The DOI matches its Crossref record; 14 of the 14 references it cites were found in a registry or an index.
Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling
Open accessAbstract
A defining cellular event in the transition from compensated hypertrophy to dilated cardiomyopathy is cardiomyocyte drop-out due to apoptosis, programmed necrosis, and autophagy. The importance of apoptosis in heart failure has been recognized for over a decade, while other forms of programmed cell death have more recently been appreciated, and their pathophysiological roles continue to be defined in experimental and clinical heart failure. The major focus of this review is on apoptosis in heart failure, with a discussion of molecular cross-talk between apoptosis, autophagy, and programmed necrosis.
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Cites
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Cited by
Functional screening identifies miRNAs inducing cardiac regeneration
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Animal Models of Heart Failure: A Scientific Statement From the American Heart Association
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Innate immune signaling in cardiac ischemia
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Metadata
- Authors
- Dorn, G. W.
- Venue
- Cardiovascular Research
- Publisher
- Oxford University Press (OUP)
- Published
- 2008-10-29
- Volume
- 81
- Issue
- 3
- Pages
- 465-473
- ISSN
- 0008-6363 (ISSN-L)
- OpenAlex
- W1986377133
- Cited by
- 305
- References
- 14
- FWCI
- 7.57
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Dorn, G. W. (2008). Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling. Cardiovascular Research. https://doi.org/10.1093/cvr/cvn243
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
