The DOI matches its Crossref record; 61 of the 61 references it cites were found in a registry or an index.
Viral activation of cellular metabolism
Open accessAbstract
To ensure optimal environments for their replication and spread, viruses have evolved to alter many host cell pathways. In the last decade, metabolomic studies have shown that eukaryotic viruses induce large-scale alterations in host cellular metabolism. Most viruses examined to date induce aerobic glycolysis also known as the Warburg effect. Many viruses tested also induce fatty acid synthesis as well as glutaminolysis. These modifications of carbon source utilization by infected cells can increase available energy for virus replication and virion production, provide specific cellular substrates for virus particles and create viral replication niches while increasing infected cell survival. Each virus species also likely requires unique metabolic changes for successful spread and recent research has identified additional virus-specific metabolic changes induced by many virus species. A better understanding of the metabolic alterations required for each virus may lead to novel therapeutic approaches through targeted inhibition of specific cellular metabolic pathways.
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1 retracted
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Reciprocal regulation of STING and TCR signaling by mTORC1 for T-cell activation and function
Imanishi, T., Unno, M., Kobayashi, W., Yoneda, N., Matsuda, S., Ikeda, K., Hoshii, T., Hirao, A., Miyake, K., Barber, G. N., et al. (2019) · Life Science Alliance · doi:10.26508/lsa.201800282 · Cited by 62
Metadata
- Authors
- Sanchez, E. L., & Lagunoff, M.
- Venue
- Virology
- Publisher
- Elsevier BV
- Published
- 2015-05
- Volume
- 479-480
- Pages
- 609-618
- ISSN
- 0042-6822 (ISSN-L)
- OpenAlex
- W2039910403
- Cited by
- 560
- References
- 61
- FWCI
- 8.15
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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