The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
First in vivo evidence of an NMDA receptor deficit in medication-free schizophrenic patients
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In the citing paper
“In humans, altered ERK activation causes a variety of developmental disorders which have ASD-like symptoms, although the pathogenic mechanisms are not precisely understood.”
From Transient blockade of ERK phosphorylation in the critical period causes autistic phenotypes as an adult in mice (2015) · cites this paper as [27]
In the citing paper
“It was reported that ASD is common in patients with 22q11.2 deletion syndrome27.”
From Transient blockade of ERK phosphorylation in the critical period causes autistic phenotypes as an adult in mice (2015) · cites this paper as [27]
In the citing paper
“An NMDAR single-photon emission computed tomography (SPECT) study pointed to lower NMDAR activity in unmedicated patients with schizophrenia 19 , although this finding awaits confirmation with newer, more specific probes 20 .”
From Linking early-life NMDAR hypofunction and oxidative stress in schizophrenia pathogenesis (2016) · cites this paper as [19]
Citations
Cites
Statistical parametric maps in functional imaging: A general linear approach
Friston, K. J., Holmes, A. P., Worsley, K. J., Poline, J. ‐., Frith, C. D., & Frackowiak, R. S. J. (1994) · Human Brain Mapping · doi:10.1002/hbm.460020402 · Cited by 9,688
Converging Evidence of NMDA Receptor Hypofunction in the Pathophysiology of Schizophrenia
Coyle, J. T., Tsai, G. E., & GOFF, D. C. (2003) · Annals of the New York Academy of Sciences · doi:10.1196/annals.1300.020 · Cited by 443
Takeuchi, R., Yonekura, Y., Matsuda, H., & Konishi, J. (2002) · European Journal of Nuclear Medicine and Molecular Imaging · doi:10.1007/s00259-001-0715-z · Cited by 134
Bressan, R. A., Erlandsson, K., Stone, J. M., Mulligan, R. S., Krystal, J. H., Ell, P. J., & Pilowsky, L. S. (2005) · Biological Psychiatry · doi:10.1016/j.biopsych.2005.03.016 · Cited by 57
Self-citationKinetic modelling of [123I]CNS 1261—a potential SPET tracer for the NMDA receptor
Erlandsson, K., Bressan, R. A., Mulligan, R. S., Gunn, R. N., Cunningham, V. J., Owens, J., Wyper, D. J., Ell, P. J., & Pilowsky, L. S. (2003) · Nuclear Medicine and Biology · doi:10.1016/s0969-8051(02)00450-x · Cited by 40
Self-citationLurito (2000) · doi:10.1002/1097-0193(200007)10:3
Research works printed but not linked: 1
- Stone J, Erlandsson K, Arstad E, Bressan RA, Teneggi V, Ell PJ et al. Schizophrenia Bull 2005; 31 (2): 446.
Cited by
Sorted by citations
Cognitive impairment in schizophrenia: aetiology, pathophysiology, and treatment
McCutcheon, R. A., Keefe, R. S. E., & McGuire, P. K. (2023) · Molecular Psychiatry · doi:10.1038/s41380-023-01949-9 · Cited by 861
Carlén, M., Meletis, K., Siegle, J. H., Cardin, J. A., Futai, K., Vierling-Claassen, D., Rühlmann, C., Jones, S. R., Deisseroth, K., Sheng, M., et al. (2012) · Molecular Psychiatry · doi:10.1038/mp.2011.31 · Cited by 669
Glutamate and dopamine in schizophrenia: An update for the 21 st century
Howes, O., McCutcheon, R., & Stone, J. (2015) · Journal of Psychopharmacology · doi:10.1177/0269881114563634 · Cited by 702
Self-citationThe Hippocampal Formation in Schizophrenia
Tamminga, C. A., Stan, A. D., & Wagner, A. D. (2010) · American Journal of Psychiatry · doi:10.1176/appi.ajp.2010.09081187 · Cited by 597
Schaefer, G. B., & Mendelsohn, N. J. (2013) · Genetics in Medicine · doi:10.1038/gim.2013.32 · Cited by 537
Metadata
- Authors
- Pilowsky, L. S., Bressan, R. A., Stone, J. M., Erlandsson, K., Mulligan, R. S., Krystal, J. H., & Ell, P. J.
- Affiliations
- King's College London (GB)
- University College London (GB)
- Yale University (US)
- Venue
- Molecular Psychiatry
- Published
- 2005-09-27
- Volume
- 11
- Issue
- 2
- Pages
- 118-119
- ISSN
- 1359-4184 (ISSN-L)
- 1476-5578
- OpenAlex
- W2066489132
- Cited by
- 278
- References
- 6
- Citations by year
- 2026: 1 · 2025: 4 · 2024: 6 · 2023: 8 · 2022: 11 · 2021: 9 · 2020: 11 · 2019: 8 · 2018: 7 · 2017: 14 · 2016: 20 · 2015: 19 · 2014: 16 · 2013: 21 · 2012: 17
- FWCI
- 6.65
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Molecular Psychiatry (publishedVersion)
- PubMed (publishedVersion)
- UCL Discovery (University College London) (submittedVersion)
- Research Portal (King's College London) (submittedVersion)
- Funders
- National Institutes of Health (5k05aa014906-03)
- National Institute on Alcohol Abuse and Alcoholism (KO5 AA 014906-02)
- Topics
- Health Sciences › Medicine › Psychiatry and Mental health › Schizophrenia research and treatment
- Life Sciences › Neuroscience › Cellular and Molecular Neuroscience › Neuroscience and Neuropharmacology Research
- Life Sciences › Neuroscience › Cellular and Molecular Neuroscience › Neurotransmitter Receptor Influence on Behavior
- Keywords
- NMDA receptor, schizophrenia, medication-free patients, receptor availability, first-episode psychosis, ketamine, brain volume
- MeSH terms
- Analysis of Variance, Clozapine, Guanidines, Hippocampus, Humans, Functional Laterality, Reference Values, Schizophrenia, Tissue Distribution, Antipsychotic Agents, Tomography, Emission-Computed, Single-Photon, Case-Control Studies, Receptors, N-Methyl-D-Aspartate, Genetic Predisposition to Disease
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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