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Human hibernating myocardium is jeopardized by apoptotic and autophagic cell death

Elsässer, Albrecht, Vogt, Achim M, Nef, Holger, Kostin, Sawa, Möllmann, Helge, Skwara, Woitek, Bode, Christoph, Hamm, Christian, Schaper, Jutta·2004·Journal of the American College of Cardiology
DOI 10.1016/j.jacc.2004.02.053
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Research works printed but not linked: 30

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  3. Heusch, 1998, Hibernating myocardium, Physiol Rev, 78, 1055, 10.1152/physrev.1998.78.4.1055 · DOI
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  10. Schiller, 1989, American Society of Echocardiography Committee on Standards Subcommittee on Quantitation of Two-Dimensional Echocardiograms, J Am Soc Echocardiogr, 2, 358, 10.1016/S0894-7317(89)80014-8 · DOI
  11. Thomas, 2002, Dissociation of regional adaptations to ischemia and global myolysis in an accelerated swine model of chronic hibernating myocardium, Circ Res, 91, 970, 10.1161/01.RES.0000040396.79379.77 · DOI
  12. Klionsky, 2000, Autophagy as a regulated pathway of cellular degradation, Science, 290, 1717, 10.1126/science.290.5497.1717 · DOI
  13. Ciechanover, 1994, The ubiquitin-mediated proteolytic pathway, FASEB J, 8, 182, 10.1096/fasebj.8.2.8119489 · DOI
  14. Blommaart, 1997, Autophagic proteolysis, Histochem J, 29, 365, 10.1023/A:1026486801018 · DOI
  15. Orlowski, 1999, The role of the ubiquitin-proteasome pathway in apoptosis, Cell Death Differ, 6, 303, 10.1038/sj.cdd.4400505 · DOI
  16. Field, 1997, Inappropriate ubiquitin conjugation, Cardiovasc Res, 33, 8, 10.1016/S0008-6363(96)00141-1 · DOI
  17. Bence, 2001, Impairment of the ubiquitin-proteasome system by protein aggregation, Science, 292, 1552, 10.1126/science.292.5521.1552 · DOI
  18. Hein, 2003, Progression from compensated hypertrophy to failure in pressure-overloaded human heart, Circulation, 107, 984, 10.1161/01.CIR.0000051865.66123.B7 · DOI
  19. Ohno, 1998, ‘Apoptotic’ myocytes in the infarct area in rabbit hearts may be oncotic myocytes with DNA fragmentation, Circulation, 98, 1422, 10.1161/01.CIR.98.14.1422 · DOI
  20. Kanoh, 1999, Significance of myocytes with positive DNA in situ nick end-labeling (TUNEL) in hearts with dilated cardiomyopathy, Circulation, 99, 2757, 10.1161/01.CIR.99.21.2757 · DOI
  21. Webster, 1999, Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53, J Clin Invest, 104, 239, 10.1172/JCI5871 · DOI
  22. Elsässer, 2002, Severe energy deprivation of human hibernating myocardium as possible common pathomechanism of contractile dysfunction, structural degeneration and cell death, J Am Coll Cardiol, 39, 1189, 10.1016/S0735-1097(02)01735-7 · DOI
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Metadata

Authors
Elsässer, A., Vogt, A. M., Nef, H., Kostin, S., Möllmann, H., Skwara, W., Bode, C., Hamm, C., & Schaper, J.
Venue
Journal of the American College of Cardiology
Publisher
Elsevier BV
Published
2004-06
Volume
43
Issue
12
Pages
2191-2199
ISSN
0735-1097
DOI
10.1016/j.jacc.2004.02.053
Cited by
114
References
0
Language
en
Licence

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