Helicobacter pylori HtrA is a new secreted virulence factor that cleaves E‐cadherin to disrupt intercellular adhesion

Hoy, Benjamin, Löwer, Martin, Weydig, Christiane, Carra, Gert, Tegtmeyer, Nicole, Geppert, Tim, Schröder, Peter, Sewald, Norbert, Backert, Steffen, Schneider, Gisbert, Wessler, Silja·2010·The EMBO Reports
DOI 10.1038/embor.2010.114

Cited by (295)

Items 201–295 of 295

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  12. Tailor-Made Detection of Individual Phosphorylated and Non-Phosphorylated EPIYA-Motifs of Helicobacter pylori Oncoprotein CagA
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  14. Fragment‐Based De Novo Design Reveals a Small‐Molecule Inhibitor of Helicobacter Pylori HtrA
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  15. Pangenomic studies of Helicobacter pylori: a key to understanding pathogenesis and human history
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  17. HtrA-Dependent E-Cadherin Shedding Impairs the Epithelial Barrier Function in Primary Gastric Epithelial Cells and Gastric Organoids
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  24. Gastrointestinal Mucosal Defense System
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  29. Targets Against Helicobacter pylori and Other Tumor-Producing Bacteria
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  30. Inhibition of Listeria Monocytogenes HtrA Protease with Camostat, Gabexate and Nafamostat Mesylates and the Binding Mode of the Inhibitors
    M C Amrutha, Silja Weßler, Karthe Ponnuraj · 2023 · The Protein Journal
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  40. Helicobacter pylori and Related Virulence Factors for Gastrointestinal Diseases
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  42. Bismuth and transition metal complexes as potent anti-Helicobacter pylori agents: A review
    Xiaofeng Zhou, Jianwei Yun · 2025 · Polyhedron
  43. Helicobacter pylori CagA EPIYA Motif Variations Affect Metabolic Activity in B Cells
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  44. Engineering of bacterial outer membrane vesicles
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