Genomic instability in laminopathy-based premature aging

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Items 401–500 of 644

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5. Molecular tools that block maturation of the nuclear lamin A and decelerate cancer cell migration
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6. Hutchinson-Gilford Progeria Syndrome: Challenges at Bench and Bedside
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7. LMNAMutation c.917T>G (p.L306R) Leads to Deleterious Hyper-Assembly of Lamin A/C and Associates with Severe Right Ventricular Cardiomyopathy and Premature Aging
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8. Accumulation of prelamin A induces premature aging through mTOR overactivation
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9. Prelamin A and ZMPSTE24 in premature and physiological aging
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10. Microcephalia with mandibular and dental dysplasia in adult Zmpste24‐deficient mice
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11. Pluripotent stem cells to model Hutchinson-Gilford progeria syndrome (HGPS): Current trends and future perspectives for drug discovery
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12. Loss ofVHLpromotes progerin expression, leading to impaired p14/ARF function and suppression of p53 activity
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13. Lamin A‐mediated nuclear lamina integrity is required for proper ciliogenesis
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14. DNA damage-induced inflammation and nuclear architecture
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15. Alteration of genetic recombination and double-strand break repair in human cells by progerin expression
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16. Mechanisms for assembly of the nucleoplasmic reticulum
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17. N6‐isopentenyladenosine improves nuclear shape in fibroblasts from humans with progeroid syndromes by inhibiting the farnesylation of prelamin A
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18. Development and validation of new predictive equations for the resting metabolic rate of older adults aged ≥65 y
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19. Laminopathies: Too Much SUN Is a Bad Thing
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20. Highlights of the 2007 Progeria Research Foundation Scientific Workshop: Progress in Translational Science
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21. Les syndromes lipodystrophiques : des adipopathies congénitales ou acquises
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22. Les lipodystrophies primitives
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23. Consequences of Lmna Exon 4 Mutations in Myoblast Function
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24. Emerging roles of interferon-stimulated gene-15 in age-related telomere attrition, the DNA damage response, and cardiovascular disease
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25. Doxycycline decelerates aging in progeria mice
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26. Advances in basic and clinical research in laminopathies.
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27. Mitotic Defects Lead to Pervasive Aneuploidy and Accompany Loss of RB1 Activity in Mouse LmnaDhe Dermal Fibroblasts
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28. Lamin A-linked progerias: is farnesylation the be all and end all?
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29. Chapter 7 Fraying at the Edge
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30. Stress-induced release of Oct-1 from the nuclear envelope is mediated by JNK phosphorylation of lamin B1
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31. Impaired end joining induces cardiac atrophy in a Hutchinson–Gilford progeria mouse model
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32. Conserved Gene Order at the Nuclear Periphery in Drosophila
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33. The lamin code
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34. Presence and distribution of progerin in HGPS cells is ameliorated by drugs that impact on the mevalonate and mTOR pathways
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35. MicroRNA transcriptome analysis identifies miR-365 as a novel negative regulator of cell proliferation in Zmpste24-deficient mouse embryonic fibroblasts
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36. Myonuclear breakdown in sporadic inclusion body myositis is accompanied by DNA double strand breaks
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37. Towards a Drosophila model of Hutchinson–Gilford progeria syndrome
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38. Effects of p21 deletion in mouse models of premature aging
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39. Genetic differences between primary larynx and pharynx carcinomas and their matched lymph node metastases by multiplex ligation-dependent probe amplification
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40. Modeling the Study of DNA Damage Responses in Mice
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41. Emerging roles of lamins and DNA damage repair mechanisms in ovarian cancer
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42. Corruption of DNA End-Joining in Mammalian Chromosomes by Progerin Expression as Revealed by a Model Cell Culture System
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43. Androgen signaling stabilizes genomes to counteract senescence by promoting XRCC4 transcription
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44. Analysing regenerative potential in zebrafish models of congenital muscular dystrophy
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45. Development of detection method for novel fusion gene using GeneChip exon array
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46. In vitro pathological modelling using patient-specific induced pluripotent stem cells: the case of progeria
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47. Zmpste24−/− Mouse Model for Senescent Wound Healing Research
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48. Altered Nuclear Functions in Progeroid Syndromes: a Paradigm for Aging Research
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49. ‘Relax and Repair’ to restrain aging
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50. miR-342-5p promotes Zmpste24-deficient mouse embryonic fibroblasts proliferation by suppressing GAS2
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51. DNA repair, insulin signaling and sirtuins: at the crossroads between cancer and aging
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52. The dual guardians of cellular stability: exploring nesprin and lamin in senescence
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53. Establishment and Characterization of hTERT Immortalized Hutchinson–Gilford Progeria Fibroblast Cell Lines
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54. NURD keeps chromatin young
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55. Lamines A et syndromes progéroïdes
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56. A reverse genetics cell‐based evaluation of genes linked to healthy human tissue age
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57. Linker histones and chromatin remodelling complexes maintain genome stability and control cellular ageing
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58. Small spaces, big problems: The abnormal nucleoplasm of micronuclei and its consequences
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59. Overexpression of Progerin Results in Impaired Proliferation and Invasion of Non-Small Cell Lung Cancer Cells
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60. MG132 Induces Progerin Clearance and Improves Disease Phenotypes in HGPS-like Patients’ Cells
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61. Nuclear Structure and Chromosome Segregation inDrosophilaMale Meiosis Depend on the Ubiquitin Ligase dTopors
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62. Laminopathies : un seul gène, de nombreuses pathologies
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63. Maillard reaction and aging: can bacteria shed light on the link?
