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Alzheimer presenilins in the nuclear membrane, interphase kinetochores, and centrosomes suggest a role in chromosome segregation.

Li, Jinhe, Xu, Min, Zhou, Hui, Ma, Jianyi, Potter, Huntington·1997·Cell
DOI 10.1016/s0092-8674(00)80356-6
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  1. Abraham, 1988, Immunochemical identification of the serine protease inhibitor a1-antichymotrypsin in the brain amyloid deposits of Alzheimer's disease, Cell, 52, 487, 10.1016/0092-8674(88)90462-X · DOI
  2. Artavanis-Tsakonas, 1995, Notch signaling, Science, 268, 225, 10.1126/science.7716513 · DOI
  3. Ashall, 1994, Role of the beta-amyloid precursor protein in Alzheimer's disease, Trends Biochem. Sci., 19, 42, 10.1016/0968-0004(94)90173-2 · DOI
  4. Blair Zajdel, 1988, The intracellular distribution of the transformation-associated protein p53 in adenovirus-transformed rodent cells, Oncogene, 2, 579
  5. Brenner, 1981, Kinetochore structure, duplication, and distribution in mammalian cells, J. Cell Biol., 91, 95, 10.1083/jcb.91.1.95 · DOI
  6. Brown, 1997, The SREBP pathway, Cell, 89, 331, 10.1016/S0092-8674(00)80213-5 · DOI · Paper page
  7. Courvalin, 1992, The lamin B receptor of the inner nuclear membrane undergoes mitosis-specific phosphorylation and is a substrate for p34cdc2-type protein kinase, J. Biol. Chem., 267, 19035, 10.1016/S0021-9258(18)41734-6 · DOI
  8. Das, 1995, Expression of the Alzheimer amyloid-promoting factor antichymotrypsin is induced in human astrocytes by IL-1, Neuron, 14, 447, 10.1016/0896-6273(95)90300-3 · DOI
  9. Deng, 1996, Alzheimer-associated presenilin-2 confers increased sensitivity to apoptosis in PC12 cells, FEBS Letters, 397, 50, 10.1016/S0014-5793(96)01142-8 · DOI
  10. De Strooper, 1997, Phosphorylation, subcellular localization, and membrane orientation of the Alzheimer's disease-associated presenilins, J. Biol. Chem., 272, 3590, 10.1074/jbc.272.6.3590 · DOI
  11. Dewji, 1996, Genetic clues to Alzheimer's disease, Science, 271, 159, 10.1126/science.271.5246.159 · DOI
  12. Doan, 1996, Protein topology of presenilin 1, Neuron, 17, 1023, 10.1016/S0896-6273(00)80232-9 · DOI
  13. Donehower, 1995, Deficiency of p53 accelerates mammary tumorigenesis in Wnt-1 transgeneic mice and promotes chromosomal instability, Genes Dev., 9, 882, 10.1101/gad.9.7.882 · DOI
  14. Duff, 1996, Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1, Nature, 383, 710, 10.1038/383710a0 · DOI
  15. Elder, 1996, Identification and neuron specific expression of the S182/presenilin 1 protein in human and rodent brains, J. Neurosci., 45, 308
  16. Fukawawa, 1996, Abnormal centrosome amplification in the absence of p53, Science, 271, 1744, 10.1126/science.271.5256.1744 · DOI
  17. Griffin, 1989, Brain interleukin-1 and S100 immunoreactivity elevated in Down's syndrome and Alzheimer's disease, Proc. Natl. Acad. Sci. USA, 86, 7611, 10.1073/pnas.86.19.7611 · DOI · Paper page
  18. Haass, 1996, Presenile because of presenilin, Curr. Opin. Neurol., 9, 254, 10.1097/00019052-199608000-00002 · DOI
