The DOI matches its Crossref record; 168 of the 168 references it cites were found in a registry or an index.
Sphingolipids and their metabolism in physiology and disease
Open accessAbstract
Studies of bioactive lipids in general and sphingolipids in particular have intensified over the past several years, revealing an unprecedented and unanticipated complexity of the lipidome and its many functions, which rivals, if not exceeds, that of the genome or proteome. These results highlight critical roles for bioactive sphingolipids in most, if not all, major cell biological responses, including all major cell signalling pathways, and they link sphingolipid metabolism to key human diseases. Nevertheless, the fairly nascent field of bioactive sphingolipids still faces challenges in its biochemical and molecular underpinnings, including defining the molecular mechanisms of pathway and enzyme regulation, the study of lipid–protein interactions and the development of cellular probes, suitable biomarkers and therapeutic approaches.
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Self-citationSecretory Mechanisms and Intercellular Transfer of MicroRNAs in Living Cells
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Gaitán, J. M., Moon, H. Y., Stremlau, M., Dubal, D. B., Cook, D. B., Okonkwo, O. C., & van Praag, H. (2021) · Frontiers in Endocrinology · doi:10.3389/fendo.2021.660181 · Cited by 160
Role of Sphingolipid Metabolism in Neurodegeneration
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Vaena, S., Chakraborty, P., Lee, H. G., Janneh, A. H., Kassir, M. F., Beeson, G., Hedley, Z., Yalcinkaya, A., Sofi, M. H., Li, H., et al. (2021) · Cell Reports · doi:10.1016/j.celrep.2021.109076 · Cited by 92
Metadata
- Authors
- Hannun, Y. A., & Obeid, L. M.
- Venue
- Nature Reviews Molecular Cell Biology
- Published
- 2017-11-22
- Volume
- 19
- Issue
- 3
- Pages
- 175-191
- ISSN
- 1471-0072 (ISSN-L)
- 1471-0080
- OpenAlex
- W2768617092
- Cited by
- 2,051
- References
- 168
- FWCI
- 8.04
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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