Soluble endoglin contributes to the pathogenesis of preeclampsia

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Items 601–700 of 1,000

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2. The Roles of Uterine Natural Killer (NK) Cells and KIR/HLA‐C Combination in the Development of Preeclampsia: A Systematic Review
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3. Procyanidin B2 ameliorates endothelial dysfunction and impaired angiogenesis via the Nrf2/PPARγ/sFlt-1 axis in preeclampsia
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4. Prediction of pre-eclampsia in early pregnancy by estimating the spot urinary albumin: creatinine ratio using high-performance liquid chromatography
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5. Preeclampsia Pathogenesis
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6. Activation of villous trophoblastic p38 and ERK1/2 signaling pathways in preterm preeclampsia and HELLP syndrome
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7. Discordant clinical presentations of preeclampsia and intrauterine fetal growth restriction with similar pro- and anti-angiogenic profiles
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13. Circulating soluble endoglin and placental abruption
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16. Maternal Serum Fatty Acid Binding Protein 4 (FABP4) and the Development of Preeclampsia
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19. Maternal choline supplementation during murine pregnancy modulates placental markers of inflammation, apoptosis and vascularization in a fetal sex-dependent manner
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25. Soluble endoglin modulates aberrant cerebral vascular remodeling
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30. Potential targets for the treatment of preeclampsia
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31. Understanding the Link Between the Placenta and Future Cardiovascular Disease
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32. Soluble endoglin production is upregulated by oxysterols but not quenched by pravastatin in primary placental and endothelial cells
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33. AGE s and HMGB 1 Increase Inflammatory Cytokine Production from Human Placental Cells, Resulting in an Enhancement of Monocyte Migration
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34. Endoglin expression in blood and endothelium is differentially regulated by modular assembly of the Ets/Gata hemangioblast code
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36. Could alterations in maternal plasma visfatin concentration participate in the phenotype definition of preeclampsia and SGA?
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38. Research on potential biomarkers in hereditary hemorrhagic telangiectasia
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39. Murine Pregnancy-Specific Glycoprotein 23 Induces the Proangiogenic Factors Transforming-Growth Factor Beta 1 and Vascular Endothelial Growth Factor A in Cell Types Involved in Vascular Remodeling in Pregnancy1
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41. Discovery of the serum biomarker proteins in severe preeclampsia by proteomic analysis
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42. The perinatal implications of angiogenic factors
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43. Onset threshold of the plasma levels of soluble fms-like tyrosine kinase 1/placental growth factor ratio for predicting the imminent onset of preeclampsia within 4 weeks after blood sampling at 19–31 weeks of gestation
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44. From preeclampsia to renal disease: a role of angiogenic factors and the renin-angiotensin aldosterone system?
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46. Quantitative proteomics-based analyses performed on pre-eclampsia samples in the 2004–2020 period: a systematic review
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47. Endoglin for tumor imaging and targeted cancer therapy
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48. Soluble endoglin is an accurate predictor and a pathogenic molecule in pre-eclampsia
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51. AICAR administration ameliorates hypertension and angiogenic imbalance in a model of preeclampsia in the rat
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53. Placenta derived factors involved in the pathogenesis of the liver in the syndrome of haemolysis, elevated liver enzymes and low platelets (HELLP): A review
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57. Renal Involvement in Preeclampsia: Similarities to VEGF Ablation Therapy
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58. Soluble endoglin and transforming growth factor‐β1 in women who subsequently developed preeclampsia
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61. Heme Oxygenase-1 Is Not Decreased in Preeclamptic Placenta and Does Not Negatively Regulate Placental Soluble fms-Like Tyrosine Kinase-1 or Soluble Endoglin Secretion
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