Novel splice variants of the receptor for advanced glycation end-products expressed in human vascular endothelial cells and pericytes, and their putative roles in diabetes-induced vascular injury

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3. Low Levels of Serum Soluble Receptors for Advanced Glycation End Products, Biomarkers for Disease State: Myth or Reality
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4. Serum levels of soluble receptor for advanced glycation end products and of S100 proteins are associated with inflammatory, autoantibody, and classical risk markers of joint and vascular damage in rheumatoid arthritis
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5. Advanced glycation end products and their circulating receptors predict cardiovascular disease mortality in older community-dwelling women
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6. Receptor for Advanced Glycation End Products Regulates Adipocyte Hypertrophy and Insulin Sensitivity in Mice
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7. Alternative splicing of the murine receptor for advanced glycation end‐products (RAGE) gene
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8. Amyloid β Interaction with Receptor for Advanced Glycation End Products Up-Regulates Brain Endothelial CCR5 Expression and Promotes T Cells Crossing the Blood-Brain Barrier
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10. Soluble receptor for advanced glycation end products triggers a proinflammatory cytokine cascade via β2 integrin Mac‐1
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13. Mechanisms for the induction of HNE- MDA- and AGE-adducts, RAGE and VEGF in retinal pigment epithelial cells.
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14. The Amphoterin (HMGB1)/Receptor for Advanced Glycation End Products (RAGE) Pair Modulates Myoblast Proliferation, Apoptosis, Adhesiveness, Migration, and Invasiveness
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15. Methylglyoxal, oxidative stress, and hypertension
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16. Soluble Levels of Receptor for Advanced Glycation Endproducts (sRAGE) and Coronary Artery Disease
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17. Clinical Significance of Serum HMGB-1 and sRAGE Levels in Systemic Sclerosis: Association with Disease Severity
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18. Advanced glycation end products and their receptor in age-related, non-communicable chronic inflammatory diseases; Overview of clinical evidence and potential contributions to disease
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19. RAGE: The Beneficial and Deleterious Effects by Diverse Mechanisms of Actions
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20. The multiple faces of RAGE – opportunities for therapeutic intervention in aging and chronic disease
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21. Advanced Glycation End Products (AGEs) and Chronic Kidney Disease: Does the Modern Diet AGE the Kidney?
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22. RAGE, vascular tone and vascular disease
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23. AGE–RAGE Stress, Stressors, and Antistressors in Health and Disease
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24. Association of the Gly82Ser polymorphism in the receptor for advanced glycation end products (RAGE) gene with circulating levels of soluble RAGE and inflammatory markers in nondiabetic and nonobese Koreans
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25. Circulating soluble receptor for advanced glycation end products is inversely associated with body mass index and waist/hip ratio in the general population
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26. RAGE Expression and ROS Generation in Neurons: Differentiation versus Damage
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27. Increased levels of HMGB-1 and endogenous secretory RAGE in induced sputum from asthmatic patients
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28. Effects of Exercise on sRAGE Levels and Cardiometabolic Risk Factors in Patients with Type 2 Diabetes: A Randomized Controlled Trial
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29. RAGE regulates oxytocin transport into the brain
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30. Telmisartan inhibits expression of a receptor for advanced glycation end products (RAGE) in angiotensin-II-exposed endothelial cells and decreases serum levels of soluble RAGE in patients with essential hypertension
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31. Association of Polymorphism in the Receptor for Advanced Glycation End Products (RAGE) Gene with Circulating RAGE Levels
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32. Soluble RAGEs — Prospects for treating & tracking metabolic and inflammatory disease
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33. Potential New Therapeutic Agents for Diabetic Kidney Disease
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34. AGE–RAGE stress: a changing landscape in pathology and treatment of Alzheimer’s disease
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35. Serum levels of soluble form of receptor for advanced glycation end products (sRAGE) are positively associated with circulating AGEs and soluble form of VCAM-1 in patients with type 2 diabetes
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36. Low Serum Level of the Endogenous Secretory Receptor for Advanced Glycation End Products (esRAGE) Is a Risk Factor for Prevalent Vertebral Fractures Independent of Bone Mineral Density in Patients With Type 2 Diabetes
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37. Association Between Circulating Soluble Receptor for Advanced Glycation End Products and Atherosclerosis
