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Cited by 424

The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

Fetal origins of cardiovascular disease

Barker, David J P·1999·Annals of Medicine

Abstract

Low birthweight, thinness and short body length at birth are now known to be associated with increased rates of cardiovascular disease and non-insulin dependent diabetes in adult life. The fetal origins hypothesis proposes that these diseases originate through adaptations which the fetus makes when it is undernourished. These adaptations may be cardiovascular, metabolic or endocrine. They permanently change the structure and function of the body. Prevention of the diseases may depend on prevention of imbalances in fetal growth or imbalances between prenatal and postnatal growth, or imbalances in nutrient supply to the fetus.

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Crossref · 10.1080/07853890.1999.11904392

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Metadata

Authors
Barker, D. J. P.
Venue
Annals of Medicine
Published
1999-01
Volume
31
Issue
sup1
Pages
3-6
ISSN
  • 0785-3890 (ISSN-L)
  • 1365-2060
OpenAlex
W4687941
Cited by
424
References
6
FWCI
16.18
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Fetal origins of cardiovascular disease. https://app.cochat.ai/papers/10.1080/07853890.1999.11904392
The paper
Barker, D. J. P. (1999). Fetal origins of cardiovascular disease. Annals of Medicine. https://doi.org/10.1080/07853890.1999.11904392

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.