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64. Prelamin A processing and heterochromatin dynamics in laminopathies
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65. Progerin can induce DNA damage in the absence of global changes in replication or cell proliferation
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66. Adenine base editing rescues pathogenic phenotypes in tissue engineered vascular model of Hutchinson-Gilford progeria syndrome
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67. Lamin A/C loss promotes R-loop-mediated genomic instability and poor survival in small-cell lung cancer
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68. Lamin A/C-regulated cysteine catabolic flux modulates stem cell fate through epigenome reprogramming
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69. Hutchinson-Gilford progeria syndrome: Cardiovascular manifestations and treatment
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70. Lamin A to Z in normal aging
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71. Multiplexed profiling of secreted proteins for the detection of potential space biomarkers
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72. Nucleoskeleton dynamics and functions in health and disease
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73. Substrate Topography Modulates Cell Aging on a Progeria Cell Model
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74. Expression of lamin C2 in mammalian oocytes
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75. Lipodystrophic Laminopathies: From Dunnigan Disease to Progeroid Syndromes
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76. Counteracting lysosome defects alleviates the cellular senescence of Hutchinson-Gilford progeria syndrome
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77. Emerging perspectives on laminopathies
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78. Mecanismos de envejecimiento vascular: ¿Qué podemos aprender del síndrome de progeria de Hutchinson-Gilford?
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79. A cytoplasmic escapee: desmin is going nuclear
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80. Clonal hematopoiesis is not prevalent in Hutchinson-Gilford progeria syndrome
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81. The Nuclear Envelope in Ageing and Progeria
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82. Unraveling the Mysteries of Aging Through a Hutchinson–Gilford Progeria Syndrome Model
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83. Identification of FAM96B as a novel prelamin A binding partner
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84. Inactivation of RAD52 and HDF1 DNA repair genes leads to premature chronological aging and cellular instability
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85. Hutchinson-Gilford Progeria Syndrome
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86. Abnormal nuclear morphology is independent of longevity in a zmpste24 -deficient fish model of Hutchinson-Gilford progeria syndrome (HGPS)
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88. Nuclear Morphology Optimized Deep Hybrid Learning (NUMODRIL): A novel architecture for accurate diagnosis/prognosis of Ovarian Cancer
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89. Chk2 deletion rescues bone loss and cellular senescence induced by Bmi1 deficiency via regulation of Cyp1a1
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90. Emergence of CpG-cluster blanket methylation in aged tissues: a novel signature of epigenomic aging
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91. Investigation of splicing changes and post-translational processing of LMNA in sporadic inclusion body myositis.
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92. Physical evidence on desmin–lamin B interaction
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93. Relationship Between DNA Damage and Energy Metabolism: Evidence from DNA Repair Deficiency Syndromes
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94. Accumulation of prelamin A compromises NF-κB-regulated B-lymphopoiesis in a progeria mouse model
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95. Expression of progerin does not result in an increased mutation rate
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96. The Most Severe Paradigm of Early Cardiovascular Disease: Hutchinson-Gilford Progeria. Focus on the Role of Oxidative Stress
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97. Deciphering the molecular interplay between pelvic inflammatory disease (PID) and ovarian cancer (OC)—A network biology approach
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98. Involvement of the nuclear structural proteins in aging-related responses of human skin to the environmental stress
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99. The impact of alterations in lamin A on genome integrity
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100. Nuclear compression-mediated DNA damage drives ATR-dependent Lamin expression and mouse ESC differentiation
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