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38. Involvement of the Toxic AGEs (TAGE)-RAGE System in the Pathogenesis of Diabetic Vascular Complications: A Novel Therapeutic Strategy
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39. Pathological Implications of Receptor for Advanced Glycation End-Product (AGER) Gene Polymorphism
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40. The S100B/RAGE Axis in Alzheimer's Disease
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41. The role of RAGE in host pathology and crosstalk between RAGE and TLR4 in innate immune signal transduction pathways
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42. Molecular Characteristics of RAGE and Advances in Small-Molecule Inhibitors
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43. Severity of diabetic microvascular complications is associated with a low soluble RAGE level
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44. Thiazolidinedione increases serum soluble receptor for advanced glycation end-products in type 2 diabetes
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45. Solution Structure of the Variable-Type Domain of the Receptor for Advanced Glycation End Products: New Insight into AGE−RAGE Interaction,
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46. Enhanced expression of receptor for advanced glycation end-products is associated with low circulating soluble isoforms of the receptor in Type 2 diabetes
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47. The receptor for advanced glycation end-products: A complex signaling scenario for a promiscuous receptor
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48. Dual coding in alternative reading frames correlates with intrinsic protein disorder
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49. Low Circulating Endogenous Secretory Receptor for AGEs Predicts Cardiovascular Mortality in Patients With End-Stage Renal Disease
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50. Alternative splicing of RAGE: roles in biology and disease
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51. Role of Advanced Glycation End Products With Oxidative Stress in Resistance Artery Dysfunction in Type 2 Diabetic Mice
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52. Increased Serum Concentrations of Soluble Receptor for Advanced Glycation Endproducts in Patients with Type 1 Diabetes
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53. Plasma advanced glycation end products are decreased in obese children compared with lean controls
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54. Role of Advanced Glycation End Products and Its Receptors in the Pathogenesis of Cigarette Smoke-Induced Cardiovascular Disease
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55. De-N-glycosylation or G82S mutation of RAGE sensitizes its interaction with advanced glycation endproducts
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56. The AGE-RAGE System and Diabetic Nephropathy
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57. Glyco-oxidation and cardiovascular complications in type 2 diabetes: a clinical update
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58. Receptor for advanced glycation end-products (RAGE) and soluble RAGE (sRAGE): cardiovascular implications
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59. High-Mobility Group Box-1 Protein and β-Amyloid Oligomers Promote Neuronal Differentiation of Adult Hippocampal Neural Progenitors via Receptor for Advanced Glycation End Products/Nuclear Factor-κB Axis: Relevance for Alzheimer's Disease
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60. THE AGE/RAGE AXIS IN DIABETES‐ACCELERATED ATHEROSCLEROSIS
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61. Implication of receptor for advanced glycation end product (RAGE) in pulmonary health and pathophysiology
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62. Receptor for advanced glycation end products--soluble form and gene polymorphisms in chronic haemodialysis patients
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63. Soluble receptor for AGE (RAGE) is a novel independent predictor of all-cause and cardiovascular mortality in type 1 diabetes
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66. Splice variants of the receptor for advanced glycosylation end products (RAGE) in human brain
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67. Increased glycated albumin and decreased esRAGE levels are related to angiographic severity and extent of coronary artery disease in patients with type 2 diabetes
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68. Serum endogenous secretory RAGE level is an independent risk factor for the progression of carotid atherosclerosis in type 1 diabetes
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70. Do Advanced Glycation End Products and Its Receptor Play a Role in Pathophysiology of Hypertension?
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71. From hyperglycemia to AGE-RAGE interaction on the cell surface: A dangerous metabolic route for diabetic patients
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72. Cancer Malignancy Is Enhanced by Glyceraldehyde-Derived Advanced Glycation End-Products
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73. Is there any evidence that AGE/sRAGE is a universal biomarker/risk marker for diseases?
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79. The −374A allele of the receptor for advanced glycation end products gene is associated with a decreased risk of ischemic heart disease in African-Brazilians with type 2 diabetes
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