Desai, A. S., Gong, J., Lefkowitz, M. P., Rizkala, A. R., Rouleau, J. L., Shi, V. C., Solomon, S. D., Swedberg, K., et al. (2014) · New England Journal of Medicine · doi:10.1056/nejmoa1409077 · Cited by 6,957
Amiodarone or an Implantable Cardioverter–Defibrillator for Congestive Heart Failure
Bardy, G. H., Lee, K. L., Mark, D. B., Poole, J. E., Packer, D. L., Boineau, R., Domanski, M., Troutman, C., Anderson, J., Johnson, G., et al. (2005) · New England Journal of Medicine · doi:10.1056/nejmoa043399 · Cited by 6,552
2016 ESC Guidelines for the management of atrial fibrillation developed in collaboration with EACTS
Kirchhof, P., Benussi, S., Kotecha, D., Ahlsson, A., Atar, D., Casadei, B., Castella, M., Diener, H.-C., Heidbuchel, H., Hendriks, J., et al. (2016) · Europace · doi:10.1093/europace/euw295 · Cited by 6,439
Research works printed but not linked: 77
- Towbin, 2011, Desmosomal gene variants in patients with “possible ARVC.”, Heart Rhythm, 8, 719, 10.1016/j.hrthm.2011.03.040 · doi:10.1016/j.hrthm.2011.03.040
- Marcus, 2009, The electrocardiogram in right ventricular cardiomyopathy/dysplasia. How can the electrocardiogram assist in understanding the pathologic and functional changes of the heart in this disease?, J Electrocardiol, 42, 10.1016/j.jelectrocard.2008.12.011 · doi:10.1016/j.jelectrocard.2008.12.011
- Lohrmann, 2016, Electrocardiographic abnormalities in disease-free black South Africans and correlations with echocardiographic indexes and early repolarization, Am J Cardiol, 118, 765, 10.1016/j.amjcard.2016.06.006 · doi:10.1016/j.amjcard.2016.06.006
- Tanawuttiwat, 2016, Electroanatomic correlates of depolarization abnormalities in arrhythmogenic right ventricular dysplasia/cardiomyopathy, J Cardiovasc Electrophysiol, 27, 443, 10.1111/jce.12925 · doi:10.1111/jce.12925
- Posch, 2008, Variations in DSG2: V56M, V158G and V920G are not pathogenic for arrhythmogenic right ventricular dysplasia/cardiomyopathy, Nat Clin Pract Cardiovasc Med, 5, E1, 10.1038/ncpcardio1434 · doi:10.1038/ncpcardio1434
- Christensen, 2010, Missense variants in plakophilin-2 in arrhythmogenic right ventricular cardiomyopathy patients—disease-causing or innocent bystanders?, Cardiology, 115, 148, 10.1159/000263456 · doi:10.1159/000263456
- Gandjbakhch, 2011, Plakophilin 2A is the dominant isoform in human heart tissue: consequences for the genetic screening of arrhythmogenic right ventricular cardiomyopathy, Heart, 97, 844, 10.1136/hrt.2010.205880 · doi:10.1136/hrt.2010.205880
- Roberts, 2013, Detection of genomic deletions of PKP2 in arrhythmogenic right ventricular cardiomyopathy, Clin Genet, 83, 452, 10.1111/j.1399-0004.2012.01950.x · doi:10.1111/j.1399-0004.2012.01950.x
- Kato, 2016, LMNA cardiomyopathy detected in Japanese arrhythmogenic right ventricular cardiomyopathy cohort, J Cardiol, 68, 346, 10.1016/j.jjcc.2015.10.013 · doi:10.1016/j.jjcc.2015.10.013
- Valtuille, 2013, A case of lamin A/C mutation cardiomyopathy with overlap features of ARVC: a critical role of genetic testing, Int J Cardiol, 168, 4325, 10.1016/j.ijcard.2013.04.177 · doi:10.1016/j.ijcard.2013.04.177
- Wlodarska, 2006, Thromboembolic complications in patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy, Europace, 8, 596, 10.1093/europace/eul053 · doi:10.1093/europace/eul053