  19. Koff, 1992, Formation and activation of a cyclin E-cdk2 complex during the G1 phase of the human cell cycle, Science, 257, 1689, 10.1126/science.1388288 · DOI
  20. Kopan, 1996, Signal transduction by activated mNotch, Proc. Natl. Acad. Sci. USA, 93, 1683, 10.1073/pnas.93.4.1683 · DOI
  21. Kovacs, 1996, Alzheimer-associated presenilins 1 and 2, Nature Med., 2, 224, 10.1038/nm0296-224 · DOI
  22. LeBlanc, 1995, Increased production of 4KDa amyloid beta peptide in serum-deprived human primary neuron cultures, J. Neurosci., 15, 7837, 10.1523/JNEUROSCI.15-12-07837.1995 · DOI
  23. Lengauer, 1997, Genetic instability on colorectal cancers, Nature, 386, 623, 10.1038/386623a0 · DOI
  24. Levitan, 1995, Facilitation of lin-12-mediated signaling by sel-12, a Caenorhabditis elegans S182 Alzheimer's disease gene, Nature, 377, 351, 10.1038/377351a0 · DOI
  25. Levy-Lahad, 1995, A familial Alzheimer's disease locus on chromosome 1, Science, 269, 970, 10.1126/science.7638621 · DOI
  26. Li, 1996, Membrane topology of the C. elegans sel-12 presenilin, Neuron, 17, 1015, 10.1016/S0896-6273(00)80231-7 · DOI
  27. Li, 1995, Identification and expression analysis of a potential familial Alzheimer's disease gene on chromosome 1 related to AD3, Proc. Natl. Acad. Sci. USA, 92, 12180, 10.1073/pnas.92.26.12180 · DOI
  28. Ma, 1996, Alzheimer Aβ neurotoxicity, Neurobiol. Aging, 17, 773, 10.1016/0197-4580(96)00112-1 · DOI · Paper page
  29. Martins, 1995, High levels of amyloid-β protein from S182 (Glu246) familial Alzheimer's cells, NeuroReport, 7, 217, 10.1097/00001756-199512000-00052 · DOI
  30. McKeon, 1983, Autoimmune response directed against conserved determinants on nuclear envelope proteins in a patient with linear scleroderma, Proc. Natl. Acad. Sci. USA, 80, 4374, 10.1073/pnas.80.14.4374 · DOI
  31. Mercken, 1996, Characterization of human presenilin 1 using N-terminal specific monoclonal antibodies, FEBS Lett., 389, 297, 10.1016/0014-5793(96)00608-4 · DOI
  32. Mitchell, 1996, The mammalian centromere, Mutation Research, 372, 153, 10.1016/S0027-5107(96)00135-2 · DOI
  33. Moroi, 1981, Distribution of kinetochore (centromere) antigen in mammalian cell nuclei, J. Cell Biol., 90, 254, 10.1083/jcb.90.1.254 · DOI
  34. Moussaoui, 1996, Immunohistochemical analysis of presenilin-1 expression in the mouse brain, FEBS Lett., 383, 219, 10.1016/0014-5793(96)00250-5 · DOI
  35. Potter, 1991, Review and hypothesis, Am. J. Hum. Genet., 48, 1192
  36. Potter, 1996, Alzheimer's disease, Down's syndrome, and chromosome segregation, The Lancet, 348, 66, 10.1016/S0140-6736(05)64399-1 · DOI
  37. Potter, 1984, Enhancer-dependent expression of human kappa immunoglobulin genes introduced into mouse pre-B lymphocytes by electroporation, Proc. Natl. Acad. Sci. USA, 81, 7161, 10.1073/pnas.81.22.7161 · DOI
  38. Potter, H., Ma, J., Das, S., Geller, L.N., Benjamin, M., Kayyali, U.S. and Dressler, D. (1995). Beyond β-protein: new steps in the pathogenic pathway to Alzheimer's disease. In Research Advances in Alzheimer's Disease and Related Disorders, K. Iqbal, J.A. Mortimer, B. Winblad, and W.H. Wisniewski, eds. (New York: John Wiley and Sons, pp. 643–654).
