The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.
Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons
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Citations
Cites
Bender, A., Krishnan, K. J., Morris, C. M., Taylor, G. A., Reeve, A. K., Perry, R. H., Jaros, E., Hersheson, J. S., Betts, J., Klopstock, T., et al. (2006) · Nature Genetics · doi:10.1038/ng1769 · Cited by 1,570
Mitochondrial DNA deletions in human brain: regional variability and increase with advanced age
Corral-Debrinski, M., Horton, T., Lott, M. T., Shoffner, J. M., Flint Beal, M., & Wallace, D. C. (1992) · Nature Genetics · doi:10.1038/ng1292-324 · Cited by 890
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- Khrapko, K., Kraytsberg, Y., de Grey, A., Vijg, J. & Schon, E.A. Aging Cell (in the press).
- Jacobs, H.T. Aging Cell 2, 11–17 (2003). · DOI
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- Itoh, K., Weis, S., Mehraein, P. & Muller-Hocker, J. Neurobiol. Aging 17, 843–848 (1996). · DOI
- Nekhaeva, E., Kraytsberg, Y. & Khrapko, K. Mech. Ageing Dev. 123, 891–898 (2002). · DOI
- Kraytsberg, Y. & Khrapko, K. Expert Rev. Mol. Diagn. 5, 809–815 (2005). · DOI
- Rossignol, R. et al. Biochem. J. 370, 751–762 (2003). · DOI · Paper page
- Khrapko, K. et al. Nucleic Acids Res. 27, 2434–2441 (1999). · DOI · Paper page
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Cited by
Mitochondrial Dysfunction and Oxidative Stress in Neurodegenerative Diseases
Lin, M. T., & Beal, M. F. (2006) · Nature · doi:10.1038/nature05292 · Cited by 6,455
Mitochondria: In Sickness and in Health
Nunnari, J., & Suomalainen, A. (2012) · Cell · doi:10.1016/j.cell.2012.02.035 · Cited by 3,479
Youle, R. J., & Narendra, D. P. (2011) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm3028 · Cited by 3,273
PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin
Narendra, D. P., Jin, S. M., Tanaka, A., Suen, D.-F., Gautier, C. A., Shen, J., Cookson, M. R., & Youle, R. J. (2010) · PLoS Biology · doi:10.1371/journal.pbio.1000298 · Cited by 2,944
The Roles of PINK1, Parkin, and Mitochondrial Fidelity in Parkinson’s Disease
Pickrell, A. M., & Youle, R. J. (2015) · Neuron · doi:10.1016/j.neuron.2014.12.007 · Cited by 2,310
Metadata
- Authors
- Kraytsberg, Y., Kudryavtseva, E., McKee, A. C., Geula, C., Kowall, N. W., & Khrapko, K.
- Venue
- Nature Genetics
- Published
- 2006-04-09
- Volume
- 38
- Issue
- 5
- Pages
- 518-520
- ISSN
- 1061-4036 (ISSN-L)
- 1546-1718
- OpenAlex
- W2015116743
- Cited by
- 887
- References
- 2
- FWCI
- 22.72
- Type
- journal article
- Language
- en
- Open access
- closed
- Licence
- www.springer.com/tdm (tdm)
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- CoChat. (2026). Citation check: Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons. https://app.cochat.ai/papers/10.1038/ng1778
- The paper
- Kraytsberg, Y., Kudryavtseva, E., McKee, A. C., Geula, C., Kowall, N. W., & Khrapko, K. (2006). Mitochondrial DNA deletions are abundant and cause functional impairment in aged human substantia nigra neurons. Nature Genetics. https://doi.org/10.1038/ng1778
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.