- Berruezo, 2017, Europace, 19, 607, 10.1093/europace/eux056 · doi:10.1093/europace/eux056
- Honarbakhsh, 2013, Successful right ventricular tachycardia ablation in a patient with left ventricular non-compaction cardiomyopathy, Indian Pacing Electrophysiol J, 13, 181, 10.1016/S0972-6292(16)30671-4 · doi:10.1016/s0972-6292(16)30671-4
- Jackson, 2015, Case report: ablation of diffuse inter-trabecular substrate in a patient with isolated ventricular non-compaction, Indian Pacing Electrophysiol J, 15, 162, 10.1016/j.ipej.2015.09.002 · doi:10.1016/j.ipej.2015.09.002
- Chung, 2017, Catheter ablation of ventricular tachycardia/fibrillation in a patient with right ventricular amyloidosis with initial manifestations mimicking arrhythmogenic right ventricular dysplasia/cardiomyopathy, Korean Circ J, 47, 282, 10.4070/kcj.2016.0328 · doi:10.4070/kcj.2016.0328
- Mlcochova, 2006, Catheter ablation of ventricular fibrillation storm in patients with infiltrative amyloidosis of the heart, J Cardiovasc Electrophysiol, 17, 426, 10.1111/j.1540-8167.2005.00321.x · doi:10.1111/j.1540-8167.2005.00321.x
- Magage, 2005, Fabry disease: percutaneous transluminal septal myocardial ablation markedly improved symptomatic left ventricular hypertrophy and outflow tract obstruction in a classically affected male, Echocardiography, 22, 333, 10.1111/j.1540-8175.2005.03191.x · doi:10.1111/j.1540-8175.2005.03191.x
- Berruezo, 2017, Epicardial ablation may not be necessary in all patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy and frequent ventricular tachycardia: author's reply, Europace, 19, 2047, 10.1093/europace/eux056 · doi:10.1093/europace/eux056
- Shi, 2008, The cardiac sodium channel mutation delQKP 1507-1509 is associated with the expanding phenotypic spectrum of LQT3, conduction disorder, dilated cardiomyopathy, and high incidence of youth sudden death, Europace, 10, 1329, 10.1093/europace/eun202 · doi:10.1093/europace/eun202
- Shan, 2008, SCN5A variants in Japanese patients with left ventricular noncompaction and arrhythmia, Mol Genet Metab, 93, 468, 10.1016/j.ymgme.2007.10.009 · doi:10.1016/j.ymgme.2007.10.009
- Beckermann, 2014, Novel SCN5A mutation in amiodarone-responsive multifocal ventricular ectopy-associated cardiomyopathy, Heart Rhythm, 11, 1446, 10.1016/j.hrthm.2014.04.042 · doi:10.1016/j.hrthm.2014.04.042
- Xi, 2012, Loss of function of hNav1.5 by a ZASP1 mutation associated with intraventricular conduction disturbances in left ventricular noncompaction, Circ Arrhythm Electrophysiol, 5, 1017, 10.1161/CIRCEP.111.969220 · DOI
- Brette, 2006, Density and sub-cellular distribution of cardiac and neuronal sodium channel isoforms in rat ventricular myocytes, Biochem Biophys Res Commun, 348, 1163, 10.1016/j.bbrc.2006.07.189 · doi:10.1016/j.bbrc.2006.07.189
- van Spaendonck-Zwarts, 2012, Recurrent and founder mutations in the Netherlands: the cardiac phenotype of DES founder mutations p.S13F and p.N342D, Neth Heart J, 20, 219, 10.1007/s12471-011-0233-y · doi:10.1007/s12471-011-0233-y
- Arita, 1993, [Cellular electrophysiological basis of proarrhythmic and antiarrhythmic effects of ischemia-related lipid metabolites], Rinsho Byori, 41, 401
