The DOI matches its Crossref record; 34 of the 34 references it cites were found in a registry or an index.
CHIP Is Associated with Parkin, a Gene Responsible for Familial Parkinson's Disease, and Enhances Its Ubiquitin Ligase Activity
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Mutation in the α-Synuclein Gene Identified in Families with Parkinson's Disease
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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism
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AlaSOPro mutation in the gene encoding α-synuclein in Parkinson's disease
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The Hsp70 and Hsp60 Chaperone Machines
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Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
Shimura, H., Hattori, N., Kubo, S.-I., Mizuno, Y., Asakawa, S., Minoshima, S., Shimizu, N., Iwai, K., Chiba, T., Tanaka, K., et al. (2000) · Nature Genetics · doi:10.1038/77060 · Cited by 2,024 · cited 1×
“…CREST Along with other researchers, we have demonstratedJapan Science and Technology Corporation that Parkin has E3 activity and that AR-JP-linked Parkin4-1-8 Honcho, Kawaguchi mutants are defective in E3 activity (Imai et al., 2000;Saitama 332-0012 Shimura et al., 2000; Zhang et al., 2000).”
BackgroundAggresomes, inclusion bodies and protein aggregation
Kopito, R. R. (2000) · Trends in Cell Biology · doi:10.1016/s0962-8924(00)01852-3 · Cited by 1,921
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Chaperone Suppression of α-Synuclein Toxicity in a Drosophila Model for Parkinson's Disease
Auluck, P. K., Chan, H. Y. E., Trojanowski, J. Q., Lee, V. M.-Y., & Bonini, N. M. (2002) · Science · doi:10.1126/science.1067389 · Cited by 1,227 · cited 1×
“This strongly suggests a funca combination of both Parkin and its cofactor CHIP func- tional link between the protein refolding and degradation tion as a ubiquitin ligase, which is able to sufficiently pathways (Auluck et al., 2002; Sherman and Goldberg, ubiquitinate Pael-R in vivo (Figure 6).”
Background
Cited by
One step at a time: endoplasmic reticulum-associated degradation
Vembar, S. S., & Brodsky, J. L. (2008) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm2546 · Cited by 1,299
A Role for Ubiquitin in Selective Autophagy
Kirkin, V., McEwan, D. G., Novak, I., & Dikic, I. (2009) · Molecular Cell · doi:10.1016/j.molcel.2009.04.026 · Cited by 1,239
UPR, autophagy, and mitochondria crosstalk underlies the ER stress response
Senft, D., & Ronai, Z. A. (2015) · Trends in Biochemical Sciences · doi:10.1016/j.tibs.2015.01.002 · Cited by 1,121
Orchestrating the unfolded protein response in health and disease
Kaufman, R. J. (2002) · Journal of Clinical Investigation · doi:10.1172/jci0216886 · Cited by 1,015
The Ubiquitin Proteasome System in Neurodegenerative Diseases
Ciechanover, A., & Brundin, P. (2003) · Neuron · doi:10.1016/s0896-6273(03)00606-8 · Cited by 982
Metadata
- Authors
- Imai, Y., Soda, M., Hatakeyama, S., Akagi, T., Hashikawa, T., Nakayama, K.-I., & Takahashi, R.
- Venue
- Molecular Cell
- Publisher
- Elsevier BV
- Published
- 2002-07
- Volume
- 10
- Issue
- 1
- Pages
- 55-67
- ISSN
- 1097-2765 (ISSN-L)
- OpenAlex
- W2110780830
- Cited by
- 473
- References
- 34
- FWCI
- 23.86
- Type
- journal article
- Language
- en
- Open access
- bronze
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- CoChat. (2026). Citation check: CHIP Is Associated with Parkin, a Gene Responsible for Familial Parkinson's Disease, and Enhances Its Ubiquitin Ligase Activity. https://app.cochat.ai/papers/10.1016/s1097-2765(02)00583-x
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- Imai, Y., Soda, M., Hatakeyama, S., Akagi, T., Hashikawa, T., Nakayama, K.-I., & Takahashi, R. (2002). CHIP Is Associated with Parkin, a Gene Responsible for Familial Parkinson's Disease, and Enhances Its Ubiquitin Ligase Activity. Molecular Cell. https://doi.org/10.1016/s1097-2765(02)00583-x
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