  39. Sanan, 1994, Apolipoprotein E associates with β amyloid peptide of Alzheimer's disease to form novel monofibrils, J. Clin. Invest., 94, 860, 10.1172/JCI117407 · DOI · Paper page
  40. Schellenberg, 1995, Genetic dissection of Alzheimer disease, a heterogeneous disorder, Proc. Natl. Acad. Sci. USA, 92, 8552, 10.1073/pnas.92.19.8552 · DOI
  41. Schellenberg, 1993, Genetic linkage evidence for a familial Alzheimer's disease locus on chromosome 14, Science, 258, 668, 10.1126/science.1411576 · DOI
  42. Schweber, 1985, A possible unitary genetic hypothesis for Alzheimer's disease and Down syndrome, Ann. NY Acad. Sci., 450, 223, 10.1111/j.1749-6632.1985.tb21495.x · DOI
  43. Selkoe, 1994, Cell biology of the amyloid β-protein precursor and the mechanism of Alzheimer's disease, Annu. Rev. Cell Biol., 10, 373, 10.1146/annurev.cb.10.110194.002105 · DOI
  44. Shen, 1997, Skeletal and CNS defects in presenilin-1-deficient mice, Cell, 89, 629, 10.1016/S0092-8674(00)80244-5 · DOI · Paper page
  45. Stone, 1995, Sex chromosome aneuploidy and aging, Mutation Res., 338, 107, 10.1016/0921-8734(95)00016-Y · DOI
  46. Strittmatter, 1995, Apolipoprotein E and Alzheimer's disease, Proc. Natl. Acad. Sci. USA, 92, 4725, 10.1073/pnas.92.11.4725 · DOI
  47. Suzuki, 1989, SPXX, a frequent sequence motif in gene regulatory proteins, J. Mol. Biol., 207, 61, 10.1016/0022-2836(89)90441-5 · DOI
  48. Takashima, 1996, Localization of Alzheimer-associated presenilin 1 in transfected COS-7 cells, Biochem. Biophys. Res. Comm., 227, 423, 10.1006/bbrc.1996.1523 · DOI
  49. Teller, 1996, Presence of soluble amyloid β-peptide precedes amyloid plaque formation in Down's syndrome, Nature Medicine, 2, 93, 10.1038/nm0196-93 · DOI
  50. Thinakaran, 1996, Endoproteolysis of presenilin 1 and accumulation of processed derivatives in vivo, Neuron, 17, 181, 10.1016/S0896-6273(00)80291-3 · DOI
  51. Tuffanelli, 1983, Anti-centrosome and anti-centriole antibodies in the scleroderma spectrum, Arch. Dermatol., 119, 560, 10.1001/archderm.1983.01650310022004 · DOI
  52. Vincent, 1996, Mitotic mechanisms in Alzheimer's disease?, J. Cell Biol., 132, 413, 10.1083/jcb.132.3.413 · DOI · Paper page
  53. Vito, 1996, Interfering with apoptosis, Science, 271, 521, 10.1126/science.271.5248.521 · DOI
  54. Vito, 1996, Requirement of the familial Alzheimer's disease gene PS2 for apoptosis, J. Biol. Chem., 271, 31025, 10.1074/jbc.271.49.31025 · DOI
  55. Walter, 1996, The Alzheimer's disease-associated presenilins are differentially phosphorylated proteins located predominantly within the endoplasmic reticulum, Mol. Med., 2, 673, 10.1007/BF03401652 · DOI
  56. Westendorf, 1994, Cloning of cDNAs for M-phase phosphoproteins recognized by the MPM2 monoclonal antibody and determination of the phosphorylated epitope, Proc. Natl. Acad. Sci. USA, 91, 714, 10.1073/pnas.91.2.714 · DOI
  57. Winey, 1993, NDC1, J. Cell Biol., 122, 743, 10.1083/jcb.122.4.743 · DOI
  58. Wisniewski, 1994, Acceleration of Alzheimer's fibril formation by apolipoprotein E in vitro, Am. J. Pathol., 145, 1030
  59. Wolozin, 1996, Participation of presenilin 2 in apoptosis, Science, 274, 1710, 10.1126/science.274.5293.1710 · DOI
  60. Yankner, 1996, Mechanisms of neuronal degeneration in Alzheimer's disease, Neuron, 16, 921, 10.1016/S0896-6273(00)80115-4 · DOI
  61. Ye, 1994, Primary structure analysis and lamin B and DNA binding of human LBR, an integral protein of the nuclear envelope inner membrane, J. Biol. Chem., 269, 11306, 10.1016/S0021-9258(19)78126-5 · DOI

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Metadata

Authors
Li, J., Xu, M., Zhou, H., Ma, J., & Potter, H.
Venue
Cell
Publisher
Elsevier BV
Published
1997-09
Volume
90
Issue
5
Pages
917-927
ISSN
0092-8674
DOI
10.1016/s0092-8674(00)80356-6
Cited by
172
References
8
Language
en
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