- Schmilinsky-Fluri, 1997, Modulation of cardiac gap junctions: the mode of action of arachidonic acid, J Mol Cell Cardiol, 29, 1703, 10.1006/jmcc.1997.0409 · doi:10.1006/jmcc.1997.0409
- Bove, 1973, Focal lipid cardiomyopathy in an infant with paroxysmal atrial tachycardia, Arch Pathol, 95, 26
- Ferrans, 1976, Infantile cardiomyopathy with histiocytoid change in cardiac muscle cells. Report of six patients, Circulation, 53, 708, 10.1161/01.CIR.53.4.708 · doi:10.1161/01.cir.53.4.708
- Shehata, 1998, Histiocytoid cardiomyopathy: three new cases and a review of the literature, Pediatr Dev Pathol, 1, 56, 10.1007/s100249900007 · doi:10.1007/s100249900007
- Malhotra, 1994, Infantile histiocytoid cardiomyopathy: three cases and literature review, Am Heart J, 128, 1009, 10.1016/0002-8703(94)90601-7 · doi:10.1016/0002-8703(94)90601-7
- MacMahon, 1971, Infantile xanthomatous cardiomyopathy, Pediatrics, 48, 312, 10.1542/peds.48.2.312 · doi:10.1542/peds.48.2.312
- Ruszkiewicz, 1995, Sudden death in an infant due to histiocytoid cardiomyopathy. A light-microscopic, ultrastructural, and immunohistochemical study, Am J Forensic Med Pathol, 16, 74, 10.1097/00000433-199503000-00017 · doi:10.1097/00000433-199503000-00017
- Prahlow, 1993, Histiocytoid cardiomyopathy: case report and literature review, J Forensic Sci, 38, 1427, 10.1520/JFS13547J · doi:10.1520/jfs13547j
- Heifetz, 1995, Pathological case of the month. Histiocytoid (oncocytic) cardiomyopathy, Arch Pediatr Adolesc Med, 149, 464, 10.1001/archpedi.1995.02170160118020 · doi:10.1001/archpedi.1995.02170160118020
- Suarez, 1987, Foamy myocardial transformation of infancy: an inherited disease, J Clin Pathol, 40, 329, 10.1136/jcp.40.3.329 · doi:10.1136/jcp.40.3.329
- Franciosi, 1988, Oncocytic cardiomyopathy syndrome, Hum Pathol, 19, 1361, 10.1016/S0046-8177(88)80296-X · doi:10.1016/s0046-8177(88)80296-x
- Grech, 2000, Cardiac malformations associated with the congenital nephrotic syndrome, Pediatr Nephrol, 14, 1115, 10.1007/s004670000401 · doi:10.1007/s004670000401
- Ferrans, 1989, Pathologic anatomy of the dilated cardiomyopathies, Am J Cardiol, 64, 9C, 10.1016/0002-9149(89)90677-2 · doi:10.1016/0002-9149(89)90677-2
- Saffitz, 1983, Histiocytoid cardiomyopathy: a cause of sudden death in apparently healthy infants, Am J Cardiol, 52, 215, 10.1016/0002-9149(83)90103-0 · doi:10.1016/0002-9149(83)90103-0
- Kauffman, 1972, Idiopathic infantile cardiomyopathy with involvement of the conduction system, Am J Cardiol, 30, 648, 10.1016/0002-9149(72)90603-0 · doi:10.1016/0002-9149(72)90603-0
- Maurer, 2019, Tafamidis for transthyretin amyloid cardiomyopathy, N Engl J Med, 380, 196, 10.1056/NEJMc1814074 · doi:10.1056/nejmc1814074
- Mueller, 2000, Symptomatic ischemic heart disease resulting from obstructive intramural coronary amyloidosis, Am J Med, 109, 181, 10.1016/S0002-9343(00)00471-X · doi:10.1016/s0002-9343(00)00471-x
- Mathew, 1997, Symptomatic conduction system disease in cardiac amyloidosis, Am J Cardiol, 80, 1491, 10.1016/S0002-9149(97)82785-3 · doi:10.1016/s0002-9149(97)82785-3
- Rezk, 2018, Role of implantable intracardiac defibrillators in patients with cardiac immunoglobulin light chain amyloidosis, Br J Haematol, 182, 145
