The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.
Loss of Gcn5l2 leads to increased apoptosis and mesodermal defects during mouse development
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Citations
Cites
DNA damage activates p53 through a phosphorylation–acetylation cascade
Sakaguchi, K., Herrera, J. E., Saito, S., Miki, T., Bustin, M., Vassilev, A., Anderson, C. W., & Appella, E. (1998) · Genes & Development · doi:10.1101/gad.12.18.2831 · Cited by 1,204
p53 Sites Acetylated In Vitro by PCAF and p300 Are Acetylated In Vivo in Response to DNA Damage
Liu, L., Scolnick, D. M., Trievel, R. C., Zhang, H. B., Marmorstein, R., Halazonetis, T. D., & Berger, S. L. (1999) · Molecular and Cellular Biology · doi:10.1128/mcb.19.2.1202 · Cited by 776
Cited by
Chromatin modifiers and remodellers: regulators of cellular differentiation
Chen, T., & Dent, S. Y. R. (2014) · Nature Reviews Genetics · doi:10.1038/nrg3607 · Cited by 619
The diverse functions of histone acetyltransferase complexes
Carrozza, M. J., Utley, R. T., Workman, J. L., & Côté, J. (2003) · Trends in Genetics · doi:10.1016/s0168-9525(03)00115-x · Cited by 487
Protein acetylation in metabolism – metabolites and cofactors
Menzies, K. J., Zhang, H., Katsyuba, E., & Auwerx, J. (2016) · Nature Reviews Endocrinology · doi:10.1038/nrendo.2015.181 · Cited by 331
Gcn5 Is Required for PU.1-Dependent IL-9 Induction in Th9 Cells
Goswami, R., & Kaplan, M. H. (2012) · The Journal of Immunology · doi:10.4049/jimmunol.1201496 · Cited by 100
“Gcn5-null embryos die during embryogenesis (28).”
The role of altered protein acetylation in neurodegenerative disease
Kabir, F., Atkinson, R., Cook, A. L., Phipps, A. J., & King, A. E. (2023) · Frontiers in Aging Neuroscience · doi:10.3389/fnagi.2022.1025473 · Cited by 75
Metadata
- Authors
- Xu, W., Edmondson, D. G., Evrard, Y. A., Wakamiya, M., Behringer, R. R., & Roth, S. Y.
- Venue
- Nature Genetics
- Published
- 2000-10
- Volume
- 26
- Issue
- 2
- Pages
- 229-232
- ISSN
- 1061-4036 (ISSN-L)
- 1546-1718
- OpenAlex
- W2076368732
- Cited by
- 269
- References
- 2
- FWCI
- 3.72
- Type
- journal article
- Language
- en
- Open access
- closed
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How to cite
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- This page
- CoChat. (2026). Citation check: Loss of Gcn5l2 leads to increased apoptosis and mesodermal defects during mouse development. https://app.cochat.ai/papers/10.1038/79973
- The paper
- Xu, W., Edmondson, D. G., Evrard, Y. A., Wakamiya, M., Behringer, R. R., & Roth, S. Y. (2000). Loss of Gcn5l2 leads to increased apoptosis and mesodermal defects during mouse development. Nature Genetics. https://doi.org/10.1038/79973
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
