The DOI matches its Crossref record; 13 of the 13 references it cites were found in a registry or an index.
Cardiac Plasticity
Abstract
This review explains how the heart responds to physiologic or pathologic conditions. Exercise, pregnancy, and postnatal growth cause physiologic growth; neurohumoral activation, hypertension, and myocardial injury cause hypertrophic growth, which increases the risk of heart failure and malignant arrhythmia. Atrophy of the heart can arise from protracted bed rest, prolonged weightlessness, or mechanical unloading with a ventricular assist device.
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Cites
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The progression from hypertension to congestive heart failure
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Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression
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The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress
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Cited by
MicroRNAs in Stress Signaling and Human Disease
Mendell, J. T., & Olson, E. N. (2012) · Cell · doi:10.1016/j.cell.2012.02.005 · Cited by 1,673
Self-citationFibrosis–a common pathway to organ injury and failure
Rockey, D. C., Bell, P. D., & Hill, J. A. (2015) · New England Journal of Medicine · doi:10.1056/nejmra1300575 · Cited by 1,428
Self-citationPervasive roles of microRNAs in cardiovascular biology
Small, E. M., & Olson, E. N. (2011) · Nature · doi:10.1038/nature09783 · Cited by 1,180
Self-citationZhang, J., Gajjala, S., Agrawal, P., Tison, G. H., Hallock, L. A., Beussink-Nelson, L., Lassen, M. H., Fan, E., Aras, M. A., Jordan, C., et al. (2018) · Circulation · doi:10.1161/circulationaha.118.034338 · Cited by 1,006
“A subset of these videos was used for the 4 main tasks of our pipeline: (1) view classification, (2) image segmentation, (3) measurements of cardiac structure and function, and (4) disease detection.”
BackgroundMethodInfluentialCardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease
Fan, D., Takawale, A., Lee, J., & Kassiri, Z. (2012) · Fibrogenesis & Tissue Repair · doi:10.1186/1755-1536-5-15 · Cited by 814
Metadata
- Authors
- Hill, J. A., & Olson, E. N.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 2008-03-27
- Volume
- 358
- Issue
- 13
- Pages
- 1370-1380
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W4232767515
- Cited by
- 1,065
- References
- 13
- FWCI
- 38.28
- Type
- journal article
- Language
- en
- Open access
- closed
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Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.