- Eriksson, 1984, The sinoatrial node in familial amyloidosis with polyneuropathy. A clinico-pathological study of nine cases from northern Sweden, Virchows Arch A Pathol Anat Histopathol, 402, 239, 10.1007/BF00695078 · doi:10.1007/bf00695078
- Eriksson, 1984, Pacemaker treatment in familial amyloidosis with polyneuropathy, Pacing Clin Electrophysiol, 7, 702, 10.1111/j.1540-8159.1984.tb05599.x · doi:10.1111/j.1540-8159.1984.tb05599.x
- Olofsson, 1983, The sick sinus syndrome in familial amyloidosis with polyneuropathy, Int J Cardiol, 4, 71, 10.1016/0167-5273(83)90217-6 · doi:10.1016/0167-5273(83)90217-6
- Barbhaiya, 2016, Electrophysiologic assessment of conduction abnormalities and atrial arrhythmias associated with amyloid cardiomyopathy, Heart Rhythm, 13, 383, 10.1016/j.hrthm.2015.09.016 · doi:10.1016/j.hrthm.2015.09.016
- Capone, 1972, Systemic amyloidosis, functional coronary insufficiency, and autonomic impairment, Ann Intern Med, 76, 599, 10.7326/0003-4819-76-4-599 · doi:10.7326/0003-4819-76-4-599
- Gertz, 1985, Worsening of congestive heart failure in amyloid heart disease treated by calcium channel-blocking agents, Am J Cardiol, 55, 1645, 10.1016/0002-9149(85)90995-6 · doi:10.1016/0002-9149(85)90995-6
- Griffiths, 1982, Cardiac amyloidosis with asymmetrical septal hypertrophy and deterioration after nifedipine, Thorax, 37, 711, 10.1136/thx.37.9.711 · doi:10.1136/thx.37.9.711
- Tan, 2016, Catheter ablation for atrial arrhythmias in patients with cardiac amyloidosis, J Cardiovasc Electrophysiol, 27, 1167, 10.1111/jce.13046 · doi:10.1111/jce.13046
- Dubrey, 1998, The clinical features of immunoglobulin light-chain (AL) amyloidosis with heart involvement, QJM, 91, 141, 10.1093/qjmed/91.2.141 · doi:10.1093/qjmed/91.2.141
- Lin, 2010, Successful termination of a ventricular arrhythmia by implantable cardioverter defibrillator therapy in a patient with cardiac amyloidosis: insight into mechanisms of sudden death, Eur Heart J, 31, 1538, 10.1093/eurheartj/ehp592 · doi:10.1093/eurheartj/ehp592
- Patel, 2014, Life-saving implantable cardioverter defibrillator therapy in cardiac AL amyloidosis, BMJ Case Rep, 2014, 10.1136/bcr-2014-206600 · doi:10.1136/bcr-2014-206600
- Tukkie, 2004, Delay in right ventricular activation contributes to Brugada syndrome, Circulation, 109, 1272, 10.1161/01.CIR.0000118467.53182.D1 · doi:10.1161/01.cir.0000118467.53182.d1
- Papavassiliu, 2010, Spontaneous type 1 electrocardiographic pattern is associated with cardiovascular magnetic resonance imaging changes in Brugada syndrome, Heart Rhythm, 7, 1790, 10.1016/j.hrthm.2010.09.004 · DOI
- Ohkubo, 2010, Right ventricular histological substrate and conduction delay in patients with Brugada syndrome, Int Heart J, 51, 17, 10.1536/ihj.51.17 · doi:10.1536/ihj.51.17
- Kharbanda, 2017, Long QT syndrome and left ventricular noncompaction in 4 family members across 2 generations with KCNQ1 mutation, Eur J Med Genet, 60, 233, 10.1016/j.ejmg.2017.02.003 · doi:10.1016/j.ejmg.2017.02.003
- Nakashima, 2013, A left ventricular noncompaction in a patient with long QT syndrome caused by a KCNQ1 mutation: a case report, Heart Vessels, 28, 126, 10.1007/s00380-012-0235-8 · doi:10.1007/s00380-012-0235-8
- Ogawa, 2009, Isolated non-compaction of the ventricular myocardium associated with long QT syndrome: a report of 2 cases, Circ J, 73, 2169, 10.1253/circj.CJ-08-0339 · doi:10.1253/circj.cj-08-0339
