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The DOI matches its Crossref record; 16 of the 16 references it cites were found in a registry or an index.

Uncovering specificity of endogenous TAU aggregation in a human iPSC-neuron TAU seeding model

Manos, Justine D., Preiss, Christina N., Venkat, Nandini, Tamm, Joseph, Reinhardt, Peter, Kwon, Taekyung, Wu, Jessica, Winter, Allison D., Jahn, Thomas R., Yanamandra, Kiran, Titterton, Katherine, Karran, Eric, Langlois, Xavier·2022·iScience
DOI 10.1016/j.isci.2021.103658PMC PMC8761709
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  • Modeling 4R tauopathy in human neurons and astrocytes has been implemented, providing a unique platform to investigate the molecular and cellular mechanisms underlying the development and progression of tau pathology with both 3R- and 4R-tau isoforms as seen in FTLD and AD patients.

    “Recently, modeling 4 R tauopathy in human neurons53,83,110,111 and astrocytes110 has been implemented, which provides a unique platform to investigate the molecular and cellular mechanisms underlying the development and progression of tau pathology with both 3R- and 4R-tau isoforms as seen in FTLD and AD patients.”

    From GRAMD1B is a regulator of lipid homeostasis, autophagic flux and phosphorylated tau (2025) · cites this paper as [111]

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Research works printed but not linked: 46

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Metadata

Authors
Manos, J. D., Preiss, C. N., Venkat, N., Tamm, J., Reinhardt, P., Kwon, T., Wu, J., Winter, A. D., Jahn, T. R., Yanamandra, K., Titterton, K., Karran, E., & Langlois, X.
Venue
iScience
Publisher
Elsevier BV
Published
2022-01
Volume
25
Issue
1
Pages
103658
ISSN
2589-0042
DOI
10.1016/j.isci.2021.103658
PMC
PMC8761709
Cited by
36
References
16
Language
en
Licence
Funders
AbbVie

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