The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
Protein accumulation and neurodegeneration in the woozy mutant mouse is caused by disruption of SIL1, a cochaperone of BiP
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Citations
Cites
Polyglutamine expansion down-regulates specific neuronal genes before pathologic changes in SCA1
Lin, X., Antalffy, B., Kang, D., Orr, H. T., & Zoghbi, H. Y. (2000) · Nature Neuroscience · doi:10.1038/72101 · Cited by 374
Cited by
Protein misfolding in the endoplasmic reticulum as a conduit to human disease
Wang, M., & Kaufman, R. J. (2016) · Nature · doi:10.1038/nature17041 · Cited by 1,630
Yoshida, H. (2007) · The FEBS Journal · doi:10.1111/j.1742-4658.2007.05639.x · Cited by 1,086
ER stress and the unfolded protein response in neurodegeneration
Hetz, C., & Saxena, S. (2017) · Nature Reviews Neurology · doi:10.1038/nrneurol.2017.99 · Cited by 1,013
The endoplasmic reticulum and the unfolded protein response
Malhotra, J. D., & Kaufman, R. J. (2007) · Seminars in Cell & Developmental Biology · doi:10.1016/j.semcdb.2007.09.003 · Cited by 986
The impact of the unfolded protein response on human disease
Wang, S., & Kaufman, R. J. (2012) · Journal of Cell Biology · doi:10.1083/jcb.201110131 · Cited by 984
Metadata
- Authors
- Zhao, L., Longo-Guess, C., Harris, B. S., Lee, J.-W., & Ackerman, S. L.
- Venue
- Nature Genetics
- Published
- 2005-08-14
- Volume
- 37
- Issue
- 9
- Pages
- 974-979
- ISSN
- 1061-4036 (ISSN-L)
- 1546-1718
- OpenAlex
- W1975987422
- Cited by
- 214
- References
- 1
- FWCI
- 5.94
- Type
- journal article
- Language
- en
- Open access
- closed
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- CoChat. (2026). Citation check: Protein accumulation and neurodegeneration in the woozy mutant mouse is caused by disruption of SIL1, a cochaperone of BiP. https://app.cochat.ai/papers/10.1038/ng1620
- The paper
- Zhao, L., Longo-Guess, C., Harris, B. S., Lee, J.-W., & Ackerman, S. L. (2005). Protein accumulation and neurodegeneration in the woozy mutant mouse is caused by disruption of SIL1, a cochaperone of BiP. Nature Genetics. https://doi.org/10.1038/ng1620
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