- Murakami, 2003, Identification and characterization of the murine TRPM4 channel, Biochem Biophys Res Commun, 307, 522, 10.1016/S0006-291X(03)01186-0 · doi:10.1016/s0006-291x(03)01186-0
- Liu, 2013, Molecular genetics and functional anomalies in a series of 248 Brugada cases with 11 mutations in the TRPM4 channel, PLoS One, 8, e54131, 10.1371/journal.pone.0054131 · doi:10.1371/journal.pone.0054131
- Saito, 2018, TRPM4 Mutation in Patients With Ventricular Noncompaction and Cardiac Conduction Disease, Circ Genom Precis Med, 11, e002103, 10.1161/CIRCGEN.118.002103 · doi:10.1161/circgen.118.002103
- Posch, 2009, Genetic deletion of arginine 14 in phospholamban causes dilated cardiomyopathy with attenuated electrocardiographic R amplitudes, Heart Rhythm, 6, 480, 10.1016/j.hrthm.2009.01.016 · doi:10.1016/j.hrthm.2009.01.016
- Groeneweg, 2013, Left-dominant arrhythmogenic cardiomyopathy in a large family: associated desmosomal or nondesmosomal genotype?, Heart Rhythm, 10, 548, 10.1016/j.hrthm.2012.12.020 · doi:10.1016/j.hrthm.2012.12.020
- Groeneweg, 2013, Arrhythmogenic right ventricular dysplasia/cardiomyopathy according to revised 2010 task force criteria with inclusion of non-desmosomal phospholamban mutation carriers, Am J Cardiol, 112, 1197, 10.1016/j.amjcard.2013.06.017 · doi:10.1016/j.amjcard.2013.06.017
- Bartram, 2007, Primary noncompaction of the ventricular myocardium from the morphogenetic standpoint, Pediatr Cardiol, 28, 325, 10.1007/s00246-006-0054-8 · doi:10.1007/s00246-006-0054-8
- Finsterer, 2017, Left ventricular noncompaction cardiomyopathy: cardiac, neuromuscular, and genetic factors, Nat Rev Cardiol, 14, 224, 10.1038/nrcardio.2016.207 · doi:10.1038/nrcardio.2016.207
- Kawel, 2012, Trabeculated (noncompacted) and compact myocardium in adults: the multi-ethnic study of atherosclerosis, Circ Cardiovasc Imaging, 5, 357, 10.1161/CIRCIMAGING.111.971713 · doi:10.1161/circimaging.111.971713
- Weir-McCall, 2016, Left ventricular noncompaction: anatomical phenotype or distinct cardiomyopathy?, J Am Coll Cardiol, 68, 2157, 10.1016/j.jacc.2016.08.054 · doi:10.1016/j.jacc.2016.08.054
- Rhee, 2017, Navigating genetic and phenotypic uncertainty in left ventricular noncompaction, Circ Cardiovasc Genet, 10, e001857, 10.1161/CIRCGENETICS.117.001857 · doi:10.1161/circgenetics.117.001857
- Bainbridge, 2015, Loss of function mutations in NNT are associated with left ventricular noncompaction, Circ Cardiovasc Genet, 8, 544, 10.1161/CIRCGENETICS.115.001026 · doi:10.1161/circgenetics.115.001026
- Andreini, 2016, Long-term prognostic value of cardiac magnetic resonance in left ventricle noncompaction: a prospective multicenter study, J Am Coll Cardiol, 68, 2166, 10.1016/j.jacc.2016.08.053 · doi:10.1016/j.jacc.2016.08.053
- Muser, 2017, Ventricular arrhythmias associated with left ventricular noncompaction: electrophysiologic characteristics, mapping, and ablation, Heart Rhythm, 14, 166, 10.1016/j.hrthm.2016.11.014 · doi:10.1016/j.hrthm.2016.11.014
- Sidhu, 2014, Defining left ventricular noncompaction using cardiac computed tomography, J Thorac Imaging, 29, 60, 10.1097/RTI.0b013e31828e9b3d · doi:10.1097/rti.0b013e31828e9b3d
