Anti-PD-1 Antibody Therapy Induces Hashimoto’s Disease with an Increase in Peripheral Blood Follicular Helper T Cells
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Histological and morphological analysis can demonstrate different pathophysiological mechanisms between autoimmune and ICPi-induced thyrotoxicosis.
“Histological and morphological analysis can demonstrate different pathophysiological mechanisms between autoimmune and ICPi-induced thyrotoxicosis [26,27].”
From Graves’ Disease during Immune Checkpoint Inhibitor Therapy (A Case Series and Literature Review) (2021) · cites this paper as [26]
Cites
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“Histological and morphological analysis can demonstrate different pathophysiological mechanisms between autoimmune and ICPi-induced thyrotoxicosis [26,27].”
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- Authors
- Torimoto, K., Okada, Y., Nakayamada, S., Kubo, S., & Tanaka, Y.
- Affiliations
- University of Occupational and Environmental Health Japan (JP)
- Venue
- Thyroid
- Publisher
- SAGE Publications
- Published
- 2017-10
- Volume
- 27
- Issue
- 10
- Pages
- 1335-1336
- ISSN
- 1050-7256 (ISSN-L)
- 1557-9077
- OpenAlex
- W2735015334
- Cited by
- 20
- References
- 4
- Citations by year
- 2025: 3 · 2024: 2 · 2023: 1 · 2022: 2 · 2021: 3 · 2020: 2 · 2019: 5 · 2018: 2
- FWCI
- 1.94
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Thyroid (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Thyroid Disorders and Treatments
- Health Sciences › Medicine › Neurology › Autoimmune Neurological Disorders and Treatments
- Life Sciences › Immunology and Microbiology › Immunology › T-cell and B-cell Immunology
- Keywords
- anti-PD-1, immune checkpoint inhibitors, Hashimoto's thyroiditis, T follicular helper cells, autoimmunity, thyroid autoimmunity
- MeSH terms
- Nivolumab, Antibodies, Monoclonal, Antineoplastic Agents, T-Lymphocytes, Helper-Inducer, Humans, Lung Neoplasms, Male, Middle Aged, Thyroxine, Treatment Outcome, Hashimoto Disease
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