- Kawel-Boehm, 2017, Hypertrabeculated left ventricular myocardium in relationship to myocardial function and fibrosis: the Multi-Ethnic Study of Atherosclerosis, Radiology, 284, 667, 10.1148/radiol.2017161995 · doi:10.1148/radiol.2017161995
Cited by
2021 ESC guidelines for the diagnosis and treatment of acute and chronic heart failure
McDonagh, T. A., Metra, M., Adamo, M., Gardner, R. S., Baumbach, A., Böhm, M., Burri, H., Butler, J., Čelutkienė, J., Chioncel, O., et al. (2021) · European Heart Journal · doi:10.1093/eurheartj/ehab368 · Cited by 13,344
Heidenreich, P. A., Bozkurt, B., Aguilar, D., Allen, L. A., Byun, J. J., Colvin, M. M., Deswal, A., Drazner, M. H., Dunlay, S. M., Evers, L. R., et al. (2022) · Circulation · doi:10.1161/cir.0000000000001063 · Cited by 4,922
Self-citation2023 ESC guidelines for the management of cardiomyopathies
Arbelo, E., Protonotarios, A., Gimeno, J. R., Arbustini, E., Barriales-Villa, R., Basso, C., Bezzina, C. R., Biagini, E., Blom, N. A., de Boer, R. A., et al. (2023) · European Heart Journal · doi:10.1093/eurheartj/ehad194 · Cited by 2,844
Self-citation2022 AHA/ACC/HFSA Guideline for the Management of Heart Failure
Heidenreich, P. A., Bozkurt, B., Aguilar, D., Allen, L. A., Byun, J. J., Colvin, M. M., Deswal, A., Drazner, M. H., Dunlay, S. M., Evers, L. R., et al. (2022) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2021.12.012 · Cited by 2,862
Self-citationSmith, E. D., Lakdawala, N. K., Papoutsidakis, N., Aubert, G., Mazzanti, A., McCanta, A. C., Agarwal, P. P., Arscott, P., Dellefave-Castillo, L. M., Vorovich, E. E., et al. (2020) · Circulation · doi:10.1161/circulationaha.119.044934 · Cited by 504
Self-citation
Metadata
- Authors
- Towbin, J. A., McKenna, W. J., Abrams, D. J., Ackerman, M. J., Calkins, H., Darrieux, F. C. C., Daubert, J. P., de Chillou, C., DePasquale, E. C., Desai, M. Y., Estes, N. A. M., Hua, W., Indik, J. H., Ingles, J., James, C. A., John, R. M., Judge, D. P., Keegan, R., Krahn, A. D., Link, M. S., Marcus, F. I., McLeod, C. J., Mestroni, L., Priori, S. G., Saffitz, J. E., Sanatani, S., Shimizu, W., van Tintelen, J. P., Wilde, A. A. M., & Zareba, W.
- Venue
- Heart Rhythm
- Publisher
- Elsevier BV
- Published
- 2019-11
- Volume
- 16
- Issue
- 11
- Pages
- e301-e372
- ISSN
- 1547-5271 (ISSN-L)
- OpenAlex
- W2944140727
- Cited by
- 846
- References
- 465
- FWCI
- 63.74
- Type
- journal article
- Language
- en
- Open access
- bronze
- Licence
- www.elsevier.com/tdm/userlicense/1.0/ (tdm)
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
How to cite
Cite CoChat for the checks on this page, and the paper for its own findings.
- This page
- CoChat. (2026). Citation check: 2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy. https://app.cochat.ai/papers/10.1016/j.hrthm.2019.05.007
- The paper
- Towbin, J. A., McKenna, W. J., Abrams, D. J., Ackerman, M. J., Calkins, H., Darrieux, F. C. C., Daubert, J. P., de Chillou, C., DePasquale, E. C., Desai, M. Y., Estes, N. A. M., Hua, W., Indik, J. H., Ingles, J., James, C. A., John, R. M., Judge, D. P., Keegan, R., Krahn, A. D., . . . Zareba, W. (2019). 2019 HRS expert consensus statement on evaluation, risk stratification, and management of arrhythmogenic cardiomyopathy. Heart Rhythm. https://doi.org/10.1016/j.hrthm.2019